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Items: 6


ABL tyrosine kinases: evolution of function, regulation, and specificity.

Colicelli J.

Sci Signal. 2010 Sep 14;3(139):re6. doi: 10.1126/scisignal.3139re6. Review. Erratum in: Sci Signal. 2011 Aug 30;4(188):er4.


Bcr-Abl induces abnormal cytoskeleton remodeling, beta1 integrin clustering and increased cell adhesion to fibronectin through the Abl interactor 1 pathway.

Li Y, Clough N, Sun X, Yu W, Abbott BL, Hogan CJ, Dai Z.

J Cell Sci. 2007 Apr 15;120(Pt 8):1436-46. Epub 2007 Mar 27.


ABI2-deficient mice exhibit defective cell migration, aberrant dendritic spine morphogenesis, and deficits in learning and memory.

Grove M, Demyanenko G, Echarri A, Zipfel PA, Quiroz ME, Rodriguiz RM, Playford M, Martensen SA, Robinson MR, Wetsel WC, Maness PF, Pendergast AM.

Mol Cell Biol. 2004 Dec;24(24):10905-22.


Oncogenic Abl and Src tyrosine kinases elicit the ubiquitin-dependent degradation of target proteins through a Ras-independent pathway.

Dai Z, Quackenbush RC, Courtney KD, Grove M, Cortez D, Reuther GW, Pendergast AM.

Genes Dev. 1998 May 15;12(10):1415-24.

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