Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 135

1.
2.
3.

Therapeutic effect of Gag-nuclease fusion protein on retrovirus-infected cell cultures.

Schumann G, Qin L, Rein A, Natsoulis G, Boeke JD.

J Virol. 1996 Jul;70(7):4329-37.

4.

Moloney murine leukemia virus protease: bacterial expression and characterization of the purified enzyme.

Menéndez-Arias L, Gotte D, Oroszlan S.

Virology. 1993 Oct;196(2):557-63.

PMID:
8372434
8.

Antiretroviral effect of a gag-RNase HI fusion gene.

Schumann G, Cannon K, Ma WP, Crouch RJ, Boeke JD.

Gene Ther. 1997 Jun;4(6):593-9.

9.

Moloney murine leukemia virus protease expressed in bacteria is enzymatically active.

Cannon K, Qin L, Schumann G, Boeke JD.

Arch Virol. 1998;143(2):381-8.

PMID:
9541621
10.
11.
12.

Possible role of splice acceptor site in expression of unspliced gag-containing message of Moloney murine leukemia virus.

Oshima M, Odawara T, Matano T, Sakahira H, Kuchino Y, Iwamoto A, Yoshikura H.

J Virol. 1996 Apr;70(4):2286-95.

13.

Targeting of Moloney murine leukemia virus gag precursor to the site of virus budding.

Suomalainen M, Hultenby K, Garoff H.

J Cell Biol. 1996 Dec;135(6 Pt 2):1841-52.

14.

Therapeutic effect of a Gag-nuclease fusion protein against retroviral infection in vivo.

Schumann G, Hermankova M, Cannon K, Mankowski JL, Boeke JD.

J Virol. 2001 Aug;75(15):7030-41.

15.

Sequence and comparative structural analysis of the murine leukaemia virus amphotropic strain 4070A RNase H domain.

Ey PL, Freeman NL, Bela B, Haese PM, Li P, McInnes JL.

Arch Virol. 1999;144(11):2185-99.

PMID:
10603172
17.

The basic loop of the RNase H domain of MLV RT is important both for RNase H and for polymerase activity.

Boyer PL, Gao HQ, Frank P, Clark PK, Hughes SH.

Virology. 2001 Mar 30;282(1):206-13.

19.

Mutational analysis of interactions between the Gag precursor proteins of murine leukemia viruses.

Alin K, Goff SP.

Virology. 1996 Feb 15;216(2):418-24. Erratum in: Virology 1996 Aug 1;222(1):297.

Supplemental Content

Support Center