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25398711
Failure of nocturnal changes in growth hormone to alter carbohydrate tolerance the following morning.
Diabetologia.
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Normal glucose-induced suppression of glucose production but impaired stimulation of glucose disposal in type 2 diabetes: evidence for a concentration-dependent defect in uptake.
Diabetes.
1998 Nov;47(11):1735-47. doi: 10.2337/diabetes.47.11.1735. PubMed PMID:
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Effect of overnight restoration of euglycemia on glucose effectiveness in type 2 diabetes mellitus.
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Impact of lack of suppression of glucagon on glucose tolerance in humans.
Am J Physiol.
1999 Aug;277(2):E283-90. doi: 10.1152/ajpendo.1999.277.2.E283. PubMed PMID:
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Effects of type 2 diabetes on the ability of insulin and glucose to regulate splanchnic and muscle glucose metabolism: evidence for a defect in hepatic glucokinase activity.
Diabetes.
2000 Feb;49(2):272-83. doi: 10.2337/diabetes.49.2.272. PubMed PMID:
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Effect of glucagon-like peptide 1(7-36) amide on glucose effectiveness and insulin action in people with type 2 diabetes.
Diabetes.
2000 Apr;49(4):611-7. doi: 10.2337/diabetes.49.4.611. PubMed PMID:
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Lack of suppression of glucagon contributes to postprandial hyperglycemia in subjects with type 2 diabetes mellitus.
J Clin Endocrinol Metab.
2000 Nov;85(11):4053-9. doi: 10.1210/jcem.85.11.6993. PubMed PMID:
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Effect of glucagon-like peptide-1(7-36)-amide on initial splanchnic glucose uptake and insulin action in humans with type 1 diabetes.
Diabetes.
2001 Mar;50(3):565-72. doi: 10.2337/diabetes.50.3.565. PubMed PMID:
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Type 2 diabetes impairs splanchnic uptake of glucose but does not alter intestinal glucose absorption during enteral glucose feeding: additional evidence for a defect in hepatic glucokinase activity.
Diabetes.
2001 Jun;50(6):1351-62. doi: 10.2337/diabetes.50.6.1351. PubMed PMID:
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Systemic and regional free fatty acid metabolism in type 2 diabetes.
Am J Physiol Endocrinol Metab.
2001 Jun;280(6):E1000-6. doi: 10.1152/ajpendo.2001.280.6.E1000. PubMed PMID:
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Type I diabetes mellitus does not alter initial splanchnic glucose extraction or hepatic UDP-glucose flux during enteral glucose administration.
Diabetologia.
2001 Jun;44(6):729-37. doi: 10.1007/s001250051682. PubMed PMID:
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Effects of free fatty acids and glycerol on splanchnic glucose metabolism and insulin extraction in nondiabetic humans.
Diabetes.
2002 Feb;51(2):301-10. doi: 10.2337/diabetes.51.2.301. PubMed PMID:
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Effect of enteral vs. parenteral glucose delivery on initial splanchnic glucose uptake in nondiabetic humans.
Am J Physiol Endocrinol Metab.
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Lack of effect of exendin-4 and glucagon-like peptide-1-(7,36)-amide on insulin action in non-diabetic humans.
Diabetologia.
2002 Oct;45(10):1410-5. doi: 10.1007/s00125-002-0924-4. Epub 2002 Sep 5. PubMed PMID:
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Elevated free fatty acids impair glucose metabolism in women: decreased stimulation of muscle glucose uptake and suppression of splanchnic glucose production during combined hyperinsulinemia and hyperglycemia.
Diabetes.
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Use of a novel triple-tracer approach to assess postprandial glucose metabolism.
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POEMS syndrome: definitions and long-term outcome.
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Mechanisms of the age-associated deterioration in glucose tolerance: contribution of alterations in insulin secretion, action, and clearance.
Diabetes.
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Glucose-induced suppression of endogenous glucose production: dynamic response to differing glucose profiles.
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Enteral infusion of glucose at rates approximating EGP enhances glucose disposal but does not cause hypoglycemia.
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Higher insulin concentrations are required to suppress gluconeogenesis than glycogenolysis in nondiabetic humans.
Diabetes.
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Both fasting glucose production and disappearance are abnormal in people with "mild" and "severe" type 2 diabetes.
Am J Physiol Endocrinol Metab.
2004 Jul;287(1):E55-62. doi: 10.1152/ajpendo.00549.2003. Epub 2004 Feb 24. PubMed PMID:
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Splanchnic cortisol production occurs in humans: evidence for conversion of cortisone to cortisol via the 11-beta hydroxysteroid dehydrogenase (11beta-hsd) type 1 pathway.
Diabetes.
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Insulin dose-response curves for stimulation of splanchnic glucose uptake and suppression of endogenous glucose production differ in nondiabetic humans and are abnormal in people with type 2 diabetes.
Diabetes.
2004 Aug;53(8):2042-50. doi: 10.2337/diabetes.53.8.2042. PubMed PMID:
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Effects of type 2 diabetes on the regulation of hepatic glucose metabolism.
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Minimal model estimation of glucose absorption and insulin sensitivity from oral test: validation with a tracer method.
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2004 Oct;287(4):E637-43. doi: 10.1152/ajpendo.00319.2003. Epub 2004 May 11. PubMed PMID:
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Insulin regulation of free fatty acid kinetics in adult cystic fibrosis patients with impaired glucose tolerance.
Metabolism.
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Obesity and type 2 diabetes do not alter splanchnic cortisol production in humans.
J Clin Endocrinol Metab.
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Obesity and type 2 diabetes impair insulin-induced suppression of glycogenolysis as well as gluconeogenesis.
Diabetes.
2005 Jul;54(7):1942-8. doi: 10.2337/diabetes.54.7.1942. PubMed PMID:
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Measurement of selective effect of insulin on glucose disposal from labeled glucose oral test minimal model.
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Two-hour seven-sample oral glucose tolerance test and meal protocol: minimal model assessment of beta-cell responsivity and insulin sensitivity in nondiabetic individuals.
Diabetes.
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A minimal model of insulin secretion and kinetics to assess hepatic insulin extraction.
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A model of glucose production during a meal.
Conf Proc IEEE Eng Med Biol Soc.
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Effect of nutrient ingestion on total-body and splanchnic cortisol production in humans.
Diabetes.
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Relationship between beta-cell mass and fasting blood glucose concentration in humans.
Diabetes Care.
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Effects of age and sex on postprandial glucose metabolism: differences in glucose turnover, insulin secretion, insulin action, and hepatic insulin extraction.
Diabetes.
2006 Jul;55(7):2001-14. doi: 10.2337/db05-1692. PubMed PMID:
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Assessment of postprandial glucose metabolism: conventional dual- vs. triple-tracer method.
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DHEA in elderly women and DHEA or testosterone in elderly men.
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Splanchnic cortisol production in dogs occurs primarily in the liver: evidence for substantial hepatic specific 11beta hydroxysteroid dehydrogenase type 1 activity.
Diabetes.
2006 Nov;55(11):3013-9. doi: 10.2337/db06-0601. PubMed PMID:
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Prediction of postprandial glycemic exposure: utility of fasting and 2-h glucose measurements alone and in combination with assessment of body composition, fitness, and strength.
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Pathogenesis of pre-diabetes: mechanisms of fasting and postprandial hyperglycemia in people with impaired fasting glucose and/or impaired glucose tolerance.
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Two years of treatment with dehydroepiandrosterone does not improve insulin secretion, insulin action, or postprandial glucose turnover in elderly men or women.
Diabetes.
2007 Mar;56(3):753-66. doi: 10.2337/db06-1504. PubMed PMID:
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Effects of nonglucose nutrients on insulin secretion and action in people with pre-diabetes.
Diabetes.
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Contribution of hepatic and extrahepatic insulin resistance to the pathogenesis of impaired fasting glucose: role of increased rates of gluconeogenesis.
Diabetes.
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Triglyceride uptake and lipoprotein lipase-generated fatty acid spillover in the splanchnic bed of dogs.
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Endocrinopathy in POEMS syndrome: the Mayo Clinic experience.
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Selective downregulation of the high molecular weight form of adiponectin in hyperinsulinemia and in type 2 diabetes: differential regulation from nondiabetic subjects.
Diabetes.
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Effect of 2 years of testosterone replacement on insulin secretion, insulin action, glucose effectiveness, hepatic insulin clearance, and postprandial glucose turnover in elderly men.
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