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Items: 14

1.

Differential activation of immune/inflammatory response-related co-expression modules in the hippocampus across the major psychiatric disorders.

Kim S, Hwang Y, Webster MJ, Lee D.

Mol Psychiatry. 2016 Mar;21(3):376-85. doi: 10.1038/mp.2015.79.

PMID:
26077692
2.

Chemokine receptors and cortical interneuron dysfunction in schizophrenia.

Volk DW, Chitrapu A, Edelson JR, Lewis DA.

Schizophr Res. 2015 Sep;167(1-3):12-7. doi: 10.1016/j.schres.2014.10.031.

3.

Cortical inhibitory neuron disturbances in schizophrenia: role of the ontogenetic transcription factor Lhx6.

Volk DW, Edelson JR, Lewis DA.

Schizophr Bull. 2014 Sep;40(5):1053-61. doi: 10.1093/schbul/sbu068.

4.

Altered cortical expression of GABA-related genes in schizophrenia: illness progression vs developmental disturbance.

Hoftman GD, Volk DW, Bazmi HH, Li S, Sampson AR, Lewis DA.

Schizophr Bull. 2015 Jan;41(1):180-91. doi: 10.1093/schbul/sbt178.

5.

Role of glutamic acid decarboxylase 67 in regulating cortical parvalbumin and GABA membrane transporter 1 expression: implications for schizophrenia.

Curley AA, Eggan SM, Lazarus MS, Huang ZJ, Volk DW, Lewis DA.

Neurobiol Dis. 2013 Feb;50:179-86. doi: 10.1016/j.nbd.2012.10.018.

6.

Deficits in transcriptional regulators of cortical parvalbumin neurons in schizophrenia.

Volk DW, Matsubara T, Li S, Sengupta EJ, Georgiev D, Minabe Y, Sampson A, Hashimoto T, Lewis DA.

Am J Psychiatry. 2012 Oct;169(10):1082-91.

7.

Hippocampal interneurons are abnormal in schizophrenia.

Konradi C, Yang CK, Zimmerman EI, Lohmann KM, Gresch P, Pantazopoulos H, Berretta S, Heckers S.

Schizophr Res. 2011 Sep;131(1-3):165-73. doi: 10.1016/j.schres.2011.06.007.

8.

Cortical deficits of glutamic acid decarboxylase 67 expression in schizophrenia: clinical, protein, and cell type-specific features.

Curley AA, Arion D, Volk DW, Asafu-Adjei JK, Sampson AR, Fish KN, Lewis DA.

Am J Psychiatry. 2011 Sep;168(9):921-9. doi: 10.1176/appi.ajp.2011.11010052.

9.

Expression of parvalbumin and glutamic acid decarboxylase-67 after acute administration of MK-801. Implications for the NMDA hypofunction model of schizophrenia.

Romón T, Mengod G, Adell A.

Psychopharmacology (Berl). 2011 Sep;217(2):231-8. doi: 10.1007/s00213-011-2268-6.

PMID:
21465242
10.
11.

Postnatal NMDA receptor ablation in corticolimbic interneurons confers schizophrenia-like phenotypes.

Belforte JE, Zsiros V, Sklar ER, Jiang Z, Yu G, Li Y, Quinlan EM, Nakazawa K.

Nat Neurosci. 2010 Jan;13(1):76-83. doi: 10.1038/nn.2447.

12.

A neonatal ventral hippocampal lesion causes functional deficits in adult prefrontal cortical interneurons.

Tseng KY, Lewis BL, Hashimoto T, Sesack SR, Kloc M, Lewis DA, O'Donnell P.

J Neurosci. 2008 Nov 26;28(48):12691-9. doi: 10.1523/JNEUROSCI.4166-08.2008.

14.

Gene expression deficits in a subclass of GABA neurons in the prefrontal cortex of subjects with schizophrenia.

Hashimoto T, Volk DW, Eggan SM, Mirnics K, Pierri JN, Sun Z, Sampson AR, Lewis DA.

J Neurosci. 2003 Jul 16;23(15):6315-26.

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