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Items: 6


Suppression of inflammation by a synthetic histone mimic.

Nicodeme E, Jeffrey KL, Schaefer U, Beinke S, Dewell S, Chung CW, Chandwani R, Marazzi I, Wilson P, Coste H, White J, Kirilovsky J, Rice CM, Lora JM, Prinjha RK, Lee K, Tarakhovsky A.

Nature. 2010 Dec 23;468(7327):1119-23. doi: 10.1038/nature09589. Epub 2010 Nov 10.


The mechanisms behind the therapeutic activity of BET bromodomain inhibition.

Shi J, Vakoc CR.

Mol Cell. 2014 Jun 5;54(5):728-36. doi: 10.1016/j.molcel.2014.05.016. Review.


Modulation of acetylation: creating a pro-survival and anti-inflammatory phenotype in lethal hemorrhagic and septic shock.

Li Y, Alam HB.

J Biomed Biotechnol. 2011;2011:523481. doi: 10.1155/2011/523481. Epub 2011 Feb 15. Review.


Epigenetics in sepsis: targeting histone deacetylases.

Ciarlo E, Savva A, Roger T.

Int J Antimicrob Agents. 2013 Jun;42 Suppl:S8-12. doi: 10.1016/j.ijantimicag.2013.04.004. Epub 2013 May 9. Review.


Bromodomain and extra-terminal (BET) family proteins: New therapeutic targets in major diseases.

Padmanabhan B, Mathur S, Manjula R, Tripathi S.

J Biosci. 2016 Jun;41(2):295-311. Review.

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