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Items: 6

1.

Self-assembly of a nine-residue amyloid-forming peptide fragment of SARS corona virus E-protein: mechanism of self aggregation and amyloid-inhibition of hIAPP.

Ghosh A, Pithadia AS, Bhat J, Bera S, Midya A, Fierke CA, Ramamoorthy A, Bhunia A.

Biochemistry. 2015 Apr 7;54(13):2249-2261. doi: 10.1021/acs.biochem.5b00061. Epub 2015 Mar 24.

2.

Alterations in nerve-evoked bladder contractions in a coronavirus-induced mouse model of multiple sclerosis.

Lamarre NS, Braverman AS, Malykhina AP, Barbe MF, Ruggieri MR Sr.

PLoS One. 2014 Oct 13;9(10):e109314. doi: 10.1371/journal.pone.0109314. eCollection 2014.

3.

Identification of common biological pathways and drug targets across multiple respiratory viruses based on human host gene expression analysis.

Smith SB, Dampier W, Tozeren A, Brown JR, Magid-Slav M.

PLoS One. 2012;7(3):e33174. doi: 10.1371/journal.pone.0033174. Epub 2012 Mar 14.

4.

Neuroprotective effect of apolipoprotein D against human coronavirus OC43-induced encephalitis in mice.

Do Carmo S, Jacomy H, Talbot PJ, Rassart E.

J Neurosci. 2008 Oct 8;28(41):10330-8. doi: 10.1523/JNEUROSCI.2644-08.2008.

5.

Human coronavirus polyadenylated RNA sequences in cerebrospinal fluid from multiple sclerosis patients.

Cristallo A, Gambaro F, Biamonti G, Ferrante P, Battaglia M, Cereda PM.

New Microbiol. 1997 Apr;20(2):105-14.

PMID:
9208420
6.

Cerebrospinal fluid antibodies to coronavirus in patients with Parkinson's disease.

Fazzini E, Fleming J, Fahn S.

Mov Disord. 1992;7(2):153-8.

PMID:
1316552

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