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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1966 1
1971 1
1972 1
1975 1
1976 1
1977 2
1978 4
1979 2
1980 1
1981 2
1983 2
1984 1
1985 3
1988 3
1990 1
1991 1
1992 1
1995 1
2024 0

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29 results

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Page 1
Selective MAO A and B inhibitors: their mechanism of action and pharmacology.
Finberg JP, Youdim MB. Finberg JP, et al. Neuropharmacology. 1983 Mar;22(3 Spec No):441-6. doi: 10.1016/0028-3908(83)90194-6. Neuropharmacology. 1983. PMID: 6304562 Review.
The 14C-selective irreversible "suicide" MAO A (clorgyline, Lilly 51641; M & B 9303) and MAO B inhibitors (deprenyl, AGN 1135 and pargyline) bind to the enzyme active site stoichiometrically mol/mol of enzyme. ...
The 14C-selective irreversible "suicide" MAO A (clorgyline, Lilly 51641; M & B 9303) and MAO B inhibitors (deprenyl, AGN 1 …
A comparison of the pharmacological and biochemical properties of substrate-selective monoamine oxidase inhibitors.
Christmas AJ, Coulson CJ, Maxwell DR, Riddell D. Christmas AJ, et al. Br J Pharmacol. 1972 Jul;45(3):490-503. doi: 10.1111/j.1476-5381.1972.tb08106.x. Br J Pharmacol. 1972. PMID: 5072232 Free PMC article.
Concentrations of M&B 9302 and Lilly 51641 required to produce 50% inhibition of 5-HT oxidation by brain mitochondrial MAO were 1.4 x 10(-8)M and 2.5 x 10(-7)M respectively. ...All the inhibitors except E-250 produced a dose-related rise in brain 5-HT levels. On …
Concentrations of M&B 9302 and Lilly 51641 required to produce 50% inhibition of 5-HT oxidation by brain mitochondrial MAO …
Spinal noradrenergic terminal system mediates antinociception.
Reddy SV, Yaksh TL. Reddy SV, et al. Brain Res. 1980 May 12;189(2):391-401. doi: 10.1016/0006-8993(80)90099-2. Brain Res. 1980. PMID: 6245762
The effect of intrathecal NE was significantly potentiated by prior administration of Lilly 51641 (a monoamine oxidase inhibitor) and protriptyline (a re-uptake inhibitor), and was not antagonized by the intrathecal administration of a non-specific vasodilator, papa …
The effect of intrathecal NE was significantly potentiated by prior administration of Lilly 51641 (a monoamine oxidase inhibit …
In vitro pigment formation from tryptamine. Role of indole-3-acetaldehyde.
Das PK, Guha SR. Das PK, et al. Biochem Pharmacol. 1985 Aug 1;34(15):2663-7. doi: 10.1016/0006-2952(85)90564-7. Biochem Pharmacol. 1985. PMID: 4015706
Among the four type selective MAO inhibitors used, pargyline and deprenyl appear to be more effective in inhibiting pigment formation from tryptamine than serotonin, while in the presence of clorgyline and Lilly 51641, pigment formation from serotonin was preferenti …
Among the four type selective MAO inhibitors used, pargyline and deprenyl appear to be more effective in inhibiting pigment formation from t …
Characteristics of the inhibition of rat brain monoamine oxidase in vitro by MD780515.
Kan JP, Strolin Benedetti M. Kan JP, et al. J Neurochem. 1981 Apr;36(4):1561-71. doi: 10.1111/j.1471-4159.1981.tb00599.x. J Neurochem. 1981. PMID: 7264651
MD780515 was a potent inhibitor (IC50 = 1-2 nM) of [3H]harmaline binding. Comparatively, clorgyline, 'cold' harmaline and Lilly 51641 inhibited 3H ligand binding, with IC50 of 5, 7 and 40 nM respectively. ...
MD780515 was a potent inhibitor (IC50 = 1-2 nM) of [3H]harmaline binding. Comparatively, clorgyline, 'cold' harmaline and Lilly 51 …
Pyridoxal 5'-phosphate levels in brain after treatments which impair cerebral glucose metabolism.
Wong KL, Tyce GM. Wong KL, et al. Neurochem Res. 1979 Dec;4(6):821-6. doi: 10.1007/BF00964478. Neurochem Res. 1979. PMID: 44547
Cerebral PLP concentrations were reduced after some of these treatments, notably injection of ethanol, or L-dopa alone or with beta-phenylisopropylhydrazine, an inhibitor of MAO, or of 5-HTP together with N-[beta-(chlorophenoxy)ethyl]cyclopropylamine hydrochloride, Lilly …
Cerebral PLP concentrations were reduced after some of these treatments, notably injection of ethanol, or L-dopa alone or with beta-phenylis …
The effect of pargyline and other monoamine oxidase inhibitors on blood acetaldehyde levels in ethanol-intoxicated mice.
Dembiec D, MacNamee D, Cohen G. Dembiec D, et al. J Pharmacol Exp Ther. 1976 May;197(2):332-9. J Pharmacol Exp Ther. 1976. PMID: 944772
The magnitude of the increase in blood acetaldehyde levels was dependent upon the dose of pargyline between 20 and 100 mg/kg; however, the elevation was relatively independent of the ethanol dose between 1 and 6 g/kg. Of the other monoamine oxidase inhibitors tested, Lilly …
The magnitude of the increase in blood acetaldehyde levels was dependent upon the dose of pargyline between 20 and 100 mg/kg; however, the e …
29 results