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Items: 1 to 20 of 124

1.

Carbo-Iron as improvement of the nanoiron technology: From laboratory design to the field test.

Mackenzie K, Bleyl S, Kopinke FD, Doose H, Bruns J.

Sci Total Environ. 2016 Sep 1;563-564:641-8. doi: 10.1016/j.scitotenv.2015.07.107. Epub 2015 Aug 20.

PMID:
26299641
2.

SCN1A mutation analysis in myoclonic astatic epilepsy and severe idiopathic generalized epilepsy of infancy with generalized tonic-clonic seizures.

Ebach K, Joos H, Doose H, Stephani U, Kurlemann G, Fiedler B, Hahn A, Hauser E, Hundt K, Holthausen H, Müller U, Neubauer BA.

Neuropediatrics. 2005 Jun;36(3):210-3.

PMID:
15944908
3.

Myoclonic-astatic epilepsy of early childhood--definition, course, nosography, and genetics.

Neubauer BA, Hahn A, Doose H, Tuxhorn I.

Adv Neurol. 2005;95:147-55. Review. No abstract available.

PMID:
15508920
4.

Clinical spectrum and genetics of Rolandic epilepsy.

Neubauer BA, Hahn A, Stephani U, Doose H.

Adv Neurol. 2002;89:475-9. Review. No abstract available.

PMID:
11968472
5.

Occipital sharp waves in idiopathic partial epilepsies--clinical and genetic aspects.

Doose H, Petersen B, Neubauer BA.

Epilepsy Res. 2002 Jan;48(1-2):121-30.

PMID:
11823116
6.

Atypical "benign" partial epilepsy of childhood or pseudo-lennox syndrome. Part II: family study.

Doose H, Hahn A, Neubauer BA, Pistohl J, Stephani U.

Neuropediatrics. 2001 Feb;32(1):9-13.

PMID:
11315204
7.

The concept of hereditary impairment of brain maturation.

Doose H, Neubauer BA, Petersen B.

Epileptic Disord. 2000;2 Suppl 1:S45-9.

8.

Multifactorial pathogenesis of neonatal seizures--relationships to the benign partial epilepsies.

Doose H, Koudriavtseva K, Neubauer BA.

Epileptic Disord. 2000 Dec;2(4):195-201.

9.
10.

Benign idiopathic partial epilepsy and brain lesion.

Stephani U, Doose H.

Epilepsia. 1999 Mar;40(3):373-6.

11.

Severe idiopathic generalized epilepsy of infancy with generalized tonic-clonic seizures.

Doose H, Lunau H, Castiglione E, Waltz S.

Neuropediatrics. 1998 Oct;29(5):229-38.

PMID:
9810557
12.

Contradictory conclusions about the possible effects of prolonged febrile convulsions.

Doose H.

Epilepsia. 1998 Jan;39(1):108-10. No abstract available.

13.

Children with focal sharp waves: clinical and genetic aspects.

Doose H, Brigger-Heuer B, Neubauer B.

Epilepsia. 1997 Jul;38(7):788-96.

14.

Seizure risk in offspring of individuals with a history of febrile convulsions.

Doose H, Maurer A.

Eur J Pediatr. 1997 Jun;156(6):476-81.

PMID:
9208247
15.

Children with benign focal sharp waves in the EEG--developmental disorders and epilepsy.

Doose H, Neubauer B, Carlsson G.

Neuropediatrics. 1996 Oct;27(5):227-41. Review.

PMID:
8971743
16.

Parental generalized EEG alpha activity predisposes to spike wave discharges in offspring.

Doose H, Castiglione E, Waltz S.

Hum Genet. 1995 Dec;96(6):695-704.

PMID:
8522330
17.

Absence epilepsy of early childhood--genetic aspects.

Doose H.

Eur J Pediatr. 1994 May;153(5):372-7.

PMID:
8033930
18.

Photosensitivity--genetics and clinical significance.

Doose H, Waltz S.

Neuropediatrics. 1993 Oct;24(5):249-55. Review.

PMID:
8309513
19.

Benign childhood epilepsy with centrotemporal spikes and the focal sharp wave trait is not linked to the fragile X region.

Rees M, Diebold U, Parker K, Doose H, Gardiner RM, Whitehouse WP.

Neuropediatrics. 1993 Aug;24(4):211-3.

PMID:
8232779
20.

Exclusion of linkage of genetic focal sharp waves to the HLA region on chromosome 6p in families with benign partial epilepsy with centrotemporal sharp waves.

Whitehouse W, Diebold U, Rees M, Parker K, Doose H, Gardiner RM.

Neuropediatrics. 1993 Aug;24(4):208-10.

PMID:
8232778

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