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Items: 1 to 20 of 37

1.

DNA Damage Responses Are Induced by tRNA Anticodon Nucleases and Hygromycin B.

Wemhoff S, Klassen R, Beetz A, Meinhardt F.

PLoS One. 2016 Jul 29;11(7):e0157611. doi: 10.1371/journal.pone.0157611.

2.

Histone H2B mono-ubiquitylation maintains genomic integrity at stalled replication forks.

Northam MR, Trujillo KM.

Nucleic Acids Res. 2016 Nov 2;44(19):9245-9255.

3.

Proficient Replication of the Yeast Genome by a Viral DNA Polymerase.

Stodola JL, Stith CM, Burgers PM.

J Biol Chem. 2016 May 27;291(22):11698-705. doi: 10.1074/jbc.M116.728741.

PMID:
27072134
4.

Clusters of Multiple Mutations: Incidence and Molecular Mechanisms.

Chan K, Gordenin DA.

Annu Rev Genet. 2015;49:243-67. doi: 10.1146/annurev-genet-112414-054714. Review.

5.

Genetic Interactions Implicating Postreplicative Repair in Okazaki Fragment Processing.

Becker JR, Pons C, Nguyen HD, Costanzo M, Boone C, Myers CL, Bielinsky AK.

PLoS Genet. 2015 Nov 6;11(11):e1005659. doi: 10.1371/journal.pgen.1005659.

6.

The Protein Level of Rev1, a TLS Polymerase in Fission Yeast, Is Strictly Regulated during the Cell Cycle and after DNA Damage.

Uchiyama M, Terunuma J, Hanaoka F.

PLoS One. 2015 Jul 6;10(7):e0130000. doi: 10.1371/journal.pone.0130000.

7.

Genetic instability in budding and fission yeast-sources and mechanisms.

Skoneczna A, Kaniak A, Skoneczny M.

FEMS Microbiol Rev. 2015 Nov;39(6):917-67. doi: 10.1093/femsre/fuv028. Review.

8.

Disruption of Transcriptional Coactivator Sub1 Leads to Genome-Wide Re-distribution of Clustered Mutations Induced by APOBEC in Active Yeast Genes.

Lada AG, Kliver SF, Dhar A, Polev DE, Masharsky AE, Rogozin IB, Pavlov YI.

PLoS Genet. 2015 May 5;11(5):e1005217. doi: 10.1371/journal.pgen.1005217.

9.

Crystal Structure of the Human Pol α B Subunit in Complex with the C-terminal Domain of the Catalytic Subunit.

Suwa Y, Gu J, Baranovskiy AG, Babayeva ND, Pavlov YI, Tahirov TH.

J Biol Chem. 2015 Jun 5;290(23):14328-37. doi: 10.1074/jbc.M115.649954.

10.

Colon cancer-associated mutator DNA polymerase δ variant causes expansion of dNTP pools increasing its own infidelity.

Mertz TM, Sharma S, Chabes A, Shcherbakova PV.

Proc Natl Acad Sci U S A. 2015 May 12;112(19):E2467-76. doi: 10.1073/pnas.1422934112.

11.

dNTP pool levels modulate mutator phenotypes of error-prone DNA polymerase ε variants.

Williams LN, Marjavaara L, Knowels GM, Schultz EM, Fox EJ, Chabes A, Herr AJ.

Proc Natl Acad Sci U S A. 2015 May 12;112(19):E2457-66. doi: 10.1073/pnas.1422948112.

12.

DNA polymerase ζ-dependent lesion bypass in Saccharomyces cerevisiae is accompanied by error-prone copying of long stretches of adjacent DNA.

Kochenova OV, Daee DL, Mertz TM, Shcherbakova PV.

PLoS Genet. 2015 Mar 31;11(3):e1005110. doi: 10.1371/journal.pgen.1005110.

13.

Eukaryotic DNA polymerase ζ.

Makarova AV, Burgers PM.

DNA Repair (Amst). 2015 May;29:47-55. doi: 10.1016/j.dnarep.2015.02.012. Review.

14.

Loss of the thioredoxin reductase Trr1 suppresses the genomic instability of peroxiredoxin tsa1 mutants.

Ragu S, Dardalhon M, Sharma S, Iraqui I, Buhagiar-Labarchède G, Grondin V, Kienda G, Vernis L, Chanet R, Kolodner RD, Huang ME, Faye G.

PLoS One. 2014 Sep 23;9(9):e108123. doi: 10.1371/journal.pone.0108123.

15.

Error-prone polymerase activity causes multinucleotide mutations in humans.

Harris K, Nielsen R.

Genome Res. 2014 Sep;24(9):1445-54. doi: 10.1101/gr.170696.113.

16.

The C-terminal domain of the DNA polymerase catalytic subunit regulates the primase and polymerase activities of the human DNA polymerase α-primase complex.

Zhang Y, Baranovskiy AG, Tahirov TH, Pavlov YI.

J Biol Chem. 2014 Aug 8;289(32):22021-34. doi: 10.1074/jbc.M114.570333.

17.

A novel variant of DNA polymerase ζ, Rev3ΔC, highlights differential regulation of Pol32 as a subunit of polymerase δ versus ζ in Saccharomyces cerevisiae.

Siebler HM, Lada AG, Baranovskiy AG, Tahirov TH, Pavlov YI.

DNA Repair (Amst). 2014 Dec;24:138-49. doi: 10.1016/j.dnarep.2014.04.013.

18.

Mcm10 deficiency causes defective-replisome-induced mutagenesis and a dependency on error-free postreplicative repair.

Becker JR, Nguyen HD, Wang X, Bielinsky AK.

Cell Cycle. 2014;13(11):1737-48. doi: 10.4161/cc.28652.

19.
20.

A reversible histone H3 acetylation cooperates with mismatch repair and replicative polymerases in maintaining genome stability.

Kadyrova LY, Mertz TM, Zhang Y, Northam MR, Sheng Z, Lobachev KS, Shcherbakova PV, Kadyrov FA.

PLoS Genet. 2013 Oct;9(10):e1003899. doi: 10.1371/journal.pgen.1003899.

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