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Items: 1 to 20 of 24

1.

Abeta oligomer-induced aberrations in synapse composition, shape, and density provide a molecular basis for loss of connectivity in Alzheimer's disease.

Lacor PN, Buniel MC, Furlow PW, Clemente AS, Velasco PT, Wood M, Viola KL, Klein WL.

J Neurosci. 2007 Jan 24;27(4):796-807.

2.

Why Alzheimer's is a disease of memory: the attack on synapses by A beta oligomers (ADDLs).

Viola KL, Velasco PT, Klein WL.

J Nutr Health Aging. 2008 Jan;12(1):51S-7S. Review.

PMID:
18165846
3.

Synaptic changes in Alzheimer's disease and its models.

Pozueta J, Lefort R, Shelanski ML.

Neuroscience. 2013 Oct 22;251:51-65. doi: 10.1016/j.neuroscience.2012.05.050. Epub 2012 Jun 9. Review.

PMID:
22687952
4.

Synapses and dendritic spines as pathogenic targets in Alzheimer's disease.

Yu W, Lu B.

Neural Plast. 2012;2012:247150. doi: 10.1155/2012/247150. Epub 2012 Feb 6. Review.

5.

Amyloid beta: a putative intra-spinal microtubule-depolymerizer to induce synapse-loss or dentritic spine shortening in Alzheimer's disease.

Mitsuyama F, Futatsugi Y, Okuya M, Karagiozov K, Peev N, Kato Y, Kanno T, Sano H, Koide T.

Ital J Anat Embryol. 2009 Apr-Sep;114(2-3):109-20. Review.

PMID:
20198823
6.

Pathogenesis of Abeta oligomers in synaptic failure.

Sivanesan S, Tan A, Rajadas J.

Curr Alzheimer Res. 2013 Mar;10(3):316-23. Review.

PMID:
23036017
7.

Amyloid β oligomers in Alzheimer's disease pathogenesis, treatment, and diagnosis.

Viola KL, Klein WL.

Acta Neuropathol. 2015 Feb;129(2):183-206. doi: 10.1007/s00401-015-1386-3. Epub 2015 Jan 22. Review.

8.
9.

Aβ oligomer-induced synapse degeneration in Alzheimer's disease.

Wilcox KC, Lacor PN, Pitt J, Klein WL.

Cell Mol Neurobiol. 2011 Aug;31(6):939-48. doi: 10.1007/s10571-011-9691-4. Epub 2011 May 3. Review.

10.

The Aβ oligomer hypothesis for synapse failure and memory loss in Alzheimer's disease.

Ferreira ST, Klein WL.

Neurobiol Learn Mem. 2011 Nov;96(4):529-43. doi: 10.1016/j.nlm.2011.08.003. Epub 2011 Sep 6. Review.

11.

Impaired regulation of synaptic actin cytoskeleton in Alzheimer's disease.

Penzes P, Vanleeuwen JE.

Brain Res Rev. 2011 Jun 24;67(1-2):184-92. doi: 10.1016/j.brainresrev.2011.01.003. Epub 2011 Jan 26. Review.

12.

Beta-amyloid oligomers affect the structure and function of the postsynaptic region: role of the Wnt signaling pathway.

Dinamarca MC, Colombres M, Cerpa W, Bonansco C, Inestrosa NC.

Neurodegener Dis. 2008;5(3-4):149-52. doi: 10.1159/000113687. Epub 2008 Mar 6. Review.

PMID:
18322375
13.
14.

The inside-out amyloid hypothesis and synapse pathology in Alzheimer's disease.

Gouras GK, Willén K, Faideau M.

Neurodegener Dis. 2014;13(2-3):142-6. doi: 10.1159/000354776. Epub 2013 Sep 24. Review.

PMID:
24080821
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17.

Synapse pathology in psychiatric and neurologic disease.

van Spronsen M, Hoogenraad CC.

Curr Neurol Neurosci Rep. 2010 May;10(3):207-14. doi: 10.1007/s11910-010-0104-8. Review.

18.

Alzheimer's disease-related alterations in synaptic density: neocortex and hippocampus.

Scheff SW, Price DA.

J Alzheimers Dis. 2006;9(3 Suppl):101-15. Review.

PMID:
16914849
19.

Pathogenesis of synaptic degeneration in Alzheimer's disease and Lewy body disease.

Overk CR, Masliah E.

Biochem Pharmacol. 2014 Apr 15;88(4):508-16. doi: 10.1016/j.bcp.2014.01.015. Epub 2014 Jan 21. Review.

20.

Analyzing dendritic spine pathology in Alzheimer's disease: problems and opportunities.

Dorostkar MM, Zou C, Blazquez-Llorca L, Herms J.

Acta Neuropathol. 2015 Jul;130(1):1-19. doi: 10.1007/s00401-015-1449-5. Epub 2015 Jun 11. Review.

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