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The binding properties and biological activities of Bcl-2 and Bax in cells exposed to apoptotic stimuli.

Otter I, Conus S, Ravn U, Rager M, Olivier R, Monney L, Fabbro D, Borner C.

J Biol Chem. 1998 Mar 13;273(11):6110-20.


NH2-terminal BH4 domain of Bcl-2 is functional for heterodimerization with Bax and inhibition of apoptosis.

Hirotani M, Zhang Y, Fujita N, Naito M, Tsuruo T.

J Biol Chem. 1999 Jul 16;274(29):20415-20.


Cytoprotection by Bcl-2 requires the pore-forming alpha5 and alpha6 helices.

Matsuyama S, Schendel SL, Xie Z, Reed JC.

J Biol Chem. 1998 Nov 20;273(47):30995-1001.


Bcl-xS and Bax induce different apoptotic pathways in PC12 cells.

Lindenboim L, Yuan J, Stein R.

Oncogene. 2000 Mar 30;19(14):1783-93.


Mutagenesis of the BH3 domain of BAX identifies residues critical for dimerization and killing.

Wang K, Gross A, Waksman G, Korsmeyer SJ.

Mol Cell Biol. 1998 Oct;18(10):6083-9.


Induction of apoptosis in prostate carcinoma cells by BH3 peptides which inhibit Bak/Bcl-2 interactions.

Finnegan NM, Curtin JF, Prevost G, Morgan B, Cotter TG.

Br J Cancer. 2001 Jul 6;85(1):115-21.


A common binding site mediates heterodimerization and homodimerization of Bcl-2 family members.

Diaz JL, Oltersdorf T, Horne W, McConnell M, Wilson G, Weeks S, Garcia T, Fritz LC.

J Biol Chem. 1997 Apr 25;272(17):11350-5.


Radiation-induced apoptosis in human myeloma cell line increases BCL-2/BAX dimer formation and does not result in BAX/BAX homodimerization.

Filippovich IV, Sorokina NI, Lisbona A, Chérel M, Chatal JF.

Int J Cancer. 2001 Jun 1;92(5):651-60.


Structural and functional complementation of an inactive Bcl-2 mutant by Bax truncation.

Ottilie S, Diaz JL, Chang J, Wilson G, Tuffo KM, Weeks S, McConnell M, Wang Y, Oltersdorf T, Fritz LC.

J Biol Chem. 1997 Jul 4;272(27):16955-61.


BID: a novel BH3 domain-only death agonist.

Wang K, Yin XM, Chao DT, Milliman CL, Korsmeyer SJ.

Genes Dev. 1996 Nov 15;10(22):2859-69.


Bax is not the heterodimerization partner necessary for sustained anti-photoreceptor-cell-death activity of Bcl-2.

Eversole-Cire P, Chen J, Simon MI.

Invest Ophthalmol Vis Sci. 2002 May;43(5):1636-44.


[Neural protective agents, propentofylline (PPF) could induce apoptotic cell death in the human glioma cells: analysis of Bcl-2 and Bax alpha/Bax beta expressions].

Satone A, Ukita H, Murakami H, Chiba S, Oshima Y, Ogata A, Ikeda M, Yamaguchi H, Matsumoto A, Ide W, Hashimoto I, Kamada H.

No To Shinkei. 1998 Apr;50(4):330-7. Japanese.


Characterization of the anti-apoptotic mechanism of Bcl-B.

Zhai D, Ke N, Zhang H, Ladror U, Joseph M, Eichinger A, Godzik A, Ng SC, Reed JC.

Biochem J. 2003 Nov 15;376(Pt 1):229-36.


MAP-1, a novel proapoptotic protein containing a BH3-like motif that associates with Bax through its Bcl-2 homology domains.

Tan KO, Tan KM, Chan SL, Yee KS, Bevort M, Ang KC, Yu VC.

J Biol Chem. 2001 Jan 26;276(4):2802-7. Epub 2000 Nov 1.

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