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Items: 1 to 20 of 138

1.

Prion infectivity is encoded exclusively within the structure of proteinase K-resistant fragments of synthetically generated recombinant PrPSc.

Wang F, Wang X, Abskharon R, Ma J.

Acta Neuropathol Commun. 2018 Apr 24;6(1):30. doi: 10.1186/s40478-018-0534-0.

2.

Pharmacological Agents Targeting the Cellular Prion Protein.

Barreca ML, Iraci N, Biggi S, Cecchetti V, Biasini E.

Pathogens. 2018 Mar 7;7(1). pii: E27. doi: 10.3390/pathogens7010027. Review.

3.

Prion Strains and Transmission Barrier Phenomena.

Igel-Egalon A, Béringue V, Rezaei H, Sibille P.

Pathogens. 2018 Jan 1;7(1). pii: E5. doi: 10.3390/pathogens7010005. Review.

4.

Formation and properties of amyloid fibrils of prion protein.

Yamaguchi KI, Kuwata K.

Biophys Rev. 2018 Apr;10(2):517-525. doi: 10.1007/s12551-017-0377-0. Epub 2017 Dec 4. Review.

5.

Amyloid polymorphisms constitute distinct clouds of conformational variants in different etiological subtypes of Alzheimer's disease.

Rasmussen J, Mahler J, Beschorner N, Kaeser SA, Häsler LM, Baumann F, Nyström S, Portelius E, Blennow K, Lashley T, Fox NC, Sepulveda-Falla D, Glatzel M, Oblak AL, Ghetti B, Nilsson KPR, Hammarström P, Staufenbiel M, Walker LC, Jucker M.

Proc Natl Acad Sci U S A. 2017 Dec 5;114(49):13018-13023. doi: 10.1073/pnas.1713215114. Epub 2017 Nov 20.

PMID:
29158413
6.

Multistep Conformation Selection in Amyloid Assembly.

Hsieh MC, Liang C, Mehta AK, Lynn DG, Grover MA.

J Am Chem Soc. 2017 Nov 29;139(47):17007-17010. doi: 10.1021/jacs.7b09362. Epub 2017 Nov 15.

7.

A dominant-negative mutant inhibits multiple prion variants through a common mechanism.

Pei F, DiSalvo S, Sindi SS, Serio TR.

PLoS Genet. 2017 Oct 30;13(10):e1007085. doi: 10.1371/journal.pgen.1007085. eCollection 2017 Oct.

8.

Reduced Abundance and Subverted Functions of Proteins in Prion-Like Diseases: Gained Functions Fascinate but Lost Functions Affect Aetiology.

Allison WT, DuVal MG, Nguyen-Phuoc K, Leighton PLA.

Int J Mol Sci. 2017 Oct 24;18(10). pii: E2223. doi: 10.3390/ijms18102223. Review.

9.

Species-dependent structural polymorphism of Y145Stop prion protein amyloid revealed by solid-state NMR spectroscopy.

Theint T, Nadaud PS, Aucoin D, Helmus JJ, Pondaven SP, Surewicz K, Surewicz WK, Jaroniec CP.

Nat Commun. 2017 Sep 29;8(1):753. doi: 10.1038/s41467-017-00794-z.

10.

Insights into Mechanisms of Transmission and Pathogenesis from Transgenic Mouse Models of Prion Diseases.

Moreno JA, Telling GC.

Methods Mol Biol. 2017;1658:219-252. doi: 10.1007/978-1-4939-7244-9_16. Review.

11.

Amyloidogenesis of Tau protein.

Nizynski B, Dzwolak W, Nieznanski K.

Protein Sci. 2017 Nov;26(11):2126-2150. doi: 10.1002/pro.3275. Epub 2017 Sep 13. Review.

PMID:
28833749
12.

An antipsychotic drug exerts anti-prion effects by altering the localization of the cellular prion protein.

Stincardini C, Massignan T, Biggi S, Elezgarai SR, Sangiovanni V, Vanni I, Pancher M, Adami V, Moreno J, Stravalaci M, Maietta G, Gobbi M, Negro A, Requena JR, Castilla J, Nonno R, Biasini E.

PLoS One. 2017 Aug 7;12(8):e0182589. doi: 10.1371/journal.pone.0182589. eCollection 2017.

13.

Prion strains depend on different endocytic routes for productive infection.

Fehlinger A, Wolf H, Hossinger A, Duernberger Y, Pleschka C, Riemschoss K, Liu S, Bester R, Paulsen L, Priola SA, Groschup MH, Schätzl HM, Vorberg IM.

Sci Rep. 2017 Jul 31;7(1):6923. doi: 10.1038/s41598-017-07260-2.

14.

The mechanism of monomer transfer between two structurally distinct PrP oligomers.

Armiento A, Moireau P, Martin D, Lepejova N, Doumic M, Rezaei H.

PLoS One. 2017 Jul 26;12(7):e0180538. doi: 10.1371/journal.pone.0180538. eCollection 2017.

15.

Self-propagating, protease-resistant, recombinant prion protein conformers with or without in vivo pathogenicity.

Wang F, Wang X, Orrú CD, Groveman BR, Surewicz K, Abskharon R, Imamura M, Yokoyama T, Kim YS, Vander Stel KJ, Sinniah K, Priola SA, Surewicz WK, Caughey B, Ma J.

PLoS Pathog. 2017 Jul 12;13(7):e1006491. doi: 10.1371/journal.ppat.1006491. eCollection 2017 Jul.

16.

Implications of peptide assemblies in amyloid diseases.

Ke PC, Sani MA, Ding F, Kakinen A, Javed I, Separovic F, Davis TP, Mezzenga R.

Chem Soc Rev. 2017 Oct 30;46(21):6492-6531. doi: 10.1039/c7cs00372b. Review.

PMID:
28702523
17.

Prion strains in mammals: Different conformations leading to disease.

Morales R.

PLoS Pathog. 2017 Jul 6;13(7):e1006323. doi: 10.1371/journal.ppat.1006323. eCollection 2017 Jul. Review. No abstract available.

18.

Types and Strains: Their Essential Role in Understanding Protein Aggregation in Neurodegenerative Diseases.

Wemheuer WM, Wrede A, Schulz-Schaeffer WJ.

Front Aging Neurosci. 2017 Jun 16;9:187. doi: 10.3389/fnagi.2017.00187. eCollection 2017. Review.

19.

Molecular Mechanisms of Chronic Wasting Disease Prion Propagation.

Moreno JA, Telling GC.

Cold Spring Harb Perspect Med. 2017 Feb 13. pii: a024448. doi: 10.1101/cshperspect.a024448. [Epub ahead of print]

PMID:
28193766
20.

Divergent prion strain evolution driven by PrPC expression level in transgenic mice.

Le Dur A, Laï TL, Stinnakre MG, Laisné A, Chenais N, Rakotobe S, Passet B, Reine F, Soulier S, Herzog L, Tilly G, Rézaei H, Béringue V, Vilotte JL, Laude H.

Nat Commun. 2017 Jan 23;8:14170. doi: 10.1038/ncomms14170.

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