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Protein misassembly and aggregation as potential convergence points for non-genetic causes of chronic mental illness.

Bradshaw NJ, Korth C.

Mol Psychiatry. 2018 Aug 8. doi: 10.1038/s41380-018-0133-2. [Epub ahead of print] Review.


Aggregation of the protein TRIOBP-1 and its potential relevance to schizophrenia.

Bradshaw NJ, Bader V, Prikulis I, Lueking A, Müllner S, Korth C.

PLoS One. 2014 Oct 21;9(10):e111196. doi: 10.1371/journal.pone.0111196. eCollection 2014.


Mice lacking collapsin response mediator protein 1 manifest hyperactivity, impaired learning and memory, and impaired prepulse inhibition.

Yamashita N, Takahashi A, Takao K, Yamamoto T, Kolattukudy P, Miyakawa T, Goshima Y.

Front Behav Neurosci. 2013 Dec 27;7:216. doi: 10.3389/fnbeh.2013.00216. eCollection 2013.


Proteomic, genomic and translational approaches identify CRMP1 for a role in schizophrenia and its underlying traits.

Bader V, Tomppo L, Trossbach SV, Bradshaw NJ, Prikulis I, Leliveld SR, Lin CY, Ishizuka K, Sawa A, Ramos A, Rosa I, García Á, Requena JR, Hipolito M, Rai N, Nwulia E, Henning U, Ferrea S, Luckhaus C, Ekelund J, Veijola J, Järvelin MR, Hennah W, Korth C.

Hum Mol Genet. 2012 Oct 15;21(20):4406-18. Epub 2012 Jul 13.


Disease-specific alterations in frontal cortex brain proteins in schizophrenia, bipolar disorder, and major depressive disorder. The Stanley Neuropathology Consortium.

Johnston-Wilson NL, Sims CD, Hofmann JP, Anderson L, Shore AD, Torrey EF, Yolken RH.

Mol Psychiatry. 2000 Mar;5(2):142-9.


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