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Items: 1 to 20 of 36

1.

BDNF regains function in hippocampal long-term potentiation deficits caused by diencephalic damage.

Vedder LC, Savage LM.

Learn Mem. 2017 Jan 17;24(2):81-85. doi: 10.1101/lm.043927.116.

PMID:
28096497
2.

Identification and characterization of PPARα ligands in the hippocampus.

Roy A, Kundu M, Jana M, Mishra RK, Yung Y, Luan CH, Gonzalez FJ, Pahan K.

Nat Chem Biol. 2016 Dec;12(12):1075-1083. doi: 10.1038/nchembio.2204.

3.
4.

Calcium activated adenylyl cyclase AC8 but not AC1 is required for prolonged behavioral anxiety.

Bernabucci M, Zhuo M.

Mol Brain. 2016 May 27;9(1):60. doi: 10.1186/s13041-016-0239-x.

5.

Characterization of a Novel Chromatin Sorting Tool Reveals Importance of Histone Variant H3.3 in Contextual Fear Memory and Motor Learning.

McNally AG, Poplawski SG, Mayweather BA, White KM, Abel T.

Front Mol Neurosci. 2016 Feb 9;9:11. doi: 10.3389/fnmol.2016.00011.

6.

Multitasking: Effects of processing multiple auditory feature patterns.

Miller T, Chen S, Lee WW, Sussman ES.

Psychophysiology. 2015 Sep;52(9):1140-8. doi: 10.1111/psyp.12446.

7.

The dendritic SNARE fusion machinery involved in AMPARs insertion during long-term potentiation.

Jurado S.

Front Cell Neurosci. 2014 Dec 22;8:407. doi: 10.3389/fncel.2014.00407. Review.

8.

Growth hormone, insulin-like growth factor-1 and the aging brain.

Ashpole NM, Sanders JE, Hodges EL, Yan H, Sonntag WE.

Exp Gerontol. 2015 Aug;68:76-81. doi: 10.1016/j.exger.2014.10.002. Review.

9.

Combination of methamphetamine and HIV-1 gp120 causes distinct long-term alterations of behavior, gene expression, and injury in the central nervous system.

Hoefer MM, Sanchez AB, Maung R, de Rozieres CM, Catalan IC, Dowling CC, Thaney VE, Piña-Crespo J, Zhang D, Roberts AJ, Kaul M.

Exp Neurol. 2015 Jan;263:221-34. doi: 10.1016/j.expneurol.2014.09.010.

10.

Repeated mild traumatic brain injury causes chronic neuroinflammation, changes in hippocampal synaptic plasticity, and associated cognitive deficits.

Aungst SL, Kabadi SV, Thompson SM, Stoica BA, Faden AI.

J Cereb Blood Flow Metab. 2014 Jul;34(7):1223-32. doi: 10.1038/jcbfm.2014.75.

11.

Excitatory amino acid transporters: roles in glutamatergic neurotransmission.

Divito CB, Underhill SM.

Neurochem Int. 2014 Jul;73:172-80. doi: 10.1016/j.neuint.2013.12.008. Review.

12.
13.

RasGRF2 Rac-GEF activity couples NMDA receptor calcium flux to enhanced synaptic transmission.

Schwechter B, Rosenmund C, Tolias KF.

Proc Natl Acad Sci U S A. 2013 Aug 27;110(35):14462-7. doi: 10.1073/pnas.1304340110.

14.

The roles of STP and LTP in synaptic encoding.

Volianskis A, Collingridge GL, Jensen MS.

PeerJ. 2013 Feb 12;1:e3. doi: 10.7717/peerj.3.

15.

Different NMDA receptor subtypes mediate induction of long-term potentiation and two forms of short-term potentiation at CA1 synapses in rat hippocampus in vitro.

Volianskis A, Bannister N, Collett VJ, Irvine MW, Monaghan DT, Fitzjohn SM, Jensen MS, Jane DE, Collingridge GL.

J Physiol. 2013 Feb 15;591(4):955-72. doi: 10.1113/jphysiol.2012.247296.

16.

CLC-3 chloride channels moderate long-term potentiation at Schaffer collateral-CA1 synapses.

Farmer LM, Le BN, Nelson DJ.

J Physiol. 2013 Feb 15;591(4):1001-15. doi: 10.1113/jphysiol.2012.243485.

17.

Mechanisms of specificity in neuronal activity-regulated gene transcription.

Lyons MR, West AE.

Prog Neurobiol. 2011 Aug;94(3):259-95. doi: 10.1016/j.pneurobio.2011.05.003. Review.

18.

D-cycloserine facilitates socially reinforced learning in an animal model relevant to autism spectrum disorders.

Modi ME, Young LJ.

Biol Psychiatry. 2011 Aug 1;70(3):298-304. doi: 10.1016/j.biopsych.2011.01.026.

19.

The effects of NR2 subunit-dependent NMDA receptor kinetics on synaptic transmission and CaMKII activation.

Santucci DM, Raghavachari S.

PLoS Comput Biol. 2008 Oct;4(10):e1000208. doi: 10.1371/journal.pcbi.1000208.

20.

A positive feedback signal transduction loop determines timing of cerebellar long-term depression.

Tanaka K, Augustine GJ.

Neuron. 2008 Aug 28;59(4):608-20. doi: 10.1016/j.neuron.2008.06.026.

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