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Items: 1 to 20 of 309

1.

Pore mutations alter closing and opening kinetics in Shaker K+ channels.

Molina A, Ortega-Sáenz P, Lopez-Barneo J.

J Physiol. 1998 Jun 1;509 ( Pt 2):327-37.

2.
3.

Effects of external cations and mutations in the pore region on C-type inactivation of Shaker potassium channels.

López-Barneo J, Hoshi T, Heinemann SH, Aldrich RW.

Receptors Channels. 1993;1(1):61-71.

PMID:
8081712
4.
5.

Collapse of conductance is prevented by a glutamate residue conserved in voltage-dependent K(+) channels.

Ortega-Sáenz P, Pardal R, Castellano A, López-Barneo J.

J Gen Physiol. 2000 Aug;116(2):181-90.

6.

Emerging issues of connexin channels: biophysics fills the gap.

Harris AL.

Q Rev Biophys. 2001 Aug;34(3):325-472. Review. Erratum in: Q Rev Biophys 2002 Feb;35(1):109.

PMID:
11838236
7.

Slow inactivation in Shaker K channels is delayed by intracellular tetraethylammonium.

González-Pérez V, Neely A, Tapia C, González-Gutiérrez G, Contreras G, Orio P, Lagos V, Rojas G, Estévez T, Stack K, Naranjo D.

J Gen Physiol. 2008 Dec;132(6):633-50. doi: 10.1085/jgp.200810057.

8.

Residues in a jellyfish shaker-like channel involved in modulation by external potassium.

Grigoriev NG, Spafford JD, Spencer AN.

J Neurophysiol. 1999 Oct;82(4):1740-7.

9.

Changes in voltage activation, Cs+ sensitivity, and ion permeability in H5 mutants of the plant K+ channel KAT1.

Becker D, Dreyer I, Hoth S, Reid JD, Busch H, Lehnen M, Palme K, Hedrich R.

Proc Natl Acad Sci U S A. 1996 Jul 23;93(15):8123-8.

10.
11.

External pore collapse as an inactivation mechanism for Kv4.3 K+ channels.

Eghbali M, Olcese R, Zarei MM, Toro L, Stefani E.

J Membr Biol. 2002 Jul 1;188(1):73-86.

PMID:
12172648
12.

External pore residue mediates slow inactivation in mu 1 rat skeletal muscle sodium channels.

Balser JR, Nuss HB, Chiamvimonvat N, Pérez-García MT, Marban E, Tomaselli GF.

J Physiol. 1996 Jul 15;494 ( Pt 2):431-42.

13.

Loss of shaker K channel conductance in 0 K+ solutions: role of the voltage sensor.

Melishchuk A, Loboda A, Armstrong CM.

Biophys J. 1998 Oct;75(4):1828-35.

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16.

A site accessible to extracellular TEA+ and K+ influences intracellular Mg2+ block of cloned potassium channels.

Ludewig U, Lorra C, Pongs O, Heinemann SH.

Eur Biophys J. 1993;22(4):237-47.

PMID:
8253052
17.
18.

Single mutations strongly alter the K+-selective pore of the K(in) channel KAT1.

Dreyer I, Becker D, Bregante M, Gambale F, Lehnen M, Palme K, Hedrich R.

FEBS Lett. 1998 Jul 3;430(3):370-6.

19.

Slow inactivation in voltage gated potassium channels is insensitive to the binding of pore occluding peptide toxins.

Oliva C, González V, Naranjo D.

Biophys J. 2005 Aug;89(2):1009-19. Epub 2005 May 27.

20.

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