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Items: 1 to 20 of 25

1.

COL4A4 gene study of a European population: description of new mutations causing autosomal dominant Alport syndrome.

Rosado C, Bueno E, Felipe C, González-Sarmiento R.

Int J Mol Epidemiol Genet. 2014 Dec 15;5(4):177-84.

2.

Identification of microRNAs and their target genes in Alport syndrome using deep sequencing of iPSCs samples.

Chen WB, Huang JR, Yu XQ, Lin XC, Dai Y.

J Zhejiang Univ Sci B. 2015 Mar;16(3):235-50. doi: 10.1631/jzus.B1400272.

3.

Laminin α2-mediated focal adhesion kinase activation triggers Alport glomerular pathogenesis.

Delimont D, Dufek BM, Meehan DT, Zallocchi M, Gratton MA, Phillips G, Cosgrove D.

PLoS One. 2014 Jun 10;9(6):e99083. doi: 10.1371/journal.pone.0099083.

4.

Laminin α1 regulates age-related mesangial cell proliferation and mesangial matrix accumulation through the TGF-β pathway.

Ning L, Kurihara H, de Vega S, Ichikawa-Tomikawa N, Xu Z, Nonaka R, Kazuno S, Yamada Y, Miner JH, Arikawa-Hirasawa E.

Am J Pathol. 2014 Jun;184(6):1683-94. doi: 10.1016/j.ajpath.2014.02.006.

5.

α1β1 integrin/Rac1-dependent mesangial invasion of glomerular capillaries in Alport syndrome.

Zallocchi M, Johnson BM, Meehan DT, Delimont D, Cosgrove D.

Am J Pathol. 2013 Oct;183(4):1269-80. doi: 10.1016/j.ajpath.2013.06.015.

6.

Upregulated expression of integrin α1 in mesangial cells and integrin α3 and vimentin in podocytes of Col4a3-null (Alport) mice.

Steenhard BM, Vanacore R, Friedman D, Zelenchuk A, Stroganova L, Isom K, St John PL, Hudson BG, Abrahamson DR.

PLoS One. 2012;7(12):e50745. doi: 10.1371/journal.pone.0050745.

7.

Role of the podocyte (and glomerular endothelium) in building the GBM.

Abrahamson DR.

Semin Nephrol. 2012 Jul;32(4):342-9. doi: 10.1016/j.semnephrol.2012.06.005. Review.

8.

Glomerular basement membrane and related glomerular disease.

Chen YM, Miner JH.

Transl Res. 2012 Oct;160(4):291-7. doi: 10.1016/j.trsl.2012.03.004.

9.

Differential expression of laminin isoforms in diabetic nephropathy and other renal diseases.

Setty S, Michael AA, Fish AJ, Michael Mauer S, Butkowski RJ, Virtanen I, Kim Y.

Mod Pathol. 2012 Jun;25(6):859-68. doi: 10.1038/modpathol.2011.216.

10.

Forced expression of laminin beta1 in podocytes prevents nephrotic syndrome in mice lacking laminin beta2, a model for Pierson syndrome.

Suh JH, Jarad G, VanDeVoorde RG, Miner JH.

Proc Natl Acad Sci U S A. 2011 Sep 13;108(37):15348-53. doi: 10.1073/pnas.1108269108.

11.

Mechanisms and consequences of TGF-ß overexpression by podocytes in progressive podocyte disease.

Lee HS.

Cell Tissue Res. 2012 Jan;347(1):129-40. doi: 10.1007/s00441-011-1169-7. Review.

12.

A missense LAMB2 mutation causes congenital nephrotic syndrome by impairing laminin secretion.

Chen YM, Kikkawa Y, Miner JH.

J Am Soc Nephrol. 2011 May;22(5):849-58. doi: 10.1681/ASN.2010060632.

13.

Glomerular pathology in Alport syndrome: a molecular perspective.

Cosgrove D.

Pediatr Nephrol. 2012 Jun;27(6):885-90. doi: 10.1007/s00467-011-1868-z. Review.

14.

Deletion of von Hippel-Lindau in glomerular podocytes results in glomerular basement membrane thickening, ectopic subepithelial deposition of collagen {alpha}1{alpha}2{alpha}1(IV), expression of neuroglobin, and proteinuria.

Steenhard BM, Isom K, Stroganova L, St John PL, Zelenchuk A, Freeburg PB, Holzman LB, Abrahamson DR.

Am J Pathol. 2010 Jul;177(1):84-96. doi: 10.2353/ajpath.2010.090767.

15.

Laminin alpha4-null mutant mice develop chronic kidney disease with persistent overexpression of platelet-derived growth factor.

Abrass CK, Hansen KM, Patton BL.

Am J Pathol. 2010 Feb;176(2):839-49. doi: 10.2353/ajpath.2010.090570.

16.

Novel targets for immunotherapy in glomerulonephritis.

Foster MH.

Biologics. 2008 Sep;2(3):531-45.

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Neutrophil elastase cleaves laminin-332 (laminin-5) generating peptides that are chemotactic for neutrophils.

Mydel P, Shipley JM, Adair-Kirk TL, Kelley DG, Broekelmann TJ, Mecham RP, Senior RM.

J Biol Chem. 2008 Apr 11;283(15):9513-22. doi: 10.1074/jbc.M706239200.

20.

Integrin beta1-mediated matrix assembly and signaling are critical for the normal development and function of the kidney glomerulus.

Kanasaki K, Kanda Y, Palmsten K, Tanjore H, Lee SB, Lebleu VS, Gattone VH Jr, Kalluri R.

Dev Biol. 2008 Jan 15;313(2):584-93.

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