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Items: 4


Early start of progressive motor deficits in Line 61 α-synuclein transgenic mice.

Rabl R, Breitschaedel C, Flunkert S, Duller S, Amschl D, Neddens J, Niederkofler V, Rockenstein E, Masliah E, Roemer H, Hutter-Paier B.

BMC Neurosci. 2017 Jan 31;18(1):22. doi: 10.1186/s12868-017-0341-8.


Time course and progression of wild type α-synuclein accumulation in a transgenic mouse model.

Amschl D, Neddens J, Havas D, Flunkert S, Rabl R, Römer H, Rockenstein E, Masliah E, Windisch M, Hutter-Paier B.

BMC Neurosci. 2013 Jan 9;14:6. doi: 10.1186/1471-2202-14-6.


The C-terminal domain of Fcj1 is required for formation of crista junctions and interacts with the TOB/SAM complex in mitochondria.

Körner C, Barrera M, Dukanovic J, Eydt K, Harner M, Rabl R, Vogel F, Rapaport D, Neupert W, Reichert AS.

Mol Biol Cell. 2012 Jun;23(11):2143-55. doi: 10.1091/mbc.E11-10-0831. Epub 2012 Apr 11.


Formation of cristae and crista junctions in mitochondria depends on antagonism between Fcj1 and Su e/g.

Rabl R, Soubannier V, Scholz R, Vogel F, Mendl N, Vasiljev-Neumeyer A, Körner C, Jagasia R, Keil T, Baumeister W, Cyrklaff M, Neupert W, Reichert AS.

J Cell Biol. 2009 Jun 15;185(6):1047-63. doi: 10.1083/jcb.200811099.

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