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SRX1330982: GSM1906377: DamIP-seq in IMR90hTert cells expressing MAF1A_DamK9A, (SS) rep2; Homo sapiens; OTHER
1 ILLUMINA (Illumina HiSeq 2500) run: 54.2M spots, 5.4G bases, 1.7Gb downloads

Submitted by: Gene Expression Omnibus (GEO)
Study: Genome-wide MAF1-dependent regulation of RNA polymerase III transcription [DamIP-Seq]
show Abstracthide Abstract
In higher eukaryotes, an important mechanism to tune translation in different tissues and conditions is mTORC1-dependent regulation of tRNAs transcription by RNA polymerase III (Pol III), as the relative amount of tRNAs tightly coordinates with the translational needs of the cell. mTORC1 contributes to regulate protein synthesis through its direct substrate MAF1, which functions as a negative regulator of Pol III transcription in response to stimuli such as serum starvation or rapamycin treatment. Here, we applied ChIP-seq to examine the Pol III occupancy profile in human fibroblasts and report evidence of a genome wide, MAF1-dependent coordinated response to favorable or stress growth conditions. Strikingly, while a set of genes is extremely responsive in terms of Pol III binding, other genes are mostly unperturbed, yet associated with transcriptionally engaged polymerases as revealed by nascent EU-labeled RNA-seq (neuRNA-seq). As shown by DamIP-seq, the responsiveness of a subset of genes is tightly connected to the rapid and transient interaction of MAF1 with DNA-bound Pol III. Overall design: We performed duplicate ChIP-seq experiments for the Rpc4 (POLR3D) subunit of RNA polymerase III in IMR90hTert cells grown in the presence of fetal bovine serum (FBS), serum starved (SS), serum starved and treated with insulin (SS+I), serum starved and treated with insulin and rapamycin (SS+R+I). Additional ChIP-seq profiles were generated in cells treated with MAF1 siRNAs and serum starved. MAF1 binding was addressed by DamIP-seq, using two replicates per clone of IMR90hTert cells expressing HA-tagged MAF1-DamK9A (2 different clones) or EGFP-DamK9A (2 different clones). To monitor dynamic transcription profiles we did neusRNA-seq in IMR90hTert cells EU-labeled or mock (DMSO)-labeled. For both DamIP-seq and neusRNA-seq, cells were either unperturbed or serum starved.
Sample: DamIP-seq in IMR90hTert cells expressing MAF1A_DamK9A, (SS) rep2
SAMN04160414 • SRS1113851 • All experiments • All runs
Organism: Homo sapiens
Library:
Instrument: Illumina HiSeq 2500
Strategy: OTHER
Source: GENOMIC
Selection: other
Layout: SINGLE
Construction protocol: DamIP-seq was performed as described (Xiao et al., 2010, 2012) with modifications. In brief, 10 µg of gDNA were sheared to 100-300bp with a Covaris S220 Focused-Ultrasonicator in 6x16mm µTUBE (Covaris, Cat. 520045). DNA was boiled 10’ at 98°C, cooled 10’ in ice, diluted with 1 volume of ice-cold 2X DamIP buffer (20 mM Tris-Cl pH 7.4, 300 mM NaCl and 0.2% NP40) and incubated O/N at 4°C under rotation with 4 µg of Anti-N6-methyladenosine antibody (Millipore, 1 µg/µl, ABE572). Antibody-DNA complexes were pulled-down with 25 µl of Dynabeads protein-A (Life technologies, 10002D) by 1h incubation at room temperature. Beads were washed four times in 1X DamIP buffer and resuspended in 300 µl of Elution buffer (50 mM Tris-Cl pH 8, 10 mM EDTA and 0.5% SDS) with 40 µg of Proteinase K. After 3h incubation at 48°C, immunoprecipitated single stranded DNA (ssDNA) was extracted with phenol chloroform, ethanol precipitated and resuspended in 17 µl of EB buffer. Libraries were prepared starting from single strand cDNA with the TruSeq RNA sample prep v2 kit (Illumina, RS-122-2001) according to manufacturer’s instructions, but using Nucleospin columns and NTC buffer when required instead of AmpureXP beads.
Experiment attributes:
GEO Accession: GSM1906377
Links:
Runs: 1 run, 54.2M spots, 5.4G bases, 1.7Gb
Run# of Spots# of BasesSizePublished
SRR263409054,185,4065.4G1.7Gb2016-02-15

ID:
1896429

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