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Items: 9

1.

Role of ROS and RNS Sources in Physiological and Pathological Conditions.

Di Meo S, Reed TT, Venditti P, Victor VM.

Oxid Med Cell Longev. 2016;2016:1245049. doi: 10.1155/2016/1245049. Review.

2.

Microglial cell dysregulation in brain aging and neurodegeneration.

von Bernhardi R, Eugenín-von Bernhardi L, Eugenín J.

Front Aging Neurosci. 2015 Jul 20;7:124. doi: 10.3389/fnagi.2015.00124. Review.

3.

Aging reduces glial uptake and promotes extracellular accumulation of Aβ from a lentiviral vector.

Zhao W, Zhang J, Davis EG, Rebeck GW.

Front Aging Neurosci. 2014 Aug 15;6:210. doi: 10.3389/fnagi.2014.00210.

4.

The role of the innate immune system in Alzheimer's disease and frontotemporal lobar degeneration: an eye on microglia.

Ridolfi E, Barone C, Scarpini E, Galimberti D.

Clin Dev Immunol. 2013;2013:939786. doi: 10.1155/2013/939786. Review.

5.

Microglia during development and aging.

Harry GJ.

Pharmacol Ther. 2013 Sep;139(3):313-26. doi: 10.1016/j.pharmthera.2013.04.013. Review.

6.

Host and viral factors influencing the pathogenesis of HIV-associated neurocognitive disorders.

Jayadev S, Garden GA.

J Neuroimmune Pharmacol. 2009 Jun;4(2):175-89. doi: 10.1007/s11481-009-9154-6. Review.

7.

Neuroinflammation and microglia: considerations and approaches for neurotoxicity assessment.

Harry GJ, Kraft AD.

Expert Opin Drug Metab Toxicol. 2008 Oct;4(10):1265-77. doi: 10.1517/17425255.4.10.1265 . Review.

9.

Chronic thrombin exposure results in an increase in apolipoprotein-E levels.

Mhatre M, Hensley K, Nguyen A, Grammas P.

J Neurosci Res. 2006 Aug 1;84(2):444-9.

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