Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 17


RNPC1 enhances progesterone receptor functions by regulating its mRNA stability in breast cancer.

Lou P, Li C, Shi L, Xia TS, Zhou W, Wu J, Zhou X, Li X, Wang Y, Wei JF, Ding Q.

Oncotarget. 2017 Mar 7;8(10):16387-16400. doi: 10.18632/oncotarget.12016.


Signaling Pathways in Leiomyoma: Understanding Pathobiology and Implications for Therapy.

Borahay MA, Al-Hendy A, Kilic GS, Boehning D.

Mol Med. 2015 Apr 13;21:242-56. doi: 10.2119/molmed.2014.00053. Review.


Interaction of JAK with steroid receptor function.

Gupta N, Mayer D.

JAKSTAT. 2013 Oct 1;2(4):e24911. doi: 10.4161/jkst.24911. Epub 2013 May 7. Review.


Lysine methylation of progesterone receptor at activation function 1 regulates both ligand-independent activity and ligand sensitivity of the receptor.

Chung HH, Sze SK, Woo AR, Sun Y, Sim KH, Dong XM, Lin VC.

J Biol Chem. 2014 Feb 28;289(9):5704-22. doi: 10.1074/jbc.M113.522839. Epub 2014 Jan 10.


Post-translational modifications of the progesterone receptors.

Abdel-Hafiz HA, Horwitz KB.

J Steroid Biochem Mol Biol. 2014 Mar;140:80-9. doi: 10.1016/j.jsbmb.2013.12.008. Epub 2013 Dec 12. Review.


Tracking progesterone receptor-mediated actions in breast cancer.

Knutson TP, Lange CA.

Pharmacol Ther. 2014 Apr;142(1):114-25. doi: 10.1016/j.pharmthera.2013.11.010. Epub 2013 Nov 26. Review.


Progesterone receptors induce FOXO1-dependent senescence in ovarian cancer cells.

Diep CH, Charles NJ, Gilks CB, Kalloger SE, Argenta PA, Lange CA.

Cell Cycle. 2013 May 1;12(9):1433-49. doi: 10.4161/cc.24550. Epub 2013 Apr 10.


CDK2-dependent activation of PARP-1 is required for hormonal gene regulation in breast cancer cells.

Wright RH, Castellano G, Bonet J, Le Dily F, Font-Mateu J, Ballaré C, Nacht AS, Soronellas D, Oliva B, Beato M.

Genes Dev. 2012 Sep 1;26(17):1972-83. doi: 10.1101/gad.193193.112.


Evidence for a genomic mechanism of action for progesterone receptor membrane component-1.

Peluso JJ, DeCerbo J, Lodde V.

Steroids. 2012 Aug;77(10):1007-12. doi: 10.1016/j.steroids.2012.01.013. Epub 2012 Feb 1. Review.


Conserved steroid hormone homology converges on nuclear factor κB to modulate inflammation in asthma.

Payne AS, Freishtat RJ.

J Investig Med. 2012 Jan;60(1):13-7. doi: 10.2310/JIM.0b013e31823d7989. Review.


The role of progesterone signaling in the pathogenesis of uterine leiomyoma.

Kim JJ, Sefton EC.

Mol Cell Endocrinol. 2012 Jul 25;358(2):223-31. doi: 10.1016/j.mce.2011.05.044. Epub 2011 Jun 6. Review.


Genomic collaboration of estrogen receptor alpha and extracellular signal-regulated kinase 2 in regulating gene and proliferation programs.

Madak-Erdogan Z, Lupien M, Stossi F, Brown M, Katzenellenbogen BS.

Mol Cell Biol. 2011 Jan;31(1):226-36. doi: 10.1128/MCB.00821-10. Epub 2010 Oct 18.


The antiproliferative effects of progestins in T47D breast cancer cells are tempered by progestin induction of the ETS transcription factor Elf5.

Hilton HN, Kalyuga M, Cowley MJ, Alles MC, Lee HJ, Caldon CE, Blazek K, Kaplan W, Musgrove EA, Daly RJ, Naylor MJ, Graham JD, Clarke CL, Ormandy CJ.

Mol Endocrinol. 2010 Jul;24(7):1380-92. doi: 10.1210/me.2009-0516. Epub 2010 Jun 2.


Progesterone action in human tissues: regulation by progesterone receptor (PR) isoform expression, nuclear positioning and coregulator expression.

Scarpin KM, Graham JD, Mote PA, Clarke CL.

Nucl Recept Signal. 2009 Dec 31;7:e009. doi: 10.1621/nrs.07009. Review.


Progestin stimulation of manganese superoxide dismutase and invasive properties in T47D human breast cancer cells.

Holley AK, Kiningham KK, Spitz DR, Edwards DP, Jenkins JT, Moore MR.

J Steroid Biochem Mol Biol. 2009 Oct;117(1-3):23-30. doi: 10.1016/j.jsbmb.2009.06.004. Epub 2009 Jun 27.


Crosstalk between the estrogen receptor and the HER tyrosine kinase receptor family: molecular mechanism and clinical implications for endocrine therapy resistance.

Arpino G, Wiechmann L, Osborne CK, Schiff R.

Endocr Rev. 2008 Apr;29(2):217-33. doi: 10.1210/er.2006-0045. Epub 2008 Jan 23. Review.

Supplemental Content

Support Center