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Items: 1 to 20 of 50

1.

Semaphorin-Plexin signaling influences early ventral telencephalic development and thalamocortical axon guidance.

Mitsogiannis MD, Little GE, Mitchell KJ.

Neural Dev. 2017 Apr 24;12(1):6. doi: 10.1186/s13064-017-0083-4.

2.

Hyperactivity of the default-mode network in first-episode, drug-naive schizophrenia at rest revealed by family-based case-control and traditional case-control designs.

Guo W, Liu F, Chen J, Wu R, Li L, Zhang Z, Chen H, Zhao J.

Medicine (Baltimore). 2017 Mar;96(13):e6223. doi: 10.1097/MD.0000000000006223.

3.

Abnormalities of localized connectivity in schizophrenia patients and their unaffected relatives: a meta-analysis of resting-state functional magnetic resonance imaging studies.

Xiao B, Wang S, Liu J, Meng T, He Y, Luo X.

Neuropsychiatr Dis Treat. 2017 Feb 14;13:467-475. doi: 10.2147/NDT.S126678. eCollection 2017.

4.

Functional connectivity when detecting rare visual targets in schizophrenia.

Jimenez AM, Lee J, Green MF, Wynn JK.

Psychiatry Res. 2017 Mar 30;261:35-43. doi: 10.1016/j.pscychresns.2017.01.007. Epub 2017 Jan 17.

PMID:
28126618
5.

Multisite reliability of MR-based functional connectivity.

Noble S, Scheinost D, Finn ES, Shen X, Papademetris X, McEwen SC, Bearden CE, Addington J, Goodyear B, Cadenhead KS, Mirzakhanian H, Cornblatt BA, Olvet DM, Mathalon DH, McGlashan TH, Perkins DO, Belger A, Seidman LJ, Thermenos H, Tsuang MT, van Erp TG, Walker EF, Hamann S, Woods SW, Cannon TD, Constable RT.

Neuroimage. 2017 Feb 1;146:959-970. doi: 10.1016/j.neuroimage.2016.10.020. Epub 2016 Oct 13.

6.

Patients with first-episode, drug-naive schizophrenia and subjects at ultra-high risk of psychosis shared increased cerebellar-default mode network connectivity at rest.

Wang H, Guo W, Liu F, Wang G, Lyu H, Wu R, Chen J, Wang S, Li L, Zhao J.

Sci Rep. 2016 May 18;6:26124. doi: 10.1038/srep26124.

7.

Resting-state cerebellar-cerebral networks are differently affected in first-episode, drug-naive schizophrenia patients and unaffected siblings.

Guo W, Liu F, Chen J, Wu R, Zhang Z, Yu M, Xiao C, Zhao J.

Sci Rep. 2015 Nov 26;5:17275. doi: 10.1038/srep17275.

8.

Increased Cerebellar Functional Connectivity With the Default-Mode Network in Unaffected Siblings of Schizophrenia Patients at Rest.

Guo W, Liu F, Zhang Z, Liu G, Liu J, Yu L, Xiao C, Zhao J.

Schizophr Bull. 2015 Nov;41(6):1317-25. doi: 10.1093/schbul/sbv062. Epub 2015 May 7.

9.

Aberrant Functional Connectivity in the Default Mode and Central Executive Networks in Subjects with Schizophrenia - A Whole-Brain Resting-State ICA Study.

Littow H, Huossa V, Karjalainen S, Jääskeläinen E, Haapea M, Miettunen J, Tervonen O, Isohanni M, Nikkinen J, Veijola J, Murray G, Kiviniemi VJ.

Front Psychiatry. 2015 Feb 26;6:26. doi: 10.3389/fpsyt.2015.00026. eCollection 2015.

10.
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12.

Early-course unmedicated schizophrenia patients exhibit elevated prefrontal connectivity associated with longitudinal change.

Anticevic A, Hu X, Xiao Y, Hu J, Li F, Bi F, Cole MW, Savic A, Yang GJ, Repovs G, Murray JD, Wang XJ, Huang X, Lui S, Krystal JH, Gong Q.

J Neurosci. 2015 Jan 7;35(1):267-86. doi: 10.1523/JNEUROSCI.2310-14.2015.

13.

PD_NGSAtlas: a reference database combining next-generation sequencing epigenomic and transcriptomic data for psychiatric disorders.

Zhao Z, Li Y, Chen H, Lu J, Thompson PM, Chen J, Wang Z, Xu J, Xu C, Li X.

BMC Med Genomics. 2014 Dec 31;7:71. doi: 10.1186/s12920-014-0071-z.

14.

Altered cognitive development in the siblings of individuals with schizophrenia.

Barch DM, Cohen R, Csernansky J.

Clin Psychol Sci. 2014 Mar 1;2(2):138-151.

15.

Altered white matter microstructure is associated with social cognition and psychotic symptoms in 22q11.2 microdeletion syndrome.

Jalbrzikowski M, Villalon-Reina JE, Karlsgodt KH, Senturk D, Chow C, Thompson PM, Bearden CE.

Front Behav Neurosci. 2014 Nov 11;8:393. doi: 10.3389/fnbeh.2014.00393. eCollection 2014.

16.

A sequence variant in human KALRN impairs protein function and coincides with reduced cortical thickness.

Russell TA, Blizinsky KD, Cobia DJ, Cahill ME, Xie Z, Sweet RA, Duan J, Gejman PV, Wang L, Csernansky JG, Penzes P.

Nat Commun. 2014 Sep 16;5:4858. doi: 10.1038/ncomms5858.

17.

Abnormal causal connectivity by structural deficits in first-episode, drug-naive schizophrenia at rest.

Guo W, Liu F, Liu J, Yu L, Zhang J, Zhang Z, Xiao C, Zhai J, Zhao J.

Schizophr Bull. 2015 Jan;41(1):57-65. doi: 10.1093/schbul/sbu126. Epub 2014 Aug 28.

18.

White matter development in the early stages of psychosis.

Peters BD, Karlsgodt KH.

Schizophr Res. 2015 Jan;161(1):61-9. doi: 10.1016/j.schres.2014.05.021. Epub 2014 Jun 2. Review.

19.

Neurological soft signs predict abnormal cerebellar-thalamic tract development and negative symptoms in adolescents at high risk for psychosis: a longitudinal perspective.

Mittal VA, Dean DJ, Bernard JA, Orr JM, Pelletier-Baldelli A, Carol EE, Gupta T, Turner J, Leopold DR, Robustelli BL, Millman ZB.

Schizophr Bull. 2014 Nov;40(6):1204-15. doi: 10.1093/schbul/sbt199. Epub 2013 Dec 27.

20.

Developmental mechanisms in the prodrome to psychosis.

Walker EF, Trotman HD, Goulding SM, Holtzman CW, Ryan AT, McDonald A, Shapiro DI, Brasfield JL.

Dev Psychopathol. 2013 Nov;25(4 Pt 2):1585-600. doi: 10.1017/S0954579413000783. Review.

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