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Items: 1 to 20 of 21


Microbial Characterization of Qatari Barchan Sand Dunes.

Abdul Majid S, Graw MF, Chatziefthimiou AD, Nguyen H, Richer R, Louge M, Sultan AA, Schloss P, Hay AG.

PLoS One. 2016 Sep 21;11(9):e0161836. doi: 10.1371/journal.pone.0161836. eCollection 2016.


Dietary exposure to an environmental toxin triggers neurofibrillary tangles and amyloid deposits in the brain.

Cox PA, Davis DA, Mash DC, Metcalf JS, Banack SA.

Proc Biol Sci. 2016 Jan 27;283(1823). pii: 20152397. doi: 10.1098/rspb.2015.2397.


Expression of microRNAs in human post-mortem amyotrophic lateral sclerosis spinal cords provides insight into disease mechanisms.

Figueroa-Romero C, Hur J, Lunn JS, Paez-Colasante X, Bender DE, Yung R, Sakowski SA, Feldman EL.

Mol Cell Neurosci. 2016 Mar;71:34-45. doi: 10.1016/j.mcn.2015.12.008. Epub 2015 Dec 17.


Acute β-N-Methylamino-L-alanine Toxicity in a Mouse Model.

Al-Sammak MA, Rogers DG, Hoagland KD.

J Toxicol. 2015;2015:739746. doi: 10.1155/2015/739746. Epub 2015 Oct 29.


Military Service and Amyotrophic Lateral Sclerosis in a Population-based Cohort.

Weisskopf MG, Cudkowicz ME, Johnson N.

Epidemiology. 2015 Nov;26(6):831-8. doi: 10.1097/EDE.0000000000000376.


Biotransfer of β-N-methylamino-L-alanine (BMAA) in a eutrophicated freshwater lake.

Lage S, Annadotter H, Rasmussen U, Rydberg S.

Mar Drugs. 2015 Mar 2;13(3):1185-201. doi: 10.3390/md13031185.


Detection of cyanotoxins, β-N-methylamino-L-alanine and microcystins, from a lake surrounded by cases of amyotrophic lateral sclerosis.

Banack SA, Caller T, Henegan P, Haney J, Murby A, Metcalf JS, Powell J, Cox PA, Stommel E.

Toxins (Basel). 2015 Jan 29;7(2):322-36. doi: 10.3390/toxins7020322.


Searching for a link between the L-BMAA neurotoxin and amyotrophic lateral sclerosis: a study protocol of the French BMAALS programme.

Delzor A, Couratier P, Boumédiène F, Nicol M, Druet-Cabanac M, Paraf F, Méjean A, Ploux O, Leleu JP, Brient L, Lengronne M, Pichon V, Combès A, El Abdellaoui S, Bonneterre V, Lagrange E, Besson G, Bicout DJ, Boutonnat J, Camu W, Pageot N, Juntas-Morales R, Rigau V, Masseret E, Abadie E, Preux PM, Marin B.

BMJ Open. 2014 Sep 1;4(8):e005528. doi: 10.1136/bmjopen-2014-005528.


ALS as a distal axonopathy: molecular mechanisms affecting neuromuscular junction stability in the presymptomatic stages of the disease.

Moloney EB, de Winter F, Verhaagen J.

Front Neurosci. 2014 Aug 14;8:252. doi: 10.3389/fnins.2014.00252. eCollection 2014. Review.


Presence of the neurotoxin BMAA in aquatic ecosystems: what do we really know?

Faassen EJ.

Toxins (Basel). 2014 Mar 21;6(3):1109-38. doi: 10.3390/toxins6031109. Review.


Co-occurrence of the cyanotoxins BMAA, DABA and anatoxin-a in Nebraska reservoirs, fish, and aquatic plants.

Al-Sammak MA, Hoagland KD, Cassada D, Snow DD.

Toxins (Basel). 2014 Jan 28;6(2):488-508. doi: 10.3390/toxins6020488.


Dietary BMAA exposure in an amyotrophic lateral sclerosis cluster from southern France.

Masseret E, Banack S, Boumédiène F, Abadie E, Brient L, Pernet F, Juntas-Morales R, Pageot N, Metcalf J, Cox P, Camu W; French Network on ALS Clusters Detection and Investigation..

PLoS One. 2013 Dec 13;8(12):e83406. doi: 10.1371/journal.pone.0083406. eCollection 2013.


The non-protein amino acid BMAA is misincorporated into human proteins in place of L-serine causing protein misfolding and aggregation.

Dunlop RA, Cox PA, Banack SA, Rodgers KJ.

PLoS One. 2013 Sep 25;8(9):e75376. doi: 10.1371/journal.pone.0075376. eCollection 2013.


Exposure to environmental toxicants and pathogenesis of amyotrophic lateral sclerosis: state of the art and research perspectives.

Trojsi F, Monsurrò MR, Tedeschi G.

Int J Mol Sci. 2013 Jul 24;14(8):15286-311. doi: 10.3390/ijms140815286. Review.


The complex molecular biology of amyotrophic lateral sclerosis (ALS).

Redler RL, Dokholyan NV.

Prog Mol Biol Transl Sci. 2012;107:215-62. doi: 10.1016/B978-0-12-385883-2.00002-3. Review.


The tip of the iceberg: RNA-binding proteins with prion-like domains in neurodegenerative disease.

King OD, Gitler AD, Shorter J.

Brain Res. 2012 Jun 26;1462:61-80. doi: 10.1016/j.brainres.2012.01.016. Epub 2012 Jan 21. Review.


The emerging science of BMAA: do cyanobacteria contribute to neurodegenerative disease?

Holtcamp W.

Environ Health Perspect. 2012 Mar;120(3):A110-6. doi: 10.1289/ehp.120-a110. No abstract available.


The cyanobacteria derived toxin Beta-N-methylamino-L-alanine and amyotrophic lateral sclerosis.

Banack SA, Caller TA, Stommel EW.

Toxins (Basel). 2010 Dec;2(12):2837-50. doi: 10.3390/toxins2122837. Epub 2010 Dec 20. Review.


Does α-amino-β-methylaminopropionic acid (BMAA) play a role in neurodegeneration?

Chiu AS, Gehringer MM, Welch JH, Neilan BA.

Int J Environ Res Public Health. 2011 Sep;8(9):3728-46. doi: 10.3390/ijerph8093728. Epub 2011 Sep 16. Review.


Prominent human health impacts from several marine microbes: history, ecology, and public health implications.

Bienfang PK, Defelice SV, Laws EA, Brand LE, Bidigare RR, Christensen S, Trapido-Rosenthal H, Hemscheidt TK, McGillicuddy DJ, Anderson DM, Solo-Gabriele HM, Boehm AB, Backer LC.

Int J Microbiol. 2011;2011:152815. doi: 10.1155/2011/152815. Epub 2010 Oct 11.

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