PMC

Distributions for three species of Nannomys in southern Africa. Dark grey indicates distribution for Mus minutoides, light grey for Mus indutus, and stippled pattern for Mus setzeri, adapted from , , and , respectively. Five trapping localities in Botswana (a); black crosses indicate captures for Mus minutoides and grey triangles for Mus indutus. Records from northwestern Botswana, Ngamiland District (b). Locality of syntopic records for Mus indutus and Mus minutoides at Koanaka Hills site (c).

Karyotypes of female TK164752 (a) and male TK164768 (c) Mus minutoides and female TK164753 Mus indutus (e) from Botswana. The chromosome arms identified in yellow on the images to the right of each karyogram correspond to regions of homology to the X chromosome of Mus musculus detected by FISH for female TK164752 (b) and male TK164768 (d) Mus minutoides and female TK164820 Mus indutus (f). Note that in Mus minutoides, a single chromosome arm shows homology to the X chromosome of the house mouse, indicating the presence of an X-autosome translocation, whereas a whole acrocentric chromosome corresponds to the X of Mus indutus. The insert on (b) represents the (1.X) translocation of individual TK164752 Mus minutoides, with the long arm corresponding to the X chromosome.

Multiple ⁷⁵As spin-echo decays under APCP pulses along with the Hahn-echo decay. Top: un-doped GaAs, Middle: Cr-doped semi-insulating GaAs, Bottom: Si-doped metallic GaAs. In each figure, the data closest to the vertical axis is the Hahn-echo decay, which is obtained by sweeping the pulse interval τ between the first π/2- and the next π-pulse. For shorter and longer pulse intervals, the decays can be fit by two-exponential functions. For intermediate pulse intervals, however, the T₂^Ls in the two-component fittings do not represent the right slope in the long-time limit^SI-3. Fitting with three time constants is found to reproduce the decays for all the pulse intervals^SI-4, resulting in the right T₂^Ls that characterizes the long-time slopes. For the un-doped GaAs, the pulse interval 2 τ are 30 μs, 40 μs, 60 μs, 100 μs, 150 μs, 170 μs, 200 μs, 250 μs, 300 μs, 400 μs, 500 μs and 600 μs. For the Cr-doped GaAs, the pulse interval 2 τ are 30 μs, 40 μs, 60 μs, 100 μs, 150 μs, 170 μs, 200 μs, 240 μs, 300 μs, 400 μs and 600 μs. For the Si-doped GaAs, the pulse interval 2 τ are 30 μs, 40 μs, 60 μs, 100 μs, 150 μs, 170 μs, 200 μs, 250 μs, 300 μs, 400 μs and 600 μs.

Expression analysis of TPM1α and TPM1κ RNA in mouse heart and skeletal muscle by RT-PCR. Plate (a) illustrates the ethidium bromide stain. Plate (b) illustrates hybridization with TPM1α probe. Plate (c) illustrates hybridization with TPM1κ probe. Lane 1: heart cDNA amplified with Mus TPM1α/κ-1 (+)/Mus UTR (−). Lane 2: heart cDNA amplified with Mus TPM1α/κ-1 (+)/Mus TPM1α/κ-2 (−). Lane 3: heart cDNA amplified with Mus TPM1κ-1 (+)/Mus TPM1α/κ-2 (−). Lane 4: heart cDNA amplified with TPM1α (+)/Mus TPM1α/κ-2 (−). Lane 5: heart cNDA amplified with Mus TPM1κ-1 (+)/Mus UTR (−). Lane 6: primer control for Mus TPM1α/κ-1 (+)/Mus UTR (−). Lane 7: primer control for Mus TPM1α/κ-1 (+)/Mus TPM1α/κ-2 (−). Lane 8: primer control for Mus TPM1κ-1 (+)/Mus TPM1α/κ-2 (−). Lane 9: skeletal cDNA amplified with Mus TPM1α/κ-1 (+)/Mus UTR (−). Lane 10: skeletal cDNA amplified with Mus TPM1α/κ-1 (+)/Mus TPM1α/κ-2 (−). Lane 11: skeletal cDNA amplified with Mus TPM1κ-1 (+)/Mus TPM1α/κ-2 (−). Lane 12: skeletal cDNA amplified with Mus TPM1α-1 (+)/Mus TPM1α/κ-2 (−).