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Items: 1 to 20 of 40

1.

Novel gene-brain structure relationships in psychotic disorder revealed using parallel independent component analyses.

Tandon N, Nanda P, Padmanabhan JL, Mathew IT, Eack SM, Narayanan B, Meda SA, Bergen SE, Ruaño G, Windemuth A, Kocherla M, Petryshen TL, Clementz B, Sweeney J, Tamminga C, Pearlson G, Keshavan MS.

Schizophr Res. 2017 Apr;182:74-83. doi: 10.1016/j.schres.2016.10.026. Epub 2016 Oct 24.

PMID:
27789186
2.

Genome-wide association study of behavioural and psychiatric features in human prion disease.

Thompson AG, Uphill J, Lowe J, Porter MC, Lukic A, Carswell C, Rudge P, MacKay A, Collinge J, Mead S.

Transl Psychiatry. 2015 Apr 21;5:e552. doi: 10.1038/tp.2015.42.

3.

Genomewide association analyses of electrophysiological endophenotypes for schizophrenia and psychotic bipolar disorders: a preliminary report.

Hall MH, Chen CY, Cohen BM, Spencer KM, Levy DL, Öngür D, Smoller JW.

Am J Med Genet B Neuropsychiatr Genet. 2015 Apr;168B(3):151-61. doi: 10.1002/ajmg.b.32298. Epub 2015 Mar 4.

4.

Variability in working memory performance explained by epistasis vs polygenic scores in the ZNF804A pathway.

Nicodemus KK, Hargreaves A, Morris D, Anney R, Gill M, Corvin A, Donohoe G; Schizophrenia Psychiatric Genome-wide Association Study (GWAS) Consortium; Wellcome Trust Case Control Consortium 2.

JAMA Psychiatry. 2014 Jul 1;71(7):778-85. doi: 10.1001/jamapsychiatry.2014.528.

5.

Are genetic risk factors for psychosis also associated with dimension-specific psychotic experiences in adolescence?

Sieradzka D, Power RA, Freeman D, Cardno AG, McGuire P, Plomin R, Meaburn EL, Dudbridge F, Ronald A.

PLoS One. 2014 Apr 9;9(4):e94398. doi: 10.1371/journal.pone.0094398. eCollection 2014.

6.

Analysis of genetic association and epistasis interactions between circadian clock genes and symptom dimensions of bipolar affective disorder.

Maciukiewicz M, Dmitrzak-Weglarz M, Pawlak J, Leszczynska-Rodziewicz A, Zaremba D, Skibinska M, Hauser J.

Chronobiol Int. 2014 Jul;31(6):770-8. doi: 10.3109/07420528.2014.899244. Epub 2014 Mar 27.

PMID:
24673294
7.

Neurophysiologic effect of GWAS derived schizophrenia and bipolar risk variants.

Hall MH, Levy DL, Salisbury DF, Haddad S, Gallagher P, Lohan M, Cohen B, Ongür D, Smoller JW.

Am J Med Genet B Neuropsychiatr Genet. 2014 Jan;165B(1):9-18. doi: 10.1002/ajmg.b.32212. Epub 2013 Nov 8.

8.

Genetic association study between the detected risk variants based upon type II diabetes GWAS and psychotic disorders in the Japanese population.

Kajio Y, Kondo K, Saito T, Iwayama Y, Aleksic B, Yamada K, Toyota T, Hattori E, Ujike H, Inada T, Kunugi H, Kato T, Yoshikawa T, Ozaki N, Ikeda M, Iwata N.

J Hum Genet. 2014 Jan;59(1):54-6. doi: 10.1038/jhg.2013.116. Epub 2013 Nov 7.

PMID:
24196380
9.

A genome-wide association analysis of a broad psychosis phenotype identifies three loci for further investigation.

Psychosis Endophenotypes International Consortium; Wellcome Trust Case-Control Consortium 2, Bramon E, Pirinen M, Strange A, Lin K, Freeman C, Bellenguez C, Su Z, Band G, Pearson R, Vukcevic D, Langford C, Deloukas P, Hunt S, Gray E, Dronov S, Potter SC, Tashakkori-Ghanbaria A, Edkins S, Bumpstead SJ, Arranz MJ, Bakker S, Bender S, Bruggeman R, Cahn W, Chandler D, Collier DA, Crespo-Facorro B, Dazzan P, de Haan L, Di Forti M, Dragović M, Giegling I, Hall J, Iyegbe C, Jablensky A, Kahn RS, Kalaydjieva L, Kravariti E, Lawrie S, Linszen DH, Mata I, McDonald C, McIntosh A, Myin-Germeys I, Ophoff RA, Pariante CM, Paunio T, Picchioni M; Psychiatric Genomics Consortium, Ripke S, Rujescu D, Sauer H, Shaikh M, Sussmann J, Suvisaari J, Tosato S, Toulopoulou T, Van Os J, Walshe M, Weisbrod M, Whalley H, Wiersma D, Blackwell JM, Brown MA, Casas JP, Corvin A, Duncanson A, Jankowski JA, Markus HS, Mathew CG, Palmer CN, Plomin R, Rautanen A, Sawcer SJ, Trembath RC, Wood NW, Barroso I, Peltonen L, Lewis CM, Murray RM, Donnelly P, Powell J, Spencer CC.

Biol Psychiatry. 2014 Mar 1;75(5):386-97. doi: 10.1016/j.biopsych.2013.03.033. Epub 2013 Jul 17.

10.

Detection of phenotype modifier genes using two-locus linkage analysis in complex disorders such as major psychosis.

Bureau A, Croteau J, Mérette C, Fournier A, Chagnon YC, Roy MA, Maziade M.

Hum Hered. 2012;73(4):195-207. doi: 10.1159/000341392. Epub 2012 Aug 15.

PMID:
22907187
11.

Polygenic dissection of the bipolar phenotype.

Hamshere ML, O'Donovan MC, Jones IR, Jones L, Kirov G, Green EK, Moskvina V, Grozeva D, Bass N, McQuillin A, Gurling H, St Clair D, Young AH, Ferrier IN, Farmer A, McGuffin P, Sklar P, Purcell S, Holmans PA, Owen MJ, Craddock N.

Br J Psychiatry. 2011 Apr;198(4):284-8. doi: 10.1192/bjp.bp.110.087866.

12.

ANK3, CACNA1C and ZNF804A gene variants in bipolar disorders and psychosis subphenotype.

Lett TA, Zai CC, Tiwari AK, Shaikh SA, Likhodi O, Kennedy JL, Müller DJ.

World J Biol Psychiatry. 2011 Aug;12(5):392-7. doi: 10.3109/15622975.2011.564655. Epub 2011 Jul 18.

PMID:
21767209
13.

Genome-wide association analysis of age at onset and psychotic symptoms in bipolar disorder.

Belmonte Mahon P, Pirooznia M, Goes FS, Seifuddin F, Steele J, Lee PH, Huang J, Hamshere ML; Bipolar Genome Study (BiGS) Consortium, The Wellcome Trust Case Control Consortium Bipolar Disorder Group, Depaulo JR Jr, Kelsoe JR, Rietschel M, Nöthen M, Cichon S, Gurling H, Purcell S, Smoller JW, Craddock N, Schulze TG, McMahon FJ, Potash JB, Zandi PP.

Am J Med Genet B Neuropsychiatr Genet. 2011 Apr;156B(3):370-8. doi: 10.1002/ajmg.b.31172. Epub 2011 Feb 8. Erratum in: Am J Med Genet B Neuropsychiatr Genet. 2011 Sep;156B(6):749-50.

14.

Evaluation of risk loci for schizophrenia derived from genome-wide association studies in a German population.

Schanze D, Ekici AB, Gawlik M, Pfuhlmann B, Reis A, Stöber G.

Am J Med Genet B Neuropsychiatr Genet. 2011 Mar;156(2):198-203. doi: 10.1002/ajmg.b.31156. Epub 2011 Jan 13.

PMID:
21302348
15.

ZNF804A may be associated with executive control of attention.

Balog Z, Kiss I, Kéri S.

Genes Brain Behav. 2011 Mar;10(2):223-7. doi: 10.1111/j.1601-183X.2010.00657.x. Epub 2010 Nov 9.

16.

Psychosis and relapse in bipolar disorder are related to GRM3, DAOA, and GRIN2B genotype.

Dalvie S, Horn N, Nossek C, van der Merwe L, Stein DJ, Ramesar R.

Afr J Psychiatry (Johannesbg). 2010 Sep;13(4):297-301.

PMID:
20957330
17.

The role of CYP2D6 and TaqI A polymorphisms in malignant neuroleptic syndrome: two case reports with three episodes.

Zivković M, Mihaljević-Peles A, Sagud M, Silić A, Mihanović M.

Psychiatr Danub. 2010 Mar;22(1):112-6.

18.

Possible association of different G72/G30 SNPs with mood episodes and persecutory delusions in bipolar I Romanian patients.

Grigoroiu-Serbanescu M, Herms S, Diaconu CC, Jamra RA, Meier S, Bleotu C, Neagu AI, Prelipceanu D, Sima D, Gherghel M, Mihailescu R, Rietschel M, Nöthen MM, Cichon S, Mühleisen TW.

Prog Neuropsychopharmacol Biol Psychiatry. 2010 May 30;34(4):657-63. doi: 10.1016/j.pnpbp.2010.03.008. Epub 2010 Mar 15.

PMID:
20230869
19.

Epigenetic mediation of environmental influences in major psychotic disorders.

Rutten BP, Mill J.

Schizophr Bull. 2009 Nov;35(6):1045-56. doi: 10.1093/schbul/sbp104. Epub 2009 Sep 25. Review.

20.

The bipolar disorder risk allele at CACNA1C also confers risk of recurrent major depression and of schizophrenia.

Green EK, Grozeva D, Jones I, Jones L, Kirov G, Caesar S, Gordon-Smith K, Fraser C, Forty L, Russell E, Hamshere ML, Moskvina V, Nikolov I, Farmer A, McGuffin P; Wellcome Trust Case Control Consortium, Holmans PA, Owen MJ, O'Donovan MC, Craddock N.

Mol Psychiatry. 2010 Oct;15(10):1016-22. doi: 10.1038/mp.2009.49. Epub 2009 Jul 21.

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