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Items: 1 to 20 of 54


Alterations in DNA Methylation Status Associated with Gulf War Illness.

Trivedi MS, Abreu MM, Sarria L, Rose N, Ahmed N, Beljanski V, Fletcher MA, Klimas NG, Nathanson L.

DNA Cell Biol. 2019 Jun;38(6):561-571. doi: 10.1089/dna.2018.4469. Epub 2019 Mar 28.


Rates of Chronic Medical Conditions in 1991 Gulf War Veterans Compared to the General Population.

Zundel CG, Krengel MH, Heeren T, Yee MK, Grasso CM, Janulewicz Lloyd PA, Coughlin SS, Sullivan K.

Int J Environ Res Public Health. 2019 Mar 16;16(6). pii: E949. doi: 10.3390/ijerph16060949.


Exploring brain mechanisms underlying Gulf War Illness with group ICA based analysis of fMRI resting state networks.

Gopinath KS, Sakoglu U, Crosson BA, Haley RW.

Neurosci Lett. 2019 May 14;701:136-141. doi: 10.1016/j.neulet.2019.02.041. Epub 2019 Feb 27.


Reproducibility and validity of a novel invasive method of assessing peripheral microvascular vasomotor function.

Kinlay S, Bundy M, Chin M, Tobin D, Quinn M, Do JM, Johnson S, Temiyasathit S, Ly S.

PLoS One. 2019 Jan 25;14(1):e0211152. doi: 10.1371/journal.pone.0211152. eCollection 2019.


Gulf War Illness: Unifying Hypothesis for a Continuing Health Problem.

Mawson AR, Croft AM.

Int J Environ Res Public Health. 2019 Jan 3;16(1). pii: E111. doi: 10.3390/ijerph16010111. Review.


Corticosterone and pyridostigmine/DEET exposure attenuate peripheral cytokine expression: Supporting a dominant role for neuroinflammation in a mouse model of Gulf War Illness.

Michalovicz LT, Locker AR, Kelly KA, Miller JV, Barnes Z, Fletcher MA, Miller DB, Klimas NG, Morris M, Lasley SM, O'Callaghan JP.

Neurotoxicology. 2019 Jan;70:26-32. doi: 10.1016/j.neuro.2018.10.006. Epub 2018 Oct 16.


Dynamic cerebral autoregulation is impaired in Veterans with Gulf War Illness: A case-control study.

Falvo MJ, Lindheimer JB, Serrador JM.

PLoS One. 2018 Oct 15;13(10):e0205393. doi: 10.1371/journal.pone.0205393. eCollection 2018.


Gulf War agents pyridostigmine bromide and permethrin cause hypersensitive nociception that is restored after vagus nerve stimulation.

Nizamutdinov D, Mukherjee S, Deng C, Stauss HM, Shapiro LA.

Neurotoxicology. 2018 Dec;69:93-96. doi: 10.1016/j.neuro.2018.09.007. Epub 2018 Sep 29.


Gulf war illness-related chemicals increase CD11b/c+ monocyte infiltration into the liver and aggravate hepatic cholestasis in a rodent model.

Petrescu AD, Grant S, Frampton G, McMillin M, Kain J, Kodali M, Shetty AK, DeMorrow S.

Sci Rep. 2018 Sep 3;8(1):13147. doi: 10.1038/s41598-018-31599-9.


Oleoylethanolamide treatment reduces neurobehavioral deficits and brain pathology in a mouse model of Gulf War Illness.

Joshi U, Evans JE, Joseph R, Emmerich T, Saltiel N, Lungmus C, Oberlin S, Langlois H, Ojo J, Mouzon B, Paris D, Mullan M, Jin C, Klimas N, Sullivan K, Crawford F, Abdullah L.

Sci Rep. 2018 Aug 27;8(1):12921. doi: 10.1038/s41598-018-31242-7. Erratum in: Sci Rep. 2019 Jul 24;9(1):11011.


Neurotoxicity in acute and repeated organophosphate exposure.

Naughton SX, Terry AV Jr.

Toxicology. 2018 Sep 1;408:101-112. doi: 10.1016/j.tox.2018.08.011. Epub 2018 Aug 23. Review.


Pathophysiology in a model of Gulf War Illness: Contributions of pyridostigmine bromide and stress.

Macht VA, Woodruff JL, Grillo CA, Wood CS, Wilson MA, Reagan LP.

Psychoneuroendocrinology. 2018 Oct;96:195-202. doi: 10.1016/j.psyneuen.2018.07.015. Epub 2018 Jul 10.


Meta-analysis of depleted uranium levels in the Middle East region.

Bešić L, Muhović I, Mrkulić F, Spahić L, Omanović A, Kurtovic-Kozaric A.

J Environ Radioact. 2018 Dec;192:67-74. doi: 10.1016/j.jenvrad.2018.06.004. Epub 2018 Jun 15. Review.


The Neuroinflammatory Phenotype in a Mouse Model of Gulf War Illness is Unrelated to Brain Regional Levels of Acetylcholine as Measured by Quantitative HILIC-UPLC-MS/MS.

Miller JV, LeBouf RF, Kelly KA, Michalovicz LT, Ranpara A, Locker AR, Miller DB, O'Callaghan JP.

Toxicol Sci. 2018 Oct 1;165(2):302-313. doi: 10.1093/toxsci/kfy130.


Behavioral, cellular and molecular maladaptations covary with exposure to pyridostigmine bromide in a rat model of gulf war illness pain.

Cooper BY, Flunker LD, Johnson RD, Nutter TJ.

Toxicol Appl Pharmacol. 2018 Aug 1;352:119-131. doi: 10.1016/j.taap.2018.05.023. Epub 2018 May 24.


Increased butyrate priming in the gut stalls microbiome associated-gastrointestinal inflammation and hepatic metabolic reprogramming in a mouse model of Gulf War Illness.

Seth RK, Kimono D, Alhasson F, Sarkar S, Albadrani M, Lasley SK, Horner R, Janulewicz P, Nagarkatti M, Nagarkatti P, Sullivan K, Chatterjee S.

Toxicol Appl Pharmacol. 2018 Jul 1;350:64-77. doi: 10.1016/j.taap.2018.05.006. Epub 2018 May 9.


Psychoneuroimmunology and Natural Killer Cells: The Chromium-Release Whole-Blood Assay.

Fletcher MA, Barnes Z, Broderick G, Klimas NG.

Methods Mol Biol. 2018;1781:209-220. doi: 10.1007/978-1-4939-7828-1_12.


Exploring the Diagnostic Potential of Immune Biomarker Co-expression in Gulf War Illness.

Broderick G, Fletcher MA, Gallagher M, Barnes Z, Vernon SD, Klimas NG.

Methods Mol Biol. 2018;1781:101-120. doi: 10.1007/978-1-4939-7828-1_7.


Chronic Neurological Morbidities and Elevated Hippocampal Calcium Levels in a DFP-Based Rat Model of Gulf War Illness.

Phillips KF, Deshpande LS.

Mil Med. 2018 Mar 1;183(suppl_1):552-555. doi: 10.1093/milmed/usx148.


Epigenetic impacts of stress priming of the neuroinflammatory response to sarin surrogate in mice: a model of Gulf War illness.

Ashbrook DG, Hing B, Michalovicz LT, Kelly KA, Miller JV, de Vega WC, Miller DB, Broderick G, O'Callaghan JP, McGowan PO.

J Neuroinflammation. 2018 Mar 17;15(1):86. doi: 10.1186/s12974-018-1113-9.

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