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Items: 1 to 20 of 22

1.

Manipulation of the Host Cell Membrane during Plasmodium Liver Stage Egress.

Burda PC, Caldelari R, Heussler VT.

MBio. 2017 Apr 11;8(2). pii: e00139-17. doi: 10.1128/mBio.00139-17.

2.

Parasitophorous vacuole poration precedes its rupture and rapid host erythrocyte cytoskeleton collapse in Plasmodium falciparum egress.

Hale VL, Watermeyer JM, Hackett F, Vizcay-Barrena G, van Ooij C, Thomas JA, Spink MC, Harkiolaki M, Duke E, Fleck RA, Blackman MJ, Saibil HR.

Proc Natl Acad Sci U S A. 2017 Mar 28;114(13):3439-3444. doi: 10.1073/pnas.1619441114. Epub 2017 Mar 14.

3.

Inhibition of an Erythrocyte Tyrosine Kinase with Imatinib Prevents Plasmodium falciparum Egress and Terminates Parasitemia.

Kesely KR, Pantaleo A, Turrini FM, Olupot-Olupot P, Low PS.

PLoS One. 2016 Oct 21;11(10):e0164895. doi: 10.1371/journal.pone.0164895. eCollection 2016.

4.

Reversible host cell remodeling underpins deformability changes in malaria parasite sexual blood stages.

Dearnley M, Chu T, Zhang Y, Looker O, Huang C, Klonis N, Yeoman J, Kenny S, Arora M, Osborne JM, Chandramohanadas R, Zhang S, Dixon MW, Tilley L.

Proc Natl Acad Sci U S A. 2016 Apr 26;113(17):4800-5. doi: 10.1073/pnas.1520194113. Epub 2016 Apr 11.

5.

Expanding the antimalarial toolkit: Targeting host-parasite interactions.

Langhorne J, Duffy PE.

J Exp Med. 2016 Feb 8;213(2):143-53. doi: 10.1084/jem.20151677. Epub 2016 Feb 1. Review.

6.

A spiral scaffold underlies cytoadherent knobs in Plasmodium falciparum-infected erythrocytes.

Watermeyer JM, Hale VL, Hackett F, Clare DK, Cutts EE, Vakonakis I, Fleck RA, Blackman MJ, Saibil HR.

Blood. 2016 Jan 21;127(3):343-51. doi: 10.1182/blood-2015-10-674002. Epub 2015 Dec 4.

7.

Dynamic morphology and cytoskeletal protein changes during spontaneous inside-out vesiculation of red blood cell membranes.

Tiffert T, Lew VL.

Pflugers Arch. 2014 Dec;466(12):2279-88. doi: 10.1007/s00424-014-1483-5. Epub 2014 Mar 12.

8.

Thioamide-based fluorescent protease sensors.

Goldberg JM, Chen X, Meinhardt N, Greenbaum DC, Petersson EJ.

J Am Chem Soc. 2014 Feb 5;136(5):2086-93. doi: 10.1021/ja412297x. Epub 2014 Jan 28.

9.

Stretching and relaxation of malaria-infected red blood cells.

Ye T, Phan-Thien N, Khoo BC, Lim CT.

Biophys J. 2013 Sep 3;105(5):1103-9. doi: 10.1016/j.bpj.2013.07.008.

10.

Membrane active chelators as novel anti-African trypanosome and anti-malarial drugs.

Grab DJ, Nenortas E, Bakshi RP, Nikolskaia OV, Friedman JE, Shapiro TA.

Parasitol Int. 2013 Oct;62(5):461-3. doi: 10.1016/j.parint.2013.06.009. Epub 2013 Jun 28.

11.

Recent insights into apicomplexan parasite egress provide new views to a kill.

Blackman MJ, Carruthers VB.

Curr Opin Microbiol. 2013 Aug;16(4):459-64. doi: 10.1016/j.mib.2013.04.008. Epub 2013 May 28. Review.

12.

Probing the cytoadherence of malaria infected red blood cells under flow.

Xu X, Efremov AK, Li A, Lai L, Dao M, Lim CT, Cao J.

PLoS One. 2013 May 28;8(5):e64763. doi: 10.1371/journal.pone.0064763. Print 2013.

13.

Malaria-infected erythrocyte-derived microvesicles mediate cellular communication within the parasite population and with the host immune system.

Mantel PY, Hoang AN, Goldowitz I, Potashnikova D, Hamza B, Vorobjev I, Ghiran I, Toner M, Irimia D, Ivanov AR, Barteneva N, Marti M.

Cell Host Microbe. 2013 May 15;13(5):521-34. doi: 10.1016/j.chom.2013.04.009.

14.

Life cycle-dependent cytoskeletal modifications in Plasmodium falciparum infected erythrocytes.

Shi H, Liu Z, Li A, Yin J, Chong AG, Tan KS, Zhang Y, Lim CT.

PLoS One. 2013 Apr 9;8(4):e61170. doi: 10.1371/journal.pone.0061170. Print 2013.

15.

Cytoplasmic free Ca2+ is essential for multiple steps in malaria parasite egress from infected erythrocytes.

Glushakova S, Lizunov V, Blank PS, Melikov K, Humphrey G, Zimmerberg J.

Malar J. 2013 Jan 30;12:41. doi: 10.1186/1475-2875-12-41.

16.

A host GPCR signaling network required for the cytolysis of infected cells facilitates release of apicomplexan parasites.

Millholland MG, Mishra S, Dupont CD, Love MS, Patel B, Shilling D, Kazanietz MG, Foskett JK, Hunter CA, Sinnis P, Greenbaum DC.

Cell Host Microbe. 2013 Jan 16;13(1):15-28. doi: 10.1016/j.chom.2012.12.001. Epub 2013 Jan 16.

17.

The antimalarial natural product symplostatin 4 is a nanomolar inhibitor of the food vacuole falcipains.

Stolze SC, Deu E, Kaschani F, Li N, Florea BI, Richau KH, Colby T, van der Hoorn RA, Overkleeft HS, Bogyo M, Kaiser M.

Chem Biol. 2012 Dec 21;19(12):1546-55. doi: 10.1016/j.chembiol.2012.09.020.

18.

Red blood cell membrane dynamics during malaria parasite egress.

Callan-Jones A, Albarran Arriagada OE, Massiera G, Lorman V, Abkarian M.

Biophys J. 2012 Dec 19;103(12):2475-83. doi: 10.1016/j.bpj.2012.11.008. Epub 2012 Dec 18.

19.

Prison break: pathogens' strategies to egress from host cells.

Friedrich N, Hagedorn M, Soldati-Favre D, Soldati T.

Microbiol Mol Biol Rev. 2012 Dec;76(4):707-20. doi: 10.1128/MMBR.00024-12. Review.

20.

A novel ENU-mutation in ankyrin-1 disrupts malaria parasite maturation in red blood cells of mice.

Greth A, Lampkin S, Mayura-Guru P, Rodda F, Drysdale K, Roberts-Thomson M, McMorran BJ, Foote SJ, Burgio G.

PLoS One. 2012;7(6):e38999. doi: 10.1371/journal.pone.0038999. Epub 2012 Jun 19.

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