Format
Sort by

Send to

Choose Destination

Search results

Items: 4

1.

Loss-of-function of PTPR γ and ζ, observed in sporadic schizophrenia, causes brain region-specific deregulation of monoamine levels and altered behavior in mice.

Cressant A, Dubreuil V, Kong J, Kranz TM, Lazarini F, Launay JM, Callebert J, Sap J, Malaspina D, Granon S, Harroch S.

Psychopharmacology (Berl). 2017 Feb;234(4):575-587. doi: 10.1007/s00213-016-4490-8. Epub 2016 Dec 26.

PMID:
28025742
2.

Increased expression of receptor phosphotyrosine phosphatase-β/ζ is associated with molecular, cellular, behavioral and cognitive schizophrenia phenotypes.

Takahashi N, Sakurai T, Bozdagi-Gunal O, Dorr NP, Moy J, Krug L, Gama-Sosa M, Elder GA, Koch RJ, Walker RH, Hof PR, Davis KL, Buxbaum JD.

Transl Psychiatry. 2011 May 10;1:e8. doi: 10.1038/tp.2011.8.

3.

No association between the protein tyrosine phosphatase, receptor-type, Z Polypeptide 1 (PTPRZ1) gene and schizophrenia in the Japanese population.

Ito Y, Yamada S, Takahashi N, Saito S, Yoshimi A, Inada T, Noda Y, Ozaki N.

Am J Med Genet B Neuropsychiatr Genet. 2008 Oct 5;147B(7):1013-8. doi: 10.1002/ajmg.b.30692.

PMID:
18186075
4.

Molecular dissection of NRG1-ERBB4 signaling implicates PTPRZ1 as a potential schizophrenia susceptibility gene.

Buxbaum JD, Georgieva L, Young JJ, Plescia C, Kajiwara Y, Jiang Y, Moskvina V, Norton N, Peirce T, Williams H, Craddock NJ, Carroll L, Corfas G, Davis KL, Owen MJ, Harroch S, Sakurai T, O'Donovan MC.

Mol Psychiatry. 2008 Feb;13(2):162-72. Epub 2007 Apr 17.

Supplemental Content

Loading ...
Support Center