Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 110

1.

Transfer of disrupted-in-schizophrenia 1 aggregates between neuronal-like cells occurs in tunnelling nanotubes and is promoted by dopamine.

Zhu S, Abounit S, Korth C, Zurzolo C.

Open Biol. 2017 Mar;7(3). pii: 160328. doi: 10.1098/rsob.160328.

2.

The Effects of Ca2+ Concentration and E200K Mutation on the Aggregation Propensity of PrPC: A Computational Study.

Marrone A, Re N, Storchi L.

PLoS One. 2016 Dec 13;11(12):e0168039. doi: 10.1371/journal.pone.0168039. eCollection 2016.

3.

Related or not? Development of spontaneous Creutzfeldt-Jakob disease in a patient with chronic, well-controlled HIV: A case report and review of the literature.

Babi MA, Kraft BD, Sengupta S, Peterson H, Orgel R, Wegermann Z, Lugogo NL, Luedke MW.

SAGE Open Med Case Rep. 2016 Oct 13;4:2050313X16672153. eCollection 2016.

4.

In Absence of the Cellular Prion Protein, Alterations in Copper Metabolism and Copper-Dependent Oxidase Activity Affect Iron Distribution.

Gasperini L, Meneghetti E, Legname G, Benetti F.

Front Neurosci. 2016 Sep 27;10:437. eCollection 2016.

5.

Simultaneous effects on parvalbumin-positive interneuron and dopaminergic system development in a transgenic rat model for sporadic schizophrenia.

Hamburg H, Trossbach SV, Bader V, Chwiesko C, Kipar A, Sauvage M, Crum WR, Vernon AC, Bidmon HJ, Korth C.

Sci Rep. 2016 Oct 10;6:34946. doi: 10.1038/srep34946.

6.

Whole Blood Gene Expression Profiling in Preclinical and Clinical Cattle Infected with Atypical Bovine Spongiform Encephalopathy.

Xerxa E, Barbisin M, Chieppa MN, Krmac H, Vallino Costassa E, Vatta P, Simmons M, Caramelli M, Casalone C, Corona C, Legname G.

PLoS One. 2016 Apr 13;11(4):e0153425. doi: 10.1371/journal.pone.0153425. eCollection 2016.

7.

Misassembly of full-length Disrupted-in-Schizophrenia 1 protein is linked to altered dopamine homeostasis and behavioral deficits.

Trossbach SV, Bader V, Hecher L, Pum ME, Masoud ST, Prikulis I, Schäble S, de Souza Silva MA, Su P, Boulat B, Chwiesko C, Poschmann G, Stühler K, Lohr KM, Stout KA, Oskamp A, Godsave SF, Müller-Schiffmann A, Bilzer T, Steiner H, Peters PJ, Bauer A, Sauvage M, Ramsey AJ, Miller GW, Liu F, Seeman P, Brandon NJ, Huston JP, Korth C.

Mol Psychiatry. 2016 Nov;21(11):1561-1572. doi: 10.1038/mp.2015.194. Epub 2016 Jan 12.

8.

Synthetic prions with novel strain-specified properties.

Moda F, Le TN, Aulić S, Bistaffa E, Campagnani I, Virgilio T, Indaco A, Palamara L, Andréoletti O, Tagliavini F, Legname G.

PLoS Pathog. 2015 Dec 31;11(12):e1005354. doi: 10.1371/journal.ppat.1005354. eCollection 2015 Dec.

9.

Amyloids in solid-state nuclear magnetic resonance: potential causes of the usually low resolution.

Espargaró A, Busquets MA, Estelrich J, Sabate R.

Int J Nanomedicine. 2015 Nov 9;10:6975-83. doi: 10.2147/IJN.S89385. eCollection 2015.

10.

M1 RNA is important for the in-cell solubility of its cognate C5 protein: Implications for RNA-mediated protein folding.

Son A, Choi SI, Han G, Seong BL.

RNA Biol. 2015;12(11):1198-208. doi: 10.1080/15476286.2015.1096487. Epub 2015 Oct 30.

11.

Aggregation of MBP in chronic demyelination.

Frid K, Einstein O, Friedman-Levi Y, Binyamin O, Ben-Hur T, Gabizon R.

Ann Clin Transl Neurol. 2015 Jul;2(7):711-21. doi: 10.1002/acn3.207. Epub 2015 Jun 6.

12.

Grass plants bind, retain, uptake, and transport infectious prions.

Pritzkow S, Morales R, Moda F, Khan U, Telling GC, Hoover E, Soto C.

Cell Rep. 2015 May 26;11(8):1168-75. doi: 10.1016/j.celrep.2015.04.036. Epub 2015 May 14.

13.

Intrinsically disordered proteins and their (disordered) proteomes in neurodegenerative disorders.

Uversky VN.

Front Aging Neurosci. 2015 Mar 2;7:18. doi: 10.3389/fnagi.2015.00018. eCollection 2015. No abstract available.

14.

Prion protein and copper cooperatively protect neurons by modulating NMDA receptor through S-nitrosylation.

Gasperini L, Meneghetti E, Pastore B, Benetti F, Legname G.

Antioxid Redox Signal. 2015 Mar 20;22(9):772-84. doi: 10.1089/ars.2014.6032. Epub 2015 Feb 4.

15.

Involvement of endogenous retroviruses in prion diseases.

Lee YJ, Jeong BH, Choi EK, Kim YS.

Pathogens. 2013 Aug 12;2(3):533-43. doi: 10.3390/pathogens2030533. Review.

16.

Prion protein and aging.

Gasperini L, Legname G.

Front Cell Dev Biol. 2014 Aug 29;2:44. doi: 10.3389/fcell.2014.00044. eCollection 2014. Review.

17.

The structure of human prions: from biology to structural models-considerations and pitfalls.

Acevedo-Morantes CY, Wille H.

Viruses. 2014 Oct 20;6(10):3875-92. doi: 10.3390/v6103875. Review.

18.

Proteopathic tau seeding predicts tauopathy in vivo.

Holmes BB, Furman JL, Mahan TE, Yamasaki TR, Mirbaha H, Eades WC, Belaygorod L, Cairns NJ, Holtzman DM, Diamond MI.

Proc Natl Acad Sci U S A. 2014 Oct 14;111(41):E4376-85. doi: 10.1073/pnas.1411649111. Epub 2014 Sep 26.

19.

Successes and challenges in phenotype-based lead discovery for prion diseases.

Ghaemmaghami S, Russo M, Renslo AR.

J Med Chem. 2014 Aug 28;57(16):6919-29. doi: 10.1021/jm5001425. Epub 2014 Apr 24. Review.

20.

Computational and experimental studies on β-sheet breakers targeting Aβ1-40 fibrils.

Minicozzi V, Chiaraluce R, Consalvi V, Giordano C, Narcisi C, Punzi P, Rossi GC, Morante S.

J Biol Chem. 2014 Apr 18;289(16):11242-52. doi: 10.1074/jbc.M113.537472. Epub 2014 Feb 28.

Supplemental Content

Support Center