Related Articles by Review for PMID: 23047475

Year	Number of Results
2008	1
2009	1
2011	1
2012	3
2013	2
2015	2
2016	1
2017	2
2019	2
2020	2
2021	1
2022	2
2023	2
2024	1

1

BCR-ABL1 kinase inhibits uracil DNA glycosylase UNG2 to enhance oxidative DNA damage and stimulate genomic instability.

Slupianek A, Falinski R, Znojek P, Stoklosa T, Flis S, Doneddu V, Pytel D, Synowiec E, Blasiak J, Bellacosa A, Skorski T. Slupianek A, et al. Leukemia. 2013 Mar;27(3):629-34. doi: 10.1038/leu.2012.294. Epub 2012 Oct 9. Leukemia. 2013. PMID: 23047475 Free PMC article.

2

Chronic myeloid leukemia cells refractory/resistant to tyrosine kinase inhibitors are genetically unstable and may cause relapse and malignant progression to the terminal disease state.

Skorski T. Skorski T. Leuk Lymphoma. 2011 Feb;52 Suppl 1(0 1):23-9. doi: 10.3109/10428194.2010.546912. Leuk Lymphoma. 2011. PMID: 21299457 Free PMC article. Review.

3

Reactive oxygen species in BCR-ABL1-expressing cells - relevance to chronic myeloid leukemia.

Antoszewska-Smith J, Pawlowska E, Blasiak J. Antoszewska-Smith J, et al. Acta Biochim Pol. 2017;64(1):1-10. doi: 10.18388/abp.2016_1396. Epub 2016 Dec 1. Acta Biochim Pol. 2017. PMID: 27904889 Free article. Review.

4

[Recommendations from the French CML Study Group (Fi-LMC) for BCR-ABL1 kinase domain mutation analysis in chronic myeloid leukemia].

Cayuela JM, Chomel JC, Coiteux V, Dulucq S, Escoffre-Barbe M, Etancelin P, Etienne G, Hayette S, Millot F, Nibourel O, Nicolini FE, Réa D; pour la France Intergroupe des leucémies myéloïdes chroniques (Fi-LMC) et le Groupe des biologistes moléculaires des hémopathies malignes (GBMHM). Cayuela JM, et al. Bull Cancer. 2020 Jan;107(1):113-128. doi: 10.1016/j.bulcan.2019.05.011. Epub 2019 Jul 26. Bull Cancer. 2020. PMID: 31353136 Review. French.

5

Molecular biology of bcr-abl1-positive chronic myeloid leukemia.

Quintás-Cardama A, Cortes J. Quintás-Cardama A, et al. Blood. 2009 Feb 19;113(8):1619-30. doi: 10.1182/blood-2008-03-144790. Epub 2008 Sep 30. Blood. 2009. PMID: 18827185 Free PMC article. Review.

6

DNA Repair--A Double-Edged Sword in the Genomic Stability of Cancer Cells--The Case of Chronic Myeloid Leukemia.

Pawlowska E, Blasiak J. Pawlowska E, et al. Int J Mol Sci. 2015 Nov 18;16(11):27535-49. doi: 10.3390/ijms161126049. Int J Mol Sci. 2015. PMID: 26593906 Free PMC article. Review.

7

Beyond BCR::ABL1-The Role of Genomic Analyses in the Management of CML.

Branford S, Fernandes A, Shahrin N, Maqsood M, Shanmuganathan N, Wadham C. Branford S, et al. J Natl Compr Canc Netw. 2024 Feb;22(1):e237335. doi: 10.6004/jnccn.2023.7335. J Natl Compr Canc Netw. 2024. PMID: 38394774 Review.

8

Genetic mechanisms of chronic myeloid leukemia blastic transformation.

Skorski T. Skorski T. Curr Hematol Malig Rep. 2012 Jun;7(2):87-93. doi: 10.1007/s11899-012-0114-5. Curr Hematol Malig Rep. 2012. PMID: 22328017 Review.

9

The specificity of asciminib, a potential treatment for chronic myeloid leukemia, as a myristate-pocket binding ABL inhibitor and analysis of its interactions with mutant forms of BCR-ABL1 kinase.

Manley PW, Barys L, Cowan-Jacob SW. Manley PW, et al. Leuk Res. 2020 Nov;98:106458. doi: 10.1016/j.leukres.2020.106458. Epub 2020 Sep 29. Leuk Res. 2020. PMID: 33096322 Review.

10

Chronic myeloid leukemia: clinical impact of BCR-ABL1 mutations and other lesions associated with disease progression.

Ernst T, Hochhaus A. Ernst T, et al. Semin Oncol. 2012 Feb;39(1):58-66. doi: 10.1053/j.seminoncol.2011.11.002. Semin Oncol. 2012. PMID: 22289492 Review.

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