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Items: 7

1.

Unidirectional cross-activation of GRPR by MOR1D uncouples itch and analgesia induced by opioids.

Liu XY, Liu ZC, Sun YG, Ross M, Kim S, Tsai FF, Li QF, Jeffry J, Kim JY, Loh HH, Chen ZF.

Cell. 2011 Oct 14;147(2):447-58. doi: 10.1016/j.cell.2011.08.043.

2.

Neural processing of itch.

Akiyama T, Carstens E.

Neuroscience. 2013 Oct 10;250:697-714. doi: 10.1016/j.neuroscience.2013.07.035. Epub 2013 Jul 24. Review.

3.

The mechanism of μ-opioid receptor (MOR)-TRPV1 crosstalk in TRPV1 activation involves morphine anti-nociception, tolerance and dependence.

Bao Y, Gao Y, Yang L, Kong X, Yu J, Hou W, Hua B.

Channels (Austin). 2015;9(5):235-43. doi: 10.1080/19336950.2015.1069450. Epub 2015 Jul 15. Review.

4.

Direct association of Mu-opioid and NMDA glutamate receptors supports their cross-regulation: molecular implications for opioid tolerance.

Garzón J, Rodríguez-Muñoz M, Sánchez-Blázquez P.

Curr Drug Abuse Rev. 2012 Sep;5(3):199-226. Review.

PMID:
22920535
5.

[Opioid-induced pruritus. Mechanisms and treatment regimens].

Schmelz M.

Anaesthesist. 2009 Jan;58(1):61-5. doi: 10.1007/s00101-008-1478-8. Review. German.

PMID:
19132330
6.

Neuraxial opioid-induced itch and its pharmacological antagonism.

Ko MC.

Handb Exp Pharmacol. 2015;226:315-35. doi: 10.1007/978-3-662-44605-8_17. Review.

7.

Itch signaling in the nervous system.

Jeffry J, Kim S, Chen ZF.

Physiology (Bethesda). 2011 Aug;26(4):286-92. doi: 10.1152/physiol.00007.2011. Review.

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