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Items: 1 to 20 of 43


Post-translationally modified muscle-specific ubiquitin ligases as circulating biomarkers in experimental cancer cachexia.

Mota R, Rodríguez JE, Bonetto A, O'Connell TM, Asher SA, Parry TL, Lockyer P, McCudden CR, Couch ME, Willis MS.

Am J Cancer Res. 2017 Sep 1;7(9):1948-1958. eCollection 2017.


The Calcineurin-FoxO-MuRF1 signaling pathway regulates myofibril integrity in cardiomyocytes.

Shimizu H, Langenbacher AD, Huang J, Wang K, Otto G, Geisler R, Wang Y, Chen JN.

Elife. 2017 Aug 19;6. pii: e27955. doi: 10.7554/eLife.27955.


Non-targeted metabolomics analysis of cardiac Muscle Ring Finger-1 (MuRF1), MuRF2, and MuRF3 in vivo reveals novel and redundant metabolic changes.

Banerjee R, He J, Spaniel C, Quintana MT, Wang Z, Bain J, Newgard CB, Muehlbauer MJ, Willis MS.

Metabolomics. 2015 Apr;11(2):312-322. doi: 10.1007/s11306-014-0695-1. Epub 2014 Jul 14.


BRG1 and BRM function antagonistically with c-MYC in adult cardiomyocytes to regulate conduction and contractility.

Willis MS, Holley DW, Wang Z, Chen X, Quintana M, Jensen BC, Tannu M, Parker J, Jeyaraj D, Jain MK, Wolfram JA, Lee HG, Bultman SJ.

J Mol Cell Cardiol. 2017 Apr;105:99-109. doi: 10.1016/j.yjmcc.2017.02.003. Epub 2017 Feb 21.


Transcriptional activation of muscle atrophy promotes cardiac muscle remodeling during mammalian hibernation.

Zhang Y, Aguilar OA, Storey KB.

PeerJ. 2016 Aug 11;4:e2317. doi: 10.7717/peerj.2317. eCollection 2016.


Cardiac ubiquitin ligases: Their role in cardiac metabolism, autophagy, cardioprotection and therapeutic potential.

Parry TL, Willis MS.

Biochim Biophys Acta. 2016 Dec;1862(12):2259-2269. doi: 10.1016/j.bbadis.2016.07.002. Epub 2016 Jul 13.


MuRF1 mono-ubiquitinates TRα to inhibit T3-induced cardiac hypertrophy in vivo.

Wadosky KM, Berthiaume JM, Tang W, Zungu M, Portman MA, Gerdes AM, Willis MS.

J Mol Endocrinol. 2016 Apr;56(3):273-90. doi: 10.1530/JME-15-0283. Epub 2016 Feb 9.


Fenofibrate unexpectedly induces cardiac hypertrophy in mice lacking MuRF1.

Parry TL, Desai G, Schisler JC, Li L, Quintana MT, Stanley N, Lockyer P, Patterson C, Willis MS.

Cardiovasc Pathol. 2016 Mar-Apr;25(2):127-140. doi: 10.1016/j.carpath.2015.09.008. Epub 2015 Oct 29.


Ubiquitin and ubiquitin-like proteins in cardiac disease and protection.

Li J, Johnson JA, Su H.

Curr Drug Targets. 2015 Dec 8. [Epub ahead of print]


Developmental MYH3 Myopathy Associated with Expression of Mutant Protein and Reduced Expression Levels of Embryonic MyHC.

Pokrzywa M, Norum M, Lengqvist J, Ghobadpour M, Abdul-Hussein S, Moslemi AR, Tajsharghi H.

PLoS One. 2015 Nov 6;10(11):e0142094. doi: 10.1371/journal.pone.0142094. eCollection 2015.


Non-targeted metabolomics of Brg1/Brm double-mutant cardiomyocytes reveals a novel role for SWI/SNF complexes in metabolic homeostasis.

Banerjee R, Bultman SJ, Holley D, Hillhouse C, Bain JR, Newgard CB, Muehlbauer MJ, Willis MS.

Metabolomics. 2015 Oct 1;11(5):1287-1301.


The ubiquitin ligase MuRF1 regulates PPARα activity in the heart by enhancing nuclear export via monoubiquitination.

Rodríguez JE, Liao JY, He J, Schisler JC, Newgard CB, Drujan D, Glass DJ, Frederick CB, Yoder BC, Lalush DS, Patterson C, Willis MS.

Mol Cell Endocrinol. 2015 Sep 15;413:36-48. doi: 10.1016/j.mce.2015.06.008. Epub 2015 Jun 25.


IMP 2.0: a multi-species functional genomics portal for integration, visualization and prediction of protein functions and networks.

Wong AK, Krishnan A, Yao V, Tadych A, Troyanskaya OG.

Nucleic Acids Res. 2015 Jul 1;43(W1):W128-33. doi: 10.1093/nar/gkv486. Epub 2015 May 12.


Natural aminoacyl tRNA synthetase fragment enhances cardiac function after myocardial infarction.

McCormick ME, Rojas M, Moser-Katz T, Tzima E, Reader JS.

PLoS One. 2014 Oct 8;9(10):e109325. doi: 10.1371/journal.pone.0109325. eCollection 2014.


Targeting the ubiquitin-proteasome system in heart disease: the basis for new therapeutic strategies.

Drews O, Taegtmeyer H.

Antioxid Redox Signal. 2014 Dec 10;21(17):2322-43. doi: 10.1089/ars.2013.5823. Epub 2014 Oct 1. Review.


Muscle RING finger-1 promotes a maladaptive phenotype in chronic hypoxia-induced right ventricular remodeling.

Campen MJ, Paffett ML, Colombo ES, Lucas SN, Anderson T, Nysus M, Norenberg JP, Gershman B, Hesterman J, Hoppin J, Willis M.

PLoS One. 2014 May 8;9(5):e97084. doi: 10.1371/journal.pone.0097084. eCollection 2014.


Small dedifferentiated cardiomyocytes bordering on microdomains of fibrosis: evidence for reverse remodeling with assisted recovery.

Al Darazi F, Zhao W, Zhao T, Sun Y, Marion TN, Ahokas RA, Bhattacharya SK, Gerling IC, Weber KT.

J Cardiovasc Pharmacol. 2014 Sep;64(3):237-46. doi: 10.1097/FJC.0000000000000111.


MuRF1 activity is present in cardiac mitochondria and regulates reactive oxygen species production in vivo.

Mattox TA, Young ME, Rubel CE, Spaniel C, Rodríguez JE, Grevengoed TJ, Gautel M, Xu Z, Anderson EJ, Willis MS.

J Bioenerg Biomembr. 2014 Jun;46(3):173-87. doi: 10.1007/s10863-014-9549-9. Epub 2014 Apr 15. Erratum in: J Bioenerg Biomembr. 2015 Jun;47(3):281.


MAFbx/Atrogin-1 is required for atrophic remodeling of the unloaded heart.

Baskin KK, Rodriguez MR, Kansara S, Chen W, Carranza S, Frazier OH, Glass DJ, Taegtmeyer H.

J Mol Cell Cardiol. 2014 Jul;72:168-76. doi: 10.1016/j.yjmcc.2014.03.006. Epub 2014 Mar 18.


Muscle RING finger-1 attenuates IGF-I-dependent cardiomyocyte hypertrophy by inhibiting JNK signaling.

Wadosky KM, Rodríguez JE, Hite RL, Min JN, Walton BL, Willis MS.

Am J Physiol Endocrinol Metab. 2014 Apr 1;306(7):E723-39. doi: 10.1152/ajpendo.00326.2013. Epub 2014 Jan 14.

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