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Items: 15


Experimental treatment of antipsychotic-induced movement disorders.

Shireen E.

J Exp Pharmacol. 2016 Aug 8;8:1-10. doi: 10.2147/JEP.S63553. eCollection 2016. Review.


Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo.

Fisher JR, Wallace CE, Tripoli DL, Sheline YI, Cirrito JR.

Mol Neurodegener. 2016 Jun 18;11(1):45. doi: 10.1186/s13024-016-0112-5.


Effects of oral and intravenous administration of buspirone on food-cocaine choice in socially housed male cynomolgus monkeys.

Czoty PW, Nader MA.

Neuropsychopharmacology. 2015 Mar 13;40(5):1072-83. doi: 10.1038/npp.2014.300.


Pinpointing brainstem mechanisms responsible for autonomic dysfunction in Rett syndrome: therapeutic perspectives for 5-HT1A agonists.

Abdala AP, Bissonnette JM, Newman-Tancredi A.

Front Physiol. 2014 May 30;5:205. doi: 10.3389/fphys.2014.00205. eCollection 2014.


Efficacy of buspirone for attenuating cocaine and methamphetamine reinstatement in rats.

Shelton KL, Hendrick ES, Beardsley PM.

Drug Alcohol Depend. 2013 May 1;129(3):210-6. doi: 10.1016/j.drugalcdep.2013.01.003. Epub 2013 Jan 29.


The sigma-1 antagonist BMY-14802 inhibits L-DOPA-induced abnormal involuntary movements by a WAY-100635-sensitive mechanism.

Paquette MA, Foley K, Brudney EG, Meshul CK, Johnson SW, Berger SP.

Psychopharmacology (Berl). 2009 Jul;204(4):743-54. doi: 10.1007/s00213-009-1505-8. Epub 2009 Mar 13. Erratum in: Psychopharmacology (Berl). 2009 May;204(1):185-7.


The stimulus effects of 8-OH-DPAT: evidence for a 5-HT2A receptor-mediated component.

Reissig CJ, Eckler JR, Rabin RA, Rice KC, Winter JC.

Pharmacol Biochem Behav. 2008 Jan;88(3):312-7. Epub 2007 Sep 19.


Needs and opportunities for innovation in psychopharmacology.

Stahl SM.

J R Soc Med. 1987 Jul;80(7):413-7. No abstract available.


The potential anxiolytic activity of GR38032F, a 5-HT3-receptor antagonist.

Jones BJ, Costall B, Domeney AM, Kelly ME, Naylor RJ, Oakley NR, Tyers MB.

Br J Pharmacol. 1988 Apr;93(4):985-93.


Behavioural evidence for a functional interaction between central 5-HT2 and 5-HT1A receptors.

Backus LI, Sharp T, Grahame-Smith DG.

Br J Pharmacol. 1990 Aug;100(4):793-9.


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