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Items: 6

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1.

Genistein induces increase in fluid pH, Na+ and HCO3(-) concentration, SLC26A6 and SLC4A4 (NBCe1)-B expression in the uteri of ovariectomized rats.

Chinigarzadeh A, Kasim NF, Muniandy S, Kassim NM, Salleh N.

Int J Mol Sci. 2014 Jan 10;15(1):958-76. doi: 10.3390/ijms15010958.

2.

The SLC26 gene family of anion transporters and channels.

Alper SL, Sharma AK.

Mol Aspects Med. 2013 Apr-Jun;34(2-3):494-515. doi: 10.1016/j.mam.2012.07.009. Review.

3.

Congenital chloride-losing diarrhea causing mutations in the STAS domain result in misfolding and mistrafficking of SLC26A3.

Dorwart MR, Shcheynikov N, Baker JM, Forman-Kay JD, Muallem S, Thomas PJ.

J Biol Chem. 2008 Mar 28;283(13):8711-22. doi: 10.1074/jbc.M704328200. Epub 2008 Jan 23.

4.

Slc26a6 (PAT1) deletion downregulates the apical Na+/H+ exchanger in the straight segment of the proximal tubule.

Petrovic S, Barone S, Wang Z, McDonough AA, Amlal H, Soleimani M.

Am J Nephrol. 2008;28(2):330-8. Epub 2007 Nov 29.

5.

Slc26a6: a cardiac chloride-hydroxyl exchanger and predominant chloride-bicarbonate exchanger of the mouse heart.

Alvarez BV, Kieller DM, Quon AL, Markovich D, Casey JR.

J Physiol. 2004 Dec 15;561(Pt 3):721-34. Epub 2004 Oct 21.

6.

Acute regulation of the SLC26A3 congenital chloride diarrhoea anion exchanger (DRA) expressed in Xenopus oocytes.

Chernova MN, Jiang L, Shmukler BE, Schweinfest CW, Blanco P, Freedman SD, Stewart AK, Alper SL.

J Physiol. 2003 May 15;549(Pt 1):3-19. Epub 2003 Mar 21.

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