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Items: 1 to 20 of 341

1.

Targeting Polyamine Oxidase to Prevent Excitotoxicity-Induced Retinal Neurodegeneration.

Pichavaram P, Palani CD, Patel C, Xu Z, Shosha E, Fouda AY, Caldwell RB, Narayanan SP.

Front Neurosci. 2019 Jan 10;12:956. doi: 10.3389/fnins.2018.00956. eCollection 2018.

2.

Hydroxyurea attenuates oxidative, metabolic, and excitotoxic stress in rat hippocampal neurons and improves spatial memory in a mouse model of Alzheimer's disease.

Brose RD, Lehrmann E, Zhang Y, Reeves RH, Smith KD, Mattson MP.

Neurobiol Aging. 2018 Dec;72:121-133. doi: 10.1016/j.neurobiolaging.2018.08.021. Epub 2018 Aug 29.

PMID:
30245242
3.

NeuroEPO Preserves Neurons from Glutamate-Induced Excitotoxicity.

Garzón F, Coimbra D, Parcerisas A, Rodriguez Y, García JC, Soriano E, Rama R.

J Alzheimers Dis. 2018;65(4):1469-1483. doi: 10.3233/JAD-180668.

PMID:
30175978
4.

Monitoring of glutamate-induced excitotoxicity by mitochondrial oxygen consumption.

Kumagai A, Sasaki T, Matsuoka K, Abe M, Tabata T, Itoh Y, Fuchino H, Wugangerile S, Suga M, Yamaguchi T, Kawahara H, Nagaoka Y, Kawabata K, Furue MK, Takemori H.

Synapse. 2019 Jan;73(1):e22067. doi: 10.1002/syn.22067. Epub 2018 Sep 6.

PMID:
30120794
5.

CYP46A1 protects against NMDA-mediated excitotoxicity in Huntington's disease: Analysis of lipid raft content.

Boussicault L, Kacher R, Lamazière A, Vanhoutte P, Caboche J, Betuing S, Potier MC.

Biochimie. 2018 Oct;153:70-79. doi: 10.1016/j.biochi.2018.07.019. Epub 2018 Aug 11.

PMID:
30107216
6.
7.

Glutamatergic nervous system degeneration in a C. elegans TauA152T tauopathy model involves pathways of excitotoxicity and Ca2+ dysregulation.

Choudhary B, Mandelkow E, Mandelkow EM, Pir GJ.

Neurobiol Dis. 2018 Sep;117:189-202. doi: 10.1016/j.nbd.2018.06.005. Epub 2018 Jun 9.

PMID:
29894752
8.

Functional up-regulation of the M-current by retigabine contrasts hyperexcitability and excitotoxicity on rat hypoglossal motoneurons.

Ghezzi F, Monni L, Nistri A.

J Physiol. 2018 Jul;596(13):2611-2629. doi: 10.1113/JP275906. Epub 2018 May 30.

PMID:
29736957
9.

Elevated MeCP2 in Mice Causes Neurodegeneration Involving Tau Dysregulation and Excitotoxicity: Implications for the Understanding and Treatment of MeCP2 Triplication Syndrome.

Montgomery KR, Louis Sam Titus ASC, Wang L, D'Mello SR.

Mol Neurobiol. 2018 Dec;55(12):9057-9074. doi: 10.1007/s12035-018-1046-4. Epub 2018 Apr 10.

PMID:
29637441
10.

Mesenchymal Stem Cell Protection of Neurons against Glutamate Excitotoxicity Involves Reduction of NMDA-Triggered Calcium Responses and Surface GluR1, and Is Partly Mediated by TNF.

Papazian I, Kyrargyri V, Evangelidou M, Voulgari-Kokota A, Probert L.

Int J Mol Sci. 2018 Feb 25;19(3). pii: E651. doi: 10.3390/ijms19030651.

11.

A Novel Tetramethylpyrazine Derivative Prophylactically Protects against Glutamate-Induced Excitotoxicity in Primary Neurons through the Blockage of N-Methyl-D-aspartate Receptor.

Hu S, Hu H, Mak S, Cui G, Lee M, Shan L, Wang Y, Lin H, Zhang Z, Han Y.

Front Pharmacol. 2018 Feb 12;9:73. doi: 10.3389/fphar.2018.00073. eCollection 2018.

12.

Excitotoxicity in the pathogenesis of neurological and psychiatric disorders: Therapeutic implications.

Olloquequi J, Cornejo-Córdova E, Verdaguer E, Soriano FX, Binvignat O, Auladell C, Camins A.

J Psychopharmacol. 2018 Mar;32(3):265-275. doi: 10.1177/0269881118754680. Epub 2018 Feb 15.

PMID:
29444621
13.

Activation of caspase-6 and cleavage of caspase-6 substrates is an early event in NMDA receptor-mediated excitotoxicity.

Girling KD, Demers MJ, Laine J, Zhang S, Wang YT, Graham RK.

J Neurosci Res. 2018 Mar;96(3):391-406. doi: 10.1002/jnr.24153. Epub 2017 Nov 29.

PMID:
29193273
14.

Propofol Protects Rat Hypoglossal Motoneurons in an In Vitro Model of Excitotoxicity by Boosting GABAergic Inhibition and Reducing Oxidative Stress.

Ghezzi F, Monni L, Corsini S, Rauti R, Nistri A.

Neuroscience. 2017 Dec 26;367:15-33. doi: 10.1016/j.neuroscience.2017.10.019. Epub 2017 Oct 22.

PMID:
29069620
15.

Synaptic Activity Protects Neurons Against Calcium-Mediated Oxidation and Contraction of Mitochondria During Excitotoxicity.

Depp C, Bas-Orth C, Schroeder L, Hellwig A, Bading H.

Antioxid Redox Signal. 2018 Oct 20;29(12):1109-1124. doi: 10.1089/ars.2017.7092. Epub 2017 Nov 14.

PMID:
28990420
16.

Olfactory function in an excitotoxic model for secondary neuronal degeneration: Role of dopaminergic interneurons.

Marin C, Laxe S, Langdon C, Berenguer J, Lehrer E, Mariño-Sánchez F, Alobid I, Bernabeu M, Mullol J.

Neuroscience. 2017 Nov 19;364:28-44. doi: 10.1016/j.neuroscience.2017.09.008. Epub 2017 Sep 14.

PMID:
28918258
17.

Tau exacerbates excitotoxic brain damage in an animal model of stroke.

Bi M, Gladbach A, van Eersel J, Ittner A, Przybyla M, van Hummel A, Chua SW, van der Hoven J, Lee WS, Müller J, Parmar J, Jonquieres GV, Stefen H, Guccione E, Fath T, Housley GD, Klugmann M, Ke YD, Ittner LM.

Nat Commun. 2017 Sep 7;8(1):473. doi: 10.1038/s41467-017-00618-0.

18.

Calpain-Dependent Degradation of Nucleoporins Contributes to Motor Neuron Death in a Mouse Model of Chronic Excitotoxicity.

Sugiyama K, Aida T, Nomura M, Takayanagi R, Zeilhofer HU, Tanaka K.

J Neurosci. 2017 Sep 6;37(36):8830-8844. doi: 10.1523/JNEUROSCI.0730-17.2017. Epub 2017 Aug 16.

19.

Phosphorylation of tau at Y18, but not tau-fyn binding, is required for tau to modulate NMDA receptor-dependent excitotoxicity in primary neuronal culture.

Miyamoto T, Stein L, Thomas R, Djukic B, Taneja P, Knox J, Vossel K, Mucke L.

Mol Neurodegener. 2017 May 19;12(1):41. doi: 10.1186/s13024-017-0176-x.

20.

Ecdysterones from Rhaponticum carthamoides (Willd.) Iljin reduce hippocampal excitotoxic cell loss and upregulate mTOR signaling in rats.

Wu J, Gao L, Shang L, Wang G, Wei N, Chu T, Chen S, Zhang Y, Huang J, Wang J, Lin R.

Fitoterapia. 2017 Jun;119:158-167. doi: 10.1016/j.fitote.2017.03.015. Epub 2017 Apr 2.

PMID:
28373010

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