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Items: 6


Internal tandem duplication of FLT3 (FLT3/ITD) induces increased ROS production, DNA damage, and misrepair: implications for poor prognosis in AML.

Sallmyr A, Fan J, Datta K, Kim KT, Grosu D, Shapiro P, Small D, Rassool F.

Blood. 2008 Mar 15;111(6):3173-82. doi: 10.1182/blood-2007-05-092510. Epub 2008 Jan 11.


Genomic instability in myeloid malignancies: increased reactive oxygen species (ROS), DNA double strand breaks (DSBs) and error-prone repair.

Sallmyr A, Fan J, Rassool FV.

Cancer Lett. 2008 Oct 18;270(1):1-9. doi: 10.1016/j.canlet.2008.03.036. Epub 2008 May 7. Review.


NOX-driven ROS formation in cell transformation of FLT3-ITD-positive AML.

Jayavelu AK, Moloney JN, Böhmer FD, Cotter TG.

Exp Hematol. 2016 Dec;44(12):1113-1122. doi: 10.1016/j.exphem.2016.08.008. Epub 2016 Sep 22. Review.


Structural and functional alterations of FLT3 in acute myeloid leukemia.

Meshinchi S, Appelbaum FR.

Clin Cancer Res. 2009 Jul 1;15(13):4263-9. doi: 10.1158/1078-0432.CCR-08-1123. Epub 2009 Jun 23. Review.


Sorafenib in combination with low-dose-homoharringtonine as a salvage therapy in primary refractory FLT3-ITD-positive AML: a case report and review of literature.

Xu G, Mao L, Liu H, Yang M, Jin J, Qian W.

Int J Clin Exp Med. 2015 Nov 15;8(11):19891-4. eCollection 2015. Review.


Excessive Reactive Oxygen Species and Exotic DNA Lesions as an Exploitable Liability.

AbdulSalam SF, Thowfeik FS, Merino EJ.

Biochemistry. 2016 Sep 27;55(38):5341-52. doi: 10.1021/acs.biochem.6b00703. Epub 2016 Sep 13. Review.

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