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Items: 14


Prioritized Expression of BTN2 of Saccharomyces cerevisiae under Pronounced Translation Repression Induced by Severe Ethanol Stress.

Yamauchi Y, Izawa S.

Front Microbiol. 2016 Aug 23;7:1319. doi: 10.3389/fmicb.2016.01319. eCollection 2016 Aug 23.


Ist2 in the yeast cortical endoplasmic reticulum promotes trafficking of the amino acid transporter Bap2 to the plasma membrane.

Wolf W, Meese K, Seedorf M.

PLoS One. 2014 Jan 8;9(1):e85418. doi: 10.1371/journal.pone.0085418. eCollection 2014 Jan 8.


Molecular mechanisms of spatial protein quality control.

Alberti S.

Prion. 2012 Nov-Dec;6(5):437-42. doi: 10.4161/pri.22470. Epub 2012 Oct 10.


Yeast Ist2 recruits the endoplasmic reticulum to the plasma membrane and creates a ribosome-free membrane microcompartment.

Wolf W, Kilic A, Schrul B, Lorenz H, Schwappach B, Seedorf M.

PLoS One. 2012;7(7):e39703. doi: 10.1371/journal.pone.0039703. Epub 2012 Jul 9.


The yeast Batten disease orthologue Btn1 controls endosome-Golgi retrograde transport via SNARE assembly.

Kama R, Kanneganti V, Ungermann C, Gerst JE.

J Cell Biol. 2011 Oct 17;195(2):203-15. doi: 10.1083/jcb.201102115. Epub 2011 Oct 10.


Btn3 is a negative regulator of Btn2-mediated endosomal protein trafficking and prion curing in yeast.

Kanneganti V, Kama R, Gerst JE.

Mol Biol Cell. 2011 May 15;22(10):1648-63. doi: 10.1091/mbc.E10-11-0878. Epub 2011 Mar 25.


A genomic approach highlights common and diverse effects and determinants of susceptibility on the yeast Saccharomyces cerevisiae exposed to distinct antimicrobial peptides.

López-García B, Gandía M, Muñoz A, Carmona L, Marcos JF.

BMC Microbiol. 2010 Nov 15;10:289. doi: 10.1186/1471-2180-10-289.


Interaction between Sdo1p and Btn1p in the Saccharomyces cerevisiae model for Batten disease.

Vitiello SP, Benedict JW, Padilla-Lopez S, Pearce DA.

Hum Mol Genet. 2010 Mar 1;19(5):931-42. doi: 10.1093/hmg/ddp560. Epub 2009 Dec 16.


Anoctamin and transmembrane channel-like proteins are evolutionarily related.

Hahn Y, Kim DS, Pastan IH, Lee B.

Int J Mol Med. 2009 Jul;24(1):51-5.


Anoctamin/TMEM16 family members are Ca2+-activated Cl- channels.

Hartzell HC, Yu K, Xiao Q, Chien LT, Qu Z.

J Physiol. 2009 May 15;587(Pt 10):2127-39. doi: 10.1113/jphysiol.2008.163709. Epub 2008 Nov 17. Review.


Curing of the [URE3] prion by Btn2p, a Batten disease-related protein.

Kryndushkin DS, Shewmaker F, Wickner RB.

EMBO J. 2008 Oct 22;27(20):2725-35. doi: 10.1038/emboj.2008.198. Epub 2008 Oct 2.


Visual deficits in a mouse model of Batten disease are the result of optic nerve degeneration and loss of dorsal lateral geniculate thalamic neurons.

Weimer JM, Custer AW, Benedict JW, Alexander NA, Kingsley E, Federoff HJ, Cooper JD, Pearce DA.

Neurobiol Dis. 2006 May;22(2):284-93. Epub 2006 Jan 18.

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