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Items: 14

1.

Association between AIRE gene polymorphism and rheumatoid arthritis: a systematic review and meta-analysis of case-control studies.

Bérczi B, Gerencsér G, Farkas N, Hegyi P, Veres G, Bajor J, Czopf L, Alizadeh H, Rakonczay Z, Vigh É, Erőss B, Szemes K, Gyöngyi Z.

Sci Rep. 2017 Oct 26;7(1):14096. doi: 10.1038/s41598-017-14375-z.

2.

A case-control study on association of proteasome subunit beta 8 (PSMB8) and transporter associated with antigen processing 1 (TAP1) polymorphisms and their transcript levels in vitiligo from Gujarat.

Jadeja SD, Mansuri MS, Singh M, Dwivedi M, Laddha NC, Begum R.

PLoS One. 2017 Jul 10;12(7):e0180958. doi: 10.1371/journal.pone.0180958. eCollection 2017.

3.

AIRE polymorphism, melanoma antigen-specific T cell immunity, and susceptibility to melanoma.

Conteduca G, Fenoglio D, Parodi A, Battaglia F, Kalli F, Negrini S, Tardito S, Ferrera F, Salis A, Millo E, Pasquale G, Barra G, Damonte G, Indiveri F, Ferrone S, Filaci G.

Oncotarget. 2016 Sep 20;7(38):60872-60884. doi: 10.18632/oncotarget.11506.

4.

The AIRE -230Y Polymorphism Affects AIRE Transcriptional Activity: Potential Influence on AIRE Function in the Thymus.

Lovewell TR, McDonagh AJ, Messenger AG, Azzouz M, Tazi-Ahnini R.

PLoS One. 2015 May 15;10(5):e0127476. doi: 10.1371/journal.pone.0127476. eCollection 2015.

5.

Transporter TAP1-637G and immunoproteasome PSMB9-60H variants influence the risk of developing vitiligo in the Saudi population.

Elhawary NA, Bogari N, Jiffri EH, Rashad M, Fatani A, Tayeb M.

Dis Markers. 2014;2014:260732. doi: 10.1155/2014/260732. Epub 2014 Dec 7.

6.

Human APECED; a Sick Thymus Syndrome?

Arstila TP, Jarva H.

Front Immunol. 2013 Oct 7;4:313. doi: 10.3389/fimmu.2013.00313. Review.

7.

Aire deficiency promotes TRP-1-specific immune rejection of melanoma.

Zhu ML, Nagavalli A, Su MA.

Cancer Res. 2013 Apr 1;73(7):2104-16. doi: 10.1158/0008-5472.CAN-12-3781. Epub 2013 Jan 31.

8.

Update on the genetics characterization of vitiligo.

Al-Shobaili HA.

Int J Health Sci (Qassim). 2011 Jul;5(2):167-79.

9.

Tumor necrosis factor-α -308G/A polymorphism is associated with active vitiligo vulgaris in a northeastern Mexican population.

Salinas-Santander M, Díaz-García D, Rojas-Martínez A, Cantú-Salinas C, Sánchez-Domínguez C, Reyes-López M, Cerda-Flores RM, Ocampo-Candiani J, Ortiz-López R.

Exp Ther Med. 2012 May;3(5):893-897. Epub 2012 Mar 9.

10.

Comprehensive association analysis of candidate genes for generalized vitiligo supports XBP1, FOXP3, and TSLP.

Birlea SA, Jin Y, Bennett DC, Herbstman DM, Wallace MR, McCormack WT, Kemp EH, Gawkrodger DJ, Weetman AP, Picardo M, Leone G, Taïeb A, Jouary T, Ezzedine K, van Geel N, Lambert J, Overbeck A, Fain PR, Spritz RA.

J Invest Dermatol. 2011 Feb;131(2):371-81. doi: 10.1038/jid.2010.337. Epub 2010 Nov 18.

11.

Vitiligo vulgaris and autoimmune diseases in Japan: A report from vitiligo clinic in Kyoto University Hospital.

Tanioka M, Yamamoto Y, Katoh M, Takahashi K, Miyachi Y.

Dermatoendocrinol. 2009 Jan;1(1):43-5.

12.

Genetic variation of promoter sequence modulates XBP1 expression and genetic risk for vitiligo.

Ren Y, Yang S, Xu S, Gao M, Huang W, Gao T, Fang Q, Quan C, Zhang C, Sun L, Liang Y, Han J, Wang Z, Zhang F, Zhou Y, Liu J, Zhang X.

PLoS Genet. 2009 Jun;5(6):e1000523. doi: 10.1371/journal.pgen.1000523. Epub 2009 Jun 19.

13.

A possible systemic rheumatic disorder in the Nova Scotia duck tolling retriever.

Hansson-Hamlin H, Lilliehöök I.

Acta Vet Scand. 2009 Mar 30;51:16. doi: 10.1186/1751-0147-51-16.

14.

Monogenic autoimmune diseases: insights into self-tolerance.

Su MA, Anderson MS.

Pediatr Res. 2009 May;65(5 Pt 2):20R-25R. doi: 10.1203/PDR.0b013e31819dc55c. Review.

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