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Items: 1 to 20 of 169

1.

Targeting Prion-like Cis Phosphorylated Tau Pathology in Neurodegenerative Diseases.

Albayram O, Angeli P, Bernstein E, Baxley S, Gao Z, Lu KP, Zhou XZ.

J Alzheimers Dis Parkinsonism. 2018;8(3). pii: 443. doi: 10.4172/2161-0460.1000443. Epub 2018 Jun 29.

PMID:
30197831
2.

Targeting metals rescues the phenotype in an animal model of tauopathy.

Sedjahtera A, Gunawan L, Bray L, Hung LW, Parsons J, Okamura N, Villemagne VL, Yanai K, Liu XM, Chan J, Bush AI, Finkelstein DI, Barnham KJ, Cherny RA, Adlard PA.

Metallomics. 2018 Sep 19;10(9):1339-1347. doi: 10.1039/c8mt00153g.

PMID:
30168573
3.

Pin1 Modulation in Physiological Status and Neurodegeneration. Any Contribution to the Pathogenesis of Type 3 Diabetes?

Bianchi M, Manco M.

Int J Mol Sci. 2018 Aug 8;19(8). pii: E2319. doi: 10.3390/ijms19082319. Review.

4.

The trans isomer of Tau peptide is prone to aggregate, and the WW domain of Pin1 drastically decreases its aggregation.

Ikura T, Tochio N, Kawasaki R, Matsuzaki M, Narita A, Kikumoto M, Utsunomiya-Tate N, Tate SI, Ito N.

FEBS Lett. 2018 Sep;592(18):3082-3091. doi: 10.1002/1873-3468.13218. Epub 2018 Aug 13.

PMID:
30079475
5.

Integrative Analysis of Hippocampus Gene Expression Profiles Identifies Network Alterations in Aging and Alzheimer's Disease.

Lanke V, Moolamalla STR, Roy D, Vinod PK.

Front Aging Neurosci. 2018 May 23;10:153. doi: 10.3389/fnagi.2018.00153. eCollection 2018.

6.

Okadaic acid-induced tau hyperphosphorylation and the downregulation of Pin1 expression in primary cortical neurons.

Metin-Armağan D, Gezen-Ak D, Dursun E, Atasoy İL, Karabay A, Yılmazer S, Öztürk M.

J Chem Neuroanat. 2018 Oct;92:41-47. doi: 10.1016/j.jchemneu.2018.05.006. Epub 2018 May 31.

PMID:
29860071
7.

Enhanced Sampling of Interdomain Motion Using Map-Restrained Langevin Dynamics and NMR: Application to Pin1.

Bouchard JJ, Xia J, Case DA, Peng JW.

J Mol Biol. 2018 Jul 6;430(14):2164-2180. doi: 10.1016/j.jmb.2018.05.007. Epub 2018 May 16.

PMID:
29775635
8.

Alzheimer's Disease, Dendritic Spines, and Calcineurin Inhibitors: A New Approach?

O'Neal MA, Stallings NR, Malter JS.

ACS Chem Neurosci. 2018 Jun 20;9(6):1233-1234. doi: 10.1021/acschemneuro.8b00213. Epub 2018 May 14.

PMID:
29757603
9.

New evidence to elucidate the controversial Pin1 concentration in human serum.

Wang JZ, Bai J.

J Clin Neurosci. 2018 Jul;53:282-283. doi: 10.1016/j.jocn.2018.04.071. Epub 2018 May 4. No abstract available.

PMID:
29735262
10.

Viral induced oxidative and inflammatory response in Alzheimer's disease pathogenesis with identification of potential drug candidates: A systematic review using systems biology approach.

Talwar P, Gupta R, Kushwaha S, Agarwal R, Saso L, Kukreti S, Kukreti R.

Curr Neuropharmacol. 2018 Apr 19. doi: 10.2174/1570159X16666180419124508. [Epub ahead of print]

PMID:
29676229
11.

Effects of peptidyl-prolyl isomerase 1 depletion in animal models of prion diseases.

Legname G, Virgilio T, Bistaffa E, De Luca CMG, Catania M, Zago P, Isopi E, Campagnani I, Tagliavini F, Giaccone G, Moda F.

Prion. 2018 Mar 4;12(2):127-137. doi: 10.1080/19336896.2018.1464367. Epub 2018 May 18.

PMID:
29676205
12.

Pin1 mediates Aβ42-induced dendritic spine loss.

Stallings NR, O'Neal MA, Hu J, Kavalali ET, Bezprozvanny I, Malter JS.

Sci Signal. 2018 Mar 20;11(522). pii: eaap8734. doi: 10.1126/scisignal.aap8734.

13.

Melatonin ameliorates Aβ42 -induced alteration of βAPP-processing secretases via the melatonin receptor through the Pin1/GSK3β/NF-κB pathway in SH-SY5Y cells.

Chinchalongporn V, Shukla M, Govitrapong P.

J Pineal Res. 2018 May;64(4):e12470. doi: 10.1111/jpi.12470. Epub 2018 Feb 8.

PMID:
29352484
14.
15.

The pathological roles of NDRG2 in Alzheimer's disease, a study using animal models and APPwt-overexpressed cells.

Rong XF, Sun YN, Liu DM, Yin HJ, Peng Y, Xu SF, Wang L, Wang XL.

CNS Neurosci Ther. 2017 Aug;23(8):667-679. doi: 10.1111/cns.12716. Epub 2017 Jul 2.

PMID:
28670853
16.

Investigation of gene expression and serum levels of PIN1 and eNOS with high blood pressure in patients with Alzheimer disease.

Azimi M, Nikanfar M, Khakikhatibi F, Rahbarghazi R, Nourazarian SM, Biray Avci C, Nourazarian A.

J Clin Neurosci. 2017 Sep;43:77-81. doi: 10.1016/j.jocn.2017.04.025. Epub 2017 May 12.

PMID:
28506742
17.

Pathological Role of Peptidyl-Prolyl Isomerase Pin1 in the Disruption of Synaptic Plasticity in Alzheimer's Disease.

Xu L, Ren Z, Chow FE, Tsai R, Liu T, Rizzolio F, Boffo S, Xu Y, Huang S, Lippa CF, Gong Y.

Neural Plast. 2017;2017:3270725. doi: 10.1155/2017/3270725. Epub 2017 Mar 26.

18.

Effects of naturally occurring charged mutations on the structure, stability, and binding of the Pin1 WW domain.

Qiao X, Liu Y, Luo L, Chen L, Zhao C, Ai X.

Biochem Biophys Res Commun. 2017 May 27;487(2):470-476. doi: 10.1016/j.bbrc.2017.04.093. Epub 2017 Apr 19.

PMID:
28431929
19.

A 4-Nitroquinoleneoxide-Induced Pleurotus eryngii Mutant Variety Increases Pin1 Expression in Rat Brain.

Jeong Y, Jung M, Kim MJ, Hwang CH.

J Med Food. 2017 Jan;20(1):65-70. doi: 10.1089/jmf.2016.3809.

PMID:
28098518
20.

Function and regulation of tau conformations in the development and treatment of traumatic brain injury and neurodegeneration.

Albayram O, Herbert MK, Kondo A, Tsai CY, Baxley S, Lian X, Hansen M, Zhou XZ, Lu KP.

Cell Biosci. 2016 Dec 5;6:59. doi: 10.1186/s13578-016-0124-4. eCollection 2016. Review.

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