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Items: 6

1.

Antipsychotic-induced vacuous chewing movements and extrapyramidal side effects are highly heritable in mice.

Crowley JJ, Adkins DE, Pratt AL, Quackenbush CR, van den Oord EJ, Moy SS, Wilhelmsen KC, Cooper TB, Bogue MA, McLeod HL, Sullivan PF.

Pharmacogenomics J. 2012 Apr;12(2):147-55. doi: 10.1038/tpj.2010.82. Epub 2010 Nov 16.

2.

Oxidative stress and the antipsychotic-induced vacuous chewing movement model of tardive dyskinesia: evidence for antioxidant-based prevention strategies.

Lister J, Nobrega JN, Fletcher PJ, Remington G.

Psychopharmacology (Berl). 2014 Jun;231(11):2237-49. doi: 10.1007/s00213-014-3582-6. Epub 2014 Apr 22. Review.

PMID:
24752659
3.

The vacuous chewing movement (VCM) model of tardive dyskinesia revisited: is there a relationship to dopamine D(2) receptor occupancy?

Turrone P, Remington G, Nobrega JN.

Neurosci Biobehav Rev. 2002 May;26(3):361-80. Review.

PMID:
12034136
4.

The opiate antagonist naloxone suppresses a rodent model of tardive dyskinesia.

Stoessl AJ, Polanski E, Frydryszak H.

Mov Disord. 1993 Oct;8(4):445-52. Review.

PMID:
7901759
5.

[Cost-effectiveness analysis of schizophrenic patient care settings: impact of an atypical antipsychotic under long-acting injection formulation].

Llorca PM, Miadi-Fargier H, Lançon C, Jasso Mosqueda G, Casadebaig F, Philippe A, Guillon P, Mehnert A, Omnès LF, Chicoye A, Durand-Zaleski I.

Encephale. 2005 Mar-Apr;31(2):235-46. Review. French.

PMID:
15959450
6.

Relevance of animal models to human tardive dyskinesia.

Blanchet PJ, Parent MT, Rompré PH, Lévesque D.

Behav Brain Funct. 2012 Mar 9;8:12. doi: 10.1186/1744-9081-8-12. Review.

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