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Best matches for hepatotoxicity AND kava AND united states food and drug administration:

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Items: 5


Measuring the chemical and cytotoxic variability of commercially available kava (Piper methysticum G. Forster).

Martin AC, Johnston E, Xing C, Hegeman AD.

PLoS One. 2014 Nov 3;9(11):e111572. doi: 10.1371/journal.pone.0111572. eCollection 2014.


Phototoxicity of kava - formation of reactive oxygen species leading to lipid peroxidation and DNA damage.

Xia Q, Chiang HM, Zhou YT, Yin JJ, Liu F, Wang C, Guo L, Fu PP.

Am J Chin Med. 2012;40(6):1271-88. doi: 10.1142/S0192415X12500942.


Liver toxicity and carcinogenicity in F344/N rats and B6C3F1 mice exposed to Kava Kava.

Behl M, Nyska A, Chhabra RS, Travlos GS, Fomby LM, Sparrow BR, Hejtmancik MR, Chan PC.

Food Chem Toxicol. 2011 Nov;49(11):2820-9. doi: 10.1016/j.fct.2011.07.067. Epub 2011 Aug 18.


Risk of kava hepatotoxicity and the FDA consumer advisory.

Teschke R, Schulze J.

JAMA. 2010 Nov 17;304(19):2174-5. doi: 10.1001/jama.2010.1689. No abstract available.


Flavokawain B, the hepatotoxic constituent from kava root, induces GSH-sensitive oxidative stress through modulation of IKK/NF-kappaB and MAPK signaling pathways.

Zhou P, Gross S, Liu JH, Yu BY, Feng LL, Nolta J, Sharma V, Piwnica-Worms D, Qiu SX.

FASEB J. 2010 Dec;24(12):4722-32. doi: 10.1096/fj.10-163311. Epub 2010 Aug 9.

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