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Items: 1 to 20 of 95

1.

NKR-P1B expression in gut-associated innate lymphoid cells is required for the control of gastrointestinal tract infections.

Abou-Samra E, Hickey Z, Aguilar OA, Scur M, Mahmoud AB, Pyatibrat S, Tu MM, Francispillai J, Mortha A, Carlyle JR, Rahim MMA, Makrigiannis AP.

Cell Mol Immunol. 2018 Oct 1. doi: 10.1038/s41423-018-0169-x. [Epub ahead of print]

PMID:
30275537
2.

The mouse NKR-P1B:Clr-b recognition system is a negative regulator of innate immune responses.

Rahim MM, Chen P, Mottashed AN, Mahmoud AB, Thomas MJ, Zhu Q, Brooks CG, Kartsogiannis V, Gillespie MT, Carlyle JR, Makrigiannis AP.

Blood. 2015 Apr 2;125(14):2217-27. doi: 10.1182/blood-2014-02-556142. Epub 2015 Jan 22.

3.

Expansion and Protection by a Virus-Specific NK Cell Subset Lacking Expression of the Inhibitory NKR-P1B Receptor during Murine Cytomegalovirus Infection.

Rahim MM, Wight A, Mahmoud AB, Aguilar OA, Lee SH, Vidal SM, Carlyle JR, Makrigiannis AP.

J Immunol. 2016 Sep 15;197(6):2325-37. doi: 10.4049/jimmunol.1600776. Epub 2016 Aug 10.

4.

Recognition of host Clr-b by the inhibitory NKR-P1B receptor provides a basis for missing-self recognition.

Balaji GR, Aguilar OA, Tanaka M, Shingu-Vazquez MA, Fu Z, Gully BS, Lanier LL, Carlyle JR, Rossjohn J, Berry R.

Nat Commun. 2018 Nov 5;9(1):4623. doi: 10.1038/s41467-018-06989-2.

5.

The Inhibitory NKR-P1B:Clr-b Recognition Axis Facilitates Detection of Oncogenic Transformation and Cancer Immunosurveillance.

Tanaka M, Fine JH, Kirkham CL, Aguilar OA, Belcheva A, Martin A, Ketela T, Moffat J, Allan DSJ, Carlyle JR.

Cancer Res. 2018 Jul 1;78(13):3589-3603. doi: 10.1158/0008-5472.CAN-17-1688. Epub 2018 Apr 24.

6.
7.

Genetic investigation of MHC-independent missing-self recognition by mouse NK cells using an in vivo bone marrow transplantation model.

Chen P, Aguilar OA, Rahim MM, Allan DS, Fine JH, Kirkham CL, Ma J, Tanaka M, Tu MM, Wight A, Kartsogiannis V, Gillespie MT, Makrigiannis AP, Carlyle JR.

J Immunol. 2015 Mar 15;194(6):2909-18. doi: 10.4049/jimmunol.1401523. Epub 2015 Feb 13.

8.

Frontline Science: A hyporesponsive subset of rat NK cells negative for Ly49s3 and NKR-P1B are precursors to the functionally mature NKR-P1B+ subset.

Sudworth A, Vaage JT, Inngjerdingen M, Kveberg L.

J Leukoc Biol. 2017 Dec;102(6):1289-1298. doi: 10.1189/jlb.1HI0517-177RR. Epub 2017 Jul 26.

PMID:
28747319
9.

Functional characterization of a conserved pair of NKR-P1 receptors expressed by NK cells and T lymphocytes in liver and gut.

Kveberg L, Sudworth A, Todros-Dawda I, Inngjerdingen M, Vaage JT.

Eur J Immunol. 2015 Feb;45(2):501-12. doi: 10.1002/eji.201444710. Epub 2014 Dec 4.

10.

Missing self-recognition of Ocil/Clr-b by inhibitory NKR-P1 natural killer cell receptors.

Carlyle JR, Jamieson AM, Gasser S, Clingan CS, Arase H, Raulet DH.

Proc Natl Acad Sci U S A. 2004 Mar 9;101(10):3527-32. Epub 2004 Feb 27.

11.

Two major groups of rat NKR-P1 receptors can be distinguished based on chromosomal localization, phylogenetic analysis and Clr ligand binding.

Kveberg L, Dai KZ, Westgaard IH, Daws MR, Fossum S, Naper C, Vaage JT.

Eur J Immunol. 2009 Feb;39(2):541-51. doi: 10.1002/eji.200838891.

12.

Two complementary rat NK cell subsets, Ly49s3+ and NKR-P1B+, differ in phenotypic characteristics and responsiveness to cytokines.

Kveberg L, Jiménez-Royo P, Naper C, Rolstad B, Butcher GW, Vaage JT, Inngjerdingen M.

J Leukoc Biol. 2010 Jul;88(1):87-93. doi: 10.1189/jlb.0110039. Epub 2010 Apr 15. Erratum in: J Leukoc Biol. 2012 Jun;91(6):1003.

PMID:
20395458
13.

The NKR-P1B gene product is an inhibitory receptor on SJL/J NK cells.

Kung SK, Su RC, Shannon J, Miller RG.

J Immunol. 1999 May 15;162(10):5876-87.

14.

Interferon-Dependent Induction of Clr-b during Mouse Cytomegalovirus Infection Protects Bystander Cells from Natural Killer Cells via NKR-P1B-Mediated Inhibition.

Kirkham CL, Aguilar OA, Yu T, Tanaka M, Mesci A, Chu KL, Fine JH, Mossman KL, Bremner R, Allan DSJ, Carlyle JR.

J Innate Immun. 2017;9(4):343-358. doi: 10.1159/000454926. Epub 2017 Mar 14.

15.

Modulation of Clr Ligand Expression and NKR-P1 Receptor Function during Murine Cytomegalovirus Infection.

Aguilar OA, Mesci A, Ma J, Chen P, Kirkham CL, Hundrieser J, Voigt S, Allan DS, Carlyle JR.

J Innate Immun. 2015;7(6):584-600. doi: 10.1159/000382032. Epub 2015 May 29.

16.

A Viral Immunoevasin Controls Innate Immunity by Targeting the Prototypical Natural Killer Cell Receptor Family.

Aguilar OA, Berry R, Rahim MMA, Reichel JJ, Popović B, Tanaka M, Fu Z, Balaji GR, Lau TNH, Tu MM, Kirkham CL, Mahmoud AB, Mesci A, Krmpotić A, Allan DSJ, Makrigiannis AP, Jonjić S, Rossjohn J, Carlyle JR.

Cell. 2017 Mar 23;169(1):58-71.e14. doi: 10.1016/j.cell.2017.03.002.

17.

Expression cloning and function of the rat NK activating and inhibitory receptors NKR-P1A and -P1B.

Li J, Rabinovich BA, Hurren R, Shannon J, Miller RG.

Int Immunol. 2003 Mar;15(3):411-6.

PMID:
12618485
18.

Cytomegalovirus evasion of innate immunity by subversion of the NKR-P1B:Clr-b missing-self axis.

Voigt S, Mesci A, Ettinger J, Fine JH, Chen P, Chou W, Carlyle JR.

Immunity. 2007 May;26(5):617-27. Epub 2007 Apr 26.

19.

Chemotherapy-induced genotoxic stress promotes sensitivity to natural killer cell cytotoxicity by enabling missing-self recognition.

Fine JH, Chen P, Mesci A, Allan DS, Gasser S, Raulet DH, Carlyle JR.

Cancer Res. 2010 Sep 15;70(18):7102-13. doi: 10.1158/0008-5472.CAN-10-1316. Epub 2010 Sep 7.

20.

Poxvirus infection-associated downregulation of C-type lectin-related-b prevents NK cell inhibition by NK receptor protein-1B.

Williams KJ, Wilson E, Davidson CL, Aguilar OA, Fu L, Carlyle JR, Burshtyn DN.

J Immunol. 2012 May 15;188(10):4980-91. doi: 10.4049/jimmunol.1103425. Epub 2012 Apr 9.

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