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Items: 7

1.

Dexras1, a small GTPase, is required for glutamate-NMDA neurotoxicity.

Chen Y, Khan RS, Cwanger A, Song Y, Steenstra C, Bang S, Cheah JH, Dunaief J, Shindler KS, Snyder SH, Kim SF.

J Neurosci. 2013 Feb 20;33(8):3582-7. doi: 10.1523/JNEUROSCI.1497-12.2013.

2.

Resetting the clock: Dexras1 defines a path.

Van Gelder RN.

Neuron. 2004 Sep 2;43(5):603-4. Review.

3.

Manganese superoxide dismutase protects nNOS neurons from NMDA and nitric oxide-mediated neurotoxicity.

Gonzalez-Zulueta M, Ensz LM, Mukhina G, Lebovitz RM, Zwacka RM, Engelhardt JF, Oberley LW, Dawson VL, Dawson TM.

J Neurosci. 1998 Mar 15;18(6):2040-55. Review.

4.

Molecular basis of glutamate toxicity in retinal ganglion cells.

Sucher NJ, Lipton SA, Dreyer EB.

Vision Res. 1997 Dec;37(24):3483-93. Review.

5.

A proposed role for excitotoxicity in glaucoma.

Dreyer EB.

J Glaucoma. 1998 Feb;7(1):62-7. Review. No abstract available.

PMID:
9493118
6.

Dexras1: shaping the responsiveness of the circadian clock.

Cheng HY, Obrietan K.

Semin Cell Dev Biol. 2006 Jun;17(3):345-51. Epub 2006 Apr 3. Review.

PMID:
16765612
7.

Small G Proteins Dexras1 and RHES and Their Role in Pathophysiological Processes.

Thapliyal A, Verma R, Kumar N.

Int J Cell Biol. 2014;2014:308535. doi: 10.1155/2014/308535. Epub 2014 Mar 20. Review.

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