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Items: 5

1.

Tumor-specific usage of alternative transcription start sites in colorectal cancer identified by genome-wide exon array analysis.

Thorsen K, Schepeler T, Øster B, Rasmussen MH, Vang S, Wang K, Hansen KQ, Lamy P, Pedersen JS, Eller A, Mansilla F, Laurila K, Wiuf C, Laurberg S, Dyrskjøt L, Ørntoft TF, Andersen CL.

BMC Genomics. 2011 Oct 14;12:505. doi: 10.1186/1471-2164-12-505.

2.

Alternative splicing within the Wnt signaling pathway: role in cancer development.

Sumithra B, Saxena U, Das AB.

Cell Oncol (Dordr). 2016 Feb;39(1):1-13. doi: 10.1007/s13402-015-0266-0. Epub 2016 Jan 13. Review.

PMID:
26762488
3.

Splice isoforms as therapeutic targets for colorectal cancer.

Miura K, Fujibuchi W, Unno M.

Carcinogenesis. 2012 Dec;33(12):2311-9. doi: 10.1093/carcin/bgs347. Epub 2012 Nov 1. Review.

PMID:
23118106
4.

TIN: An R Package for Transcriptome Instability Analysis.

Johannessen B, Sveen A, Skotheim RI.

Cancer Inform. 2015 Sep 20;14:109-12. doi: 10.4137/CIN.S31363. eCollection 2015. Review.

5.

What Is the Impact of mRNA 5' TL Heterogeneity on Translational Start Site Selection and the Mammalian Cellular Phenotype?

Curran JA, Weiss B.

Front Genet. 2016 Aug 31;7:156. doi: 10.3389/fgene.2016.00156. eCollection 2016. Review.

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