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Items: 13


TIMP3 is Regulated by Pericytes upon Shear Stress Detection Leading to a Modified Endothelial Cell Response.

Schrimpf C, Koppen T, Duffield JS, Böer U, David S, Ziegler W, Haverich A, Teebken OE, Wilhelmi M.

Eur J Vasc Endovasc Surg. 2017 Oct;54(4):524-533. doi: 10.1016/j.ejvs.2017.07.002. Epub 2017 Aug 12.


Matrix Metalloproteinase-2 Knockout and Heterozygote Mice Are Protected from Hydronephrosis and Kidney Fibrosis after Unilateral Ureteral Obstruction.

Tveitarås MK, Skogstrand T, Leh S, Helle F, Iversen BM, Chatziantoniou C, Reed RK, Hultström M.

PLoS One. 2015 Dec 16;10(12):e0143390. doi: 10.1371/journal.pone.0143390. eCollection 2015.


[Diagnostic criteria of atypical hemolytic uremic syndrome].

Kagami S, Okada H, Kaname S, Sato W, Nangaku M, Yasuda T, Hattori M, Ashida A, Hataya H, Hidaka Y, Sawai T, Fujimaru R, Fujimura Y, Yoshida Y.

Nihon Jinzo Gakkai Shi. 2013;55(2):91-3. Japanese. No abstract available.


Activated pericytes and the inhibition of renal vascular stability: obstacles for kidney repair.

Basile DP, Sutton TA.

J Am Soc Nephrol. 2012 May;23(5):767-9. doi: 10.1681/ASN.2012030282. Epub 2012 Apr 12. No abstract available.


Pericyte TIMP3 and ADAMTS1 modulate vascular stability after kidney injury.

Schrimpf C, Xin C, Campanholle G, Gill SE, Stallcup W, Lin SL, Davis GE, Gharib SA, Humphreys BD, Duffield JS.

J Am Soc Nephrol. 2012 May;23(5):868-83. doi: 10.1681/ASN.2011080851. Epub 2012 Mar 1.


Increased expression of HIF-1alpha, VEGF-A and its receptors, MMP-2, TIMP-1, and ADAMTS-1 at the venous stenosis of arteriovenous fistula in a mouse model with renal insufficiency.

Misra S, Shergill U, Yang B, Janardhanan R, Misra KD.

J Vasc Interv Radiol. 2010 Aug;21(8):1255-61. doi: 10.1016/j.jvir.2010.02.043. Epub 2010 Jul 3.


HNF4 alpha orchestrates a set of 14 genes to down-regulate cell proliferation in kidney cells.

Grigo K, Wirsing A, Lucas B, Klein-Hitpass L, Ryffel GU.

Biol Chem. 2008 Feb;389(2):179-87. doi: 10.1515/BC.2008.011.


Amino acid regions 572-579 and 657-666 of the spacer domain of ADAMTS13 provide a common antigenic core required for binding of antibodies in patients with acquired TTP.

Luken BM, Turenhout EA, Kaijen PH, Greuter MJ, Pos W, van Mourik JA, Fijnheer R, Voorberg J.

Thromb Haemost. 2006 Sep;96(3):295-301.


ADAMTS1 interacts with, cleaves, and modifies the extracellular location of the matrix inhibitor tissue factor pathway inhibitor-2.

Torres-Collado AX, Kisiel W, Iruela-Arispe ML, Rodríguez-Manzaneque JC.

J Biol Chem. 2006 Jun 30;281(26):17827-37. Epub 2006 Apr 26.


[Molecular basis of hydronephrosis].

Hayasaka K, Ogino D, Matsunaga A.

Nihon Rinsho. 2006 Feb;64 Suppl 2:545-9. Review. Japanese. No abstract available.


Neonatal calyceal dilation and renal fibrosis resulting from loss of Adamts-1 in mouse kidney is due to a developmental dysgenesis.

Mittaz L, Ricardo S, Martinez G, Kola I, Kelly DJ, Little MH, Hertzog PJ, Pritchard MA.

Nephrol Dial Transplant. 2005 Feb;20(2):419-23. Epub 2004 Dec 22.


A disintegrin and metalloproteinase with thrombospondin motifs (ADAMTS)-1 null mutant mice develop renal lesions mimicking obstructive nephropathy.

Yokoyama H, Wada T, Kobayashi K, Kuno K, Kurihara H, Shindo T, Matsushima K.

Nephrol Dial Transplant. 2002;17 Suppl 9:39-41.


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