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Items: 1 to 20 of 107

1.

Genetic association and epistatic interaction of the interleukin-10 signaling pathway in pediatric inflammatory bowel disease.

Lin Z, Wang Z, Hegarty JP, Lin TR, Wang Y, Deiling S, Wu R, Thomas NJ, Floros J.

World J Gastroenterol. 2017 Jul 21;23(27):4897-4909. doi: 10.3748/wjg.v23.i27.4897.

2.

Pathomechanisms of Oxidative Stress in Inflammatory Bowel Disease and Potential Antioxidant Therapies.

Tian T, Wang Z, Zhang J.

Oxid Med Cell Longev. 2017;2017:4535194. doi: 10.1155/2017/4535194. Epub 2017 Jun 28. Review.

3.

Association of Ribonuclease T2 Gene Polymorphisms With Decreased Expression and Clinical Characteristics of Severity in Crohn's Disease.

Gonsky R, Fleshner P, Deem RL, Biener-Ramanujan E, Li D, Potdar AA, Bilsborough J, Yang S, McGovern DPB, Targan SR.

Gastroenterology. 2017 Jul;153(1):219-232. doi: 10.1053/j.gastro.2017.04.002. Epub 2017 Apr 9.

PMID:
28400196
4.

LRRK2 but not ATG16L1 is associated with Paneth cell defect in Japanese Crohn's disease patients.

Liu TC, Naito T, Liu Z, VanDussen KL, Haritunians T, Li D, Endo K, Kawai Y, Nagasaki M, Kinouchi Y, McGovern DP, Shimosegawa T, Kakuta Y, Stappenbeck TS.

JCI Insight. 2017 Mar 23;2(6):e91917. doi: 10.1172/jci.insight.91917.

5.

Directed evolution of a soluble human DR3 receptor for the inhibition of TL1A induced cytokine secretion.

Levin I, Zaretsky M, Aharoni A.

PLoS One. 2017 Mar 9;12(3):e0173460. doi: 10.1371/journal.pone.0173460. eCollection 2017.

6.

Opportunities and challenges of whole-genome and -exome sequencing.

Petersen BS, Fredrich B, Hoeppner MP, Ellinghaus D, Franke A.

BMC Genet. 2017 Feb 14;18(1):14. doi: 10.1186/s12863-017-0479-5. Review.

7.

Lymphocyte Antigen 75 Polymorphisms Are Associated with Disease Susceptibility and Phenotype in Japanese Patients with Inflammatory Bowel Disease.

Hirayama A, Joshita S, Kitahara K, Mukawa K, Suga T, Umemura T, Tanaka E, Ota M.

Dis Markers. 2016;2016:6485343. Epub 2016 Nov 14.

8.

Genome-Wide Association Study Identifies African-Specific Susceptibility Loci in African Americans With Inflammatory Bowel Disease.

Brant SR, Okou DT, Simpson CL, Cutler DJ, Haritunians T, Bradfield JP, Chopra P, Prince J, Begum F, Kumar A, Huang C, Venkateswaran S, Datta LW, Wei Z, Thomas K, Herrinton LJ, Klapproth JA, Quiros AJ, Seminerio J, Liu Z, Alexander JS, Baldassano RN, Dudley-Brown S, Cross RK, Dassopoulos T, Denson LA, Dhere TA, Dryden GW, Hanson JS, Hou JK, Hussain SZ, Hyams JS, Isaacs KL, Kader H, Kappelman MD, Katz J, Kellermayer R, Kirschner BS, Kuemmerle JF, Kwon JH, Lazarev M, Li E, Mack D, Mannon P, Moulton DE, Newberry RD, Osuntokun BO, Patel AS, Saeed SA, Targan SR, Valentine JF, Wang MH, Zonca M, Rioux JD, Duerr RH, Silverberg MS, Cho JH, Hakonarson H, Zwick ME, McGovern DP, Kugathasan S.

Gastroenterology. 2017 Jan;152(1):206-217.e2. doi: 10.1053/j.gastro.2016.09.032. Epub 2016 Sep 28.

PMID:
27693347
9.

Death Receptor 3 Promotes Chemokine-Directed Leukocyte Recruitment in Acute Resolving Inflammation and Is Essential for Pathological Development of Mesothelial Fibrosis in Chronic Disease.

Perks WV, Singh RK, Jones GW, Twohig JP, Williams AS, Humphreys IR, Taylor PR, Jones SA, Wang ECY.

Am J Pathol. 2016 Nov;186(11):2813-2823. doi: 10.1016/j.ajpath.2016.07.021. Epub 2016 Sep 21.

10.

Variant detection and runs of homozygosity in next generation sequencing data elucidate the genetic background of Lundehund syndrome.

Metzger J, Pfahler S, Distl O.

BMC Genomics. 2016 Aug 2;17:535. doi: 10.1186/s12864-016-2844-6.

11.

Genetic variation in IBD: progress, clues to pathogenesis and possible clinical utility.

Ye BD, McGovern DP.

Expert Rev Clin Immunol. 2016 Oct;12(10):1091-107. doi: 10.1080/1744666X.2016.1184972. Epub 2016 Jun 15. Review.

12.

Identification of OCTN2 variants and their association with phenotypes of Crohn's disease in a Korean population.

Park HJ, Jung ES, Kong KA, Park EM, Cheon JH, Choi JH.

Sci Rep. 2016 Mar 11;6:22887. doi: 10.1038/srep22887.

13.

Differential Levels of Tl1a Affect the Expansion and Function of Regulatory T Cells in Modulating Murine Colitis.

Sidhu-Varma M, Shih DQ, Targan SR.

Inflamm Bowel Dis. 2016 Mar;22(3):548-59. doi: 10.1097/MIB.0000000000000653.

14.

Role of HLA typing on Crohn's disease pathogenesis.

Mahdi BM.

Ann Med Surg (Lond). 2015 Aug 4;4(3):248-53. doi: 10.1016/j.amsu.2015.07.020. eCollection 2015 Sep.

15.

Genetics of Inflammatory Bowel Diseases.

McGovern DP, Kugathasan S, Cho JH.

Gastroenterology. 2015 Oct;149(5):1163-1176.e2. doi: 10.1053/j.gastro.2015.08.001. Epub 2015 Aug 7. Review.

16.

The TNF-family cytokine TL1A: from lymphocyte costimulator to disease co-conspirator.

Richard AC, Ferdinand JR, Meylan F, Hayes ET, Gabay O, Siegel RM.

J Leukoc Biol. 2015 Sep;98(3):333-45. doi: 10.1189/jlb.3RI0315-095R. Epub 2015 Jul 17. Review.

17.

Polymorphisms in PRKCDBP, a Transcriptional Target of TNF-α, Are Associated With Inflammatory Bowel Disease in Korean.

Kim JW, Lee CK, Kim HJ, Shim JJ, Jang JY, Dong SH, Kim BH, Chang YW, Chi SG.

Intest Res. 2015 Jul;13(3):242-9. doi: 10.5217/ir.2015.13.3.242. Epub 2015 Jun 9.

18.

Exome sequencing identifies novel compound heterozygous IFNA4 and IFNA10 mutations as a cause of impaired function in Crohn's disease patients.

Xiao CX, Xiao JJ, Xu HZ, Wang HH, Chen X, Liu YS, Li P, Shi Y, Nie YZ, Li S, Wu KC, Liu ZJ, Ren JL, Guleng B.

Sci Rep. 2015 May 22;5:10514. doi: 10.1038/srep10514.

19.

Protective association of tumor necrosis factor superfamily 15 (TNFSF15) polymorphic haplotype with Ulcerative Colitis and Crohn's disease in an Indian population.

Baskaran K, Pugazhendhi S, Ramakrishna BS.

PLoS One. 2014 Dec 12;9(12):e114665. doi: 10.1371/journal.pone.0114665. eCollection 2014.

20.

Immunochip analysis identification of 6 additional susceptibility loci for Crohn's disease in Koreans.

Yang SK, Hong M, Choi H, Zhao W, Jung Y, Haritunians T, Ye BD, Kim KJ, Park SH, Lee I, Kim WH, Cheon JH, Kim YH, Jang BI, Kim HS, Choi JH, Koo JS, Lee JH, Jung SA, Shin HD, Kang D, Youn HS, Taylor KD, Rotter JI, Liu J, McGovern DP, Song K.

Inflamm Bowel Dis. 2015 Jan;21(1):1-7. doi: 10.1097/MIB.0000000000000268.

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