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Items: 7

1.

Mdm2 Phosphorylation Regulates Its Stability and Has Contrasting Effects on Oncogene and Radiation-Induced Tumorigenesis.

Carr MI, Roderick JE, Gannon HS, Kelliher MA, Jones SN.

Cell Rep. 2016 Sep 6;16(10):2618-29. doi: 10.1016/j.celrep.2016.08.014. Epub 2016 Aug 25.

2.

DNA repair synthesis and ligation affect the processing of excised oligonucleotides generated by human nucleotide excision repair.

Kemp MG, Gaddameedhi S, Choi JH, Hu J, Sancar A.

J Biol Chem. 2014 Sep 19;289(38):26574-83. doi: 10.1074/jbc.M114.597088. Epub 2014 Aug 8.

3.

Mutant TP53 posttranslational modifications: challenges and opportunities.

Nguyen TA, Menendez D, Resnick MA, Anderson CW.

Hum Mutat. 2014 Jun;35(6):738-55. doi: 10.1002/humu.22506. Epub 2014 Feb 11. Review.

4.

ATM phosphorylation of Mdm2 Ser394 regulates the amplitude and duration of the DNA damage response in mice.

Gannon HS, Woda BA, Jones SN.

Cancer Cell. 2012 May 15;21(5):668-79. doi: 10.1016/j.ccr.2012.04.011.

5.

Loss of p53 Ser18 and Atm results in embryonic lethality without cooperation in tumorigenesis.

Armata HL, Shroff P, Garlick DE, Penta K, Tapper AR, Sluss HK.

PLoS One. 2011;6(9):e24813. doi: 10.1371/journal.pone.0024813. Epub 2011 Sep 27.

6.

Apoptosis is the essential target of selective pressure against p53, whereas loss of additional p53 functions facilitates carcinoma progression.

Lu X, Yang C, Yin C, Van Dyke T, Simin K.

Mol Cancer Res. 2011 Apr;9(4):430-9. doi: 10.1158/1541-7786.MCR-10-0277. Epub 2011 Mar 8.

7.

Hexavalent chromium-induced apoptosis of granulosa cells involves selective sub-cellular translocation of Bcl-2 members, ERK1/2 and p53.

Banu SK, Stanley JA, Lee J, Stephen SD, Arosh JA, Hoyer PB, Burghardt RC.

Toxicol Appl Pharmacol. 2011 Mar 15;251(3):253-66. doi: 10.1016/j.taap.2011.01.011. Epub 2011 Jan 22.

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