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Items: 1 to 20 of 49


Bik reduces hyperplastic cells by increasing Bak and activating DAPk1 to juxtapose ER and mitochondria.

Mebratu YA, Leyva-Baca I, Wathelet MG, Lacey N, Chand HS, Choi AMK, Tesfaigzi Y.

Nat Commun. 2017 Oct 6;8(1):803. doi: 10.1038/s41467-017-00975-w.


Mitochondria-Associated Membranes As Networking Platforms and Regulators of Cancer Cell Fate.

Sassano ML, van Vliet AR, Agostinis P.

Front Oncol. 2017 Aug 18;7:174. doi: 10.3389/fonc.2017.00174. eCollection 2017. Review.


On BH3 Mimetics and Ca2+ Signaling.

Ferdek PE, Jakubowska MA.

Drug Dev Res. 2017 Sep;78(6):313-318. doi: 10.1002/ddr.21405. Epub 2017 Aug 13.


BCL-2 family: integrating stress responses at the ER to control cell demise.

Pihán P, Carreras-Sureda A, Hetz C.

Cell Death Differ. 2017 Sep;24(9):1478-1487. doi: 10.1038/cdd.2017.82. Epub 2017 Jun 16. Review.


Modulation of Ca2+ Signaling by Anti-apoptotic B-Cell Lymphoma 2 Proteins at the Endoplasmic Reticulum-Mitochondrial Interface.

Vervliet T, Clerix E, Seitaj B, Ivanova H, Monaco G, Bultynck G.

Front Oncol. 2017 May 3;7:75. doi: 10.3389/fonc.2017.00075. eCollection 2017. Review.


Smooth Muscle Ion Channels and Regulation of Vascular Tone in Resistance Arteries and Arterioles.

Tykocki NR, Boerman EM, Jackson WF.

Compr Physiol. 2017 Mar 16;7(2):485-581. doi: 10.1002/cphy.c160011. Review.


Non-apoptotic functions of BCL-2 family proteins.

Gross A, Katz SG.

Cell Death Differ. 2017 Aug;24(8):1348-1358. doi: 10.1038/cdd.2017.22. Epub 2017 Feb 24. Review.


The trans-membrane domain of Bcl-2α, but not its hydrophobic cleft, is a critical determinant for efficient IP3 receptor inhibition.

Ivanova H, Ritaine A, Wagner L, Luyten T, Shapovalov G, Welkenhuyzen K, Seitaj B, Monaco G, De Smedt H, Prevarskaya N, Yule DI, Parys JB, Bultynck G.

Oncotarget. 2016 Aug 23;7(34):55704-55720. doi: 10.18632/oncotarget.11005.


Oncogenic and oncosuppressive signal transduction at mitochondria-associated endoplasmic reticulum membranes.

Marchi S, Giorgi C, Oparka M, Duszynski J, Wieckowski MR, Pinton P.

Mol Cell Oncol. 2014 Oct 29;1(2):e956469. doi: 10.4161/23723548.2014.956469. eCollection 2014 Apr-Jun. Review.


Impact of intracellular ion channels on cancer development and progression.

Peruzzo R, Biasutto L, Szabò I, Leanza L.

Eur Biophys J. 2016 Oct;45(7):685-707. Epub 2016 Jun 11. Review.


Biphasic regulation of InsP3 receptor gating by dual Ca2+ release channel BH3-like domains mediates Bcl-xL control of cell viability.

Yang J, Vais H, Gu W, Foskett JK.

Proc Natl Acad Sci U S A. 2016 Mar 29;113(13):E1953-62. doi: 10.1073/pnas.1517935113. Epub 2016 Mar 14.


Inositol 1,4,5-trisphosphate receptors and their protein partners as signalling hubs.

Prole DL, Taylor CW.

J Physiol. 2016 Jun 1;594(11):2849-66. doi: 10.1113/JP271139. Epub 2016 Feb 24. Review.


Mcl-1 involvement in mitochondrial dynamics is associated with apoptotic cell death.

Morciano G, Giorgi C, Balestra D, Marchi S, Perrone D, Pinotti M, Pinton P.

Mol Biol Cell. 2016 Jan 1;27(1):20-34. doi: 10.1091/mbc.E15-01-0028. Epub 2015 Nov 4.


Bcl-2 proteins, cell migration and embryonic development: lessons from zebrafish.

Prudent J, Popgeorgiev N, Bonneau B, Gillet G.

Cell Death Dis. 2015 Oct 15;6:e1910. doi: 10.1038/cddis.2015.286. No abstract available.


Mitochondria and endoplasmic reticulum crosstalk in amyotrophic lateral sclerosis.

Manfredi G, Kawamata H.

Neurobiol Dis. 2016 Jun;90:35-42. doi: 10.1016/j.nbd.2015.08.004. Epub 2015 Aug 15. Review.


p53 and Ca(2+) signaling from the endoplasmic reticulum: partners in anti-cancer therapies.

Bittremieux M, Bultynck G.

Oncoscience. 2015 Mar 7;2(3):233-8. eCollection 2015.


The BH4 domain of anti-apoptotic Bcl-XL, but not that of the related Bcl-2, limits the voltage-dependent anion channel 1 (VDAC1)-mediated transfer of pro-apoptotic Ca2+ signals to mitochondria.

Monaco G, Decrock E, Arbel N, van Vliet AR, La Rovere RM, De Smedt H, Parys JB, Agostinis P, Leybaert L, Shoshan-Barmatz V, Bultynck G.

J Biol Chem. 2015 Apr 3;290(14):9150-61. doi: 10.1074/jbc.M114.622514. Epub 2015 Feb 13.


Mcl-1 promotes lung cancer cell migration by directly interacting with VDAC to increase mitochondrial Ca2+ uptake and reactive oxygen species generation.

Huang H, Shah K, Bradbury NA, Li C, White C.

Cell Death Dis. 2014 Oct 23;5:e1482. doi: 10.1038/cddis.2014.419.

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