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Items: 1 to 20 of 92

1.

Direct interaction of Jak1 and v-Abl is required for v-Abl-induced activation of STATs and proliferation.

Danial NN, Losman JA, Lu T, Yip N, Krishnan K, Krolewski J, Goff SP, Wang JY, Rothman PB.

Mol Cell Biol. 1998 Nov;18(11):6795-804.

2.
3.

JAK-STAT signaling activated by Abl oncogenes.

Danial NN, Rothman P.

Oncogene. 2000 May 15;19(21):2523-31. Review.

4.

Jak-STAT signaling induced by the v-abl oncogene.

Danial NN, Pernis A, Rothman PB.

Science. 1995 Sep 29;269(5232):1875-7.

PMID:
7569929
6.

Jak1 plays an essential role for receptor phosphorylation and Stat activation in response to granulocyte colony-stimulating factor.

Shimoda K, Feng J, Murakami H, Nagata S, Watling D, Rogers NC, Stark GR, Kerr IM, Ihle JN.

Blood. 1997 Jul 15;90(2):597-604.

7.

v-Abl signaling disrupts SOCS-1 function in transformed pre-B cells.

Limnander A, Danial NN, Rothman PB.

Mol Cell. 2004 Aug 13;15(3):329-41.

8.

Beta interferon and oncostatin M activate Raf-1 and mitogen-activated protein kinase through a JAK1-dependent pathway.

Stancato LF, Sakatsume M, David M, Dent P, Dong F, Petricoin EF, Krolewski JJ, Silvennoinen O, Saharinen P, Pierce J, Marshall CJ, Sturgill T, Finbloom DS, Larner AC.

Mol Cell Biol. 1997 Jul;17(7):3833-40.

9.

IL-7 reconstitutes multiple aspects of v-Abl-mediated signaling.

Banerjee A, Rothman P.

J Immunol. 1998 Nov 1;161(9):4611-7.

10.

Transforming properties of chimeric TEL-JAK proteins in Ba/F3 cells.

Lacronique V, Boureux A, Monni R, Dumon S, Mauchauffé M, Mayeux P, Gouilleux F, Berger R, Gisselbrecht S, Ghysdael J, Bernard OA.

Blood. 2000 Mar 15;95(6):2076-83.

11.

Abl oncogene bypasses normal regulation of Jak/STAT activation.

Limnander A, Rothman PB.

Cell Cycle. 2004 Dec;3(12):1486-8. Epub 2004 Dec 12. Review.

PMID:
15611644
12.

A major role for the protein tyrosine kinase JAK1 in the JAK/STAT signal transduction pathway in response to interleukin-6.

Guschin D, Rogers N, Briscoe J, Witthuhn B, Watling D, Horn F, Pellegrini S, Yasukawa K, Heinrich P, Stark GR, et al.

EMBO J. 1995 Apr 3;14(7):1421-9.

13.
14.

Rac is required for v-Abl tyrosine kinase to activate mitogenesis.

Renshaw MW, Lea-Chou E, Wang JY.

Curr Biol. 1996 Jan 1;6(1):76-83.

15.

Constitutive activation of STAT5 by the BCR-ABL oncogene in chronic myelogenous leukemia.

Shuai K, Halpern J, ten Hoeve J, Rao X, Sawyers CL.

Oncogene. 1996 Jul 18;13(2):247-54.

PMID:
8710363
16.

The role of the growth hormone (GH) receptor and JAK1 and JAK2 kinases in the activation of Stats 1, 3, and 5 by GH.

Smit LS, Meyer DJ, Billestrup N, Norstedt G, Schwartz J, Carter-Su C.

Mol Endocrinol. 1996 May;10(5):519-33.

PMID:
8732683
17.

Jak1 deficiency leads to enhanced Abelson-induced B-cell tumor formation.

Sexl V, Kovacic B, Piekorz R, Moriggl R, Stoiber D, Hoffmeyer A, Liebminger R, Kudlacek O, Weisz E, Rothammer K, Ihle JN.

Blood. 2003 Jun 15;101(12):4937-43. Epub 2003 Feb 6.

18.

Src kinases and not JAKs activate STATs during IL-3 induced myeloid cell proliferation.

Chaturvedi P, Reddy MV, Reddy EP.

Oncogene. 1998 Apr 2;16(13):1749-58.

19.

Constitutive activation of JAK1 in Src-transformed cells.

Campbell GS, Yu CL, Jove R, Carter-Su C.

J Biol Chem. 1997 Jan 31;272(5):2591-4.

20.

Activation of JAK3, but not JAK1, is critical for IL-2-induced proliferation and STAT5 recruitment by a COOH-terminal region of the IL-2 receptor beta-chain.

Kirken RA, Rui H, Malabarba MG, Howard OM, Kawamura M, O'Shea JJ, Farrar WL.

Cytokine. 1995 Oct;7(7):689-700.

PMID:
8580378

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