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Items: 6


Self-assembly of a nine-residue amyloid-forming peptide fragment of SARS corona virus E-protein: mechanism of self aggregation and amyloid-inhibition of hIAPP.

Ghosh A, Pithadia AS, Bhat J, Bera S, Midya A, Fierke CA, Ramamoorthy A, Bhunia A.

Biochemistry. 2015 Apr 7;54(13):2249-2261. doi: 10.1021/acs.biochem.5b00061. Epub 2015 Mar 24.


Alterations in nerve-evoked bladder contractions in a coronavirus-induced mouse model of multiple sclerosis.

Lamarre NS, Braverman AS, Malykhina AP, Barbe MF, Ruggieri MR Sr.

PLoS One. 2014 Oct 13;9(10):e109314. doi: 10.1371/journal.pone.0109314. eCollection 2014.


Identification of common biological pathways and drug targets across multiple respiratory viruses based on human host gene expression analysis.

Smith SB, Dampier W, Tozeren A, Brown JR, Magid-Slav M.

PLoS One. 2012;7(3):e33174. doi: 10.1371/journal.pone.0033174. Epub 2012 Mar 14.


Neuroprotective effect of apolipoprotein D against human coronavirus OC43-induced encephalitis in mice.

Do Carmo S, Jacomy H, Talbot PJ, Rassart E.

J Neurosci. 2008 Oct 8;28(41):10330-8. doi: 10.1523/JNEUROSCI.2644-08.2008.


Human coronavirus polyadenylated RNA sequences in cerebrospinal fluid from multiple sclerosis patients.

Cristallo A, Gambaro F, Biamonti G, Ferrante P, Battaglia M, Cereda PM.

New Microbiol. 1997 Apr;20(2):105-14.


Cerebrospinal fluid antibodies to coronavirus in patients with Parkinson's disease.

Fazzini E, Fleming J, Fahn S.

Mov Disord. 1992;7(2):153-8.


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