Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 92

1.

Plasma membrane damage caused by listeriolysin O is not repaired through endocytosis of the membrane pore.

Nygård Skalman L, Holst MR, Larsson E, Lundmark R.

Biol Open. 2018 Oct 22;7(10). pii: bio035287. doi: 10.1242/bio.035287.

2.

The pore-forming toxin listeriolysin O mediates a novel entry pathway of L. monocytogenes into human hepatocytes.

Vadia S, Arnett E, Haghighat AC, Wilson-Kubalek EM, Tweten RK, Seveau S.

PLoS Pathog. 2011 Nov;7(11):e1002356. doi: 10.1371/journal.ppat.1002356. Epub 2011 Nov 3.

3.

Endocytic crosstalk: cavins, caveolins, and caveolae regulate clathrin-independent endocytosis.

Chaudhary N, Gomez GA, Howes MT, Lo HP, McMahon KA, Rae JA, Schieber NL, Hill MM, Gaus K, Yap AS, Parton RG.

PLoS Biol. 2014 Apr 8;12(4):e1001832. doi: 10.1371/journal.pbio.1001832. eCollection 2014 Apr.

4.

Mechanisms of endocytosis.

Doherty GJ, McMahon HT.

Annu Rev Biochem. 2009;78:857-902. doi: 10.1146/annurev.biochem.78.081307.110540. Review.

PMID:
19317650
5.

Directly Observing the Lipid-Dependent Self-Assembly and Pore-Forming Mechanism of the Cytolytic Toxin Listeriolysin O.

Mulvihill E, van Pee K, Mari SA, Müller DJ, Yildiz Ö.

Nano Lett. 2015 Oct 14;15(10):6965-73. doi: 10.1021/acs.nanolett.5b02963. Epub 2015 Aug 28.

PMID:
26302195
6.

Uptake of Helicobacter pylori vesicles is facilitated by clathrin-dependent and clathrin-independent endocytic pathways.

Olofsson A, Nygård Skalman L, Obi I, Lundmark R, Arnqvist A.

MBio. 2014 May 20;5(3):e00979-14. doi: 10.1128/mBio.00979-14.

7.

Fluxes of Ca2+ and K+ are required for the listeriolysin O-dependent internalization pathway of Listeria monocytogenes.

Vadia S, Seveau S.

Infect Immun. 2014 Mar;82(3):1084-91. doi: 10.1128/IAI.01067-13. Epub 2013 Dec 23.

8.

Local actin polymerization during endocytic carrier formation.

Hinze C, Boucrot E.

Biochem Soc Trans. 2018 Jun 19;46(3):565-576. doi: 10.1042/BST20170355. Epub 2018 Apr 20. Review.

PMID:
29678956
9.

Damage of eukaryotic cells by the pore-forming toxin sticholysin II: Consequences of the potassium efflux.

Cabezas S, Ho S, Ros U, Lanio ME, Alvarez C, van der Goot FG.

Biochim Biophys Acta Biomembr. 2017 May;1859(5):982-992. doi: 10.1016/j.bbamem.2017.02.001. Epub 2017 Feb 4.

10.

Two-way traffic on the road to plasma membrane repair.

Idone V, Tam C, Andrews NW.

Trends Cell Biol. 2008 Nov;18(11):552-9. doi: 10.1016/j.tcb.2008.09.001. Epub 2008 Oct 9. Review.

11.

Clathrin-independent carriers form a high capacity endocytic sorting system at the leading edge of migrating cells.

Howes MT, Kirkham M, Riches J, Cortese K, Walser PJ, Simpson F, Hill MM, Jones A, Lundmark R, Lindsay MR, Hernandez-Deviez DJ, Hadzic G, McCluskey A, Bashir R, Liu L, Pilch P, McMahon H, Robinson PJ, Hancock JF, Mayor S, Parton RG.

J Cell Biol. 2010 Aug 23;190(4):675-91. doi: 10.1083/jcb.201002119. Epub 2010 Aug 16.

12.

Calcium influx rescues adenylate cyclase-hemolysin from rapid cell membrane removal and enables phagocyte permeabilization by toxin pores.

Fiser R, Masin J, Bumba L, Pospisilova E, Fayolle C, Basler M, Sadilkova L, Adkins I, Kamanova J, Cerny J, Konopasek I, Osicka R, Leclerc C, Sebo P.

PLoS Pathog. 2012;8(4):e1002580. doi: 10.1371/journal.ppat.1002580. Epub 2012 Apr 5.

13.

Listeriolysin O Binding Affects Cholesterol and Phospholipid Acyl Chain Dynamics in Fluid Cholesterol-Rich Bilayers.

Kozorog M, Sani MA, Separovic F, Anderluh G.

Chemistry. 2018 Sep 20;24(53):14220-14225. doi: 10.1002/chem.201802575. Epub 2018 Aug 28.

PMID:
29979814
14.

Endocytosis without clathrin coats.

Nichols BJ, Lippincott-Schwartz J.

Trends Cell Biol. 2001 Oct;11(10):406-12. Review.

PMID:
11567873
15.

Crystal structure of listeriolysin O reveals molecular details of oligomerization and pore formation.

Köster S, van Pee K, Hudel M, Leustik M, Rhinow D, Kühlbrandt W, Chakraborty T, Yildiz Ö.

Nat Commun. 2014 Apr 22;5:3690. doi: 10.1038/ncomms4690.

PMID:
24751541
16.

The pore-forming toxin listeriolysin O is degraded by neutrophil metalloproteinase-8 and fails to mediate Listeria monocytogenes intracellular survival in neutrophils.

Arnett E, Vadia S, Nackerman CC, Oghumu S, Satoskar AR, McLeish KR, Uriarte SM, Seveau S.

J Immunol. 2014 Jan 1;192(1):234-44. doi: 10.4049/jimmunol.1301302. Epub 2013 Dec 6.

17.
18.

Intrinsic repair protects cells from pore-forming toxins by microvesicle shedding.

Romero M, Keyel M, Shi G, Bhattacharjee P, Roth R, Heuser JE, Keyel PA.

Cell Death Differ. 2017 May;24(5):798-808. doi: 10.1038/cdd.2017.11. Epub 2017 Feb 10.

19.

Super-resolution Stimulated Emission Depletion-Fluorescence Correlation Spectroscopy Reveals Nanoscale Membrane Reorganization Induced by Pore-Forming Proteins.

Sarangi NK, P II, Ayappa KG, Visweswariah SS, Basu JK.

Langmuir. 2016 Sep 20;32(37):9649-57. doi: 10.1021/acs.langmuir.6b01848. Epub 2016 Sep 8.

PMID:
27564541
20.

Toxin pores endocytosed during plasma membrane repair traffic into the lumen of MVBs for degradation.

Corrotte M, Fernandes MC, Tam C, Andrews NW.

Traffic. 2012 Mar;13(3):483-94. doi: 10.1111/j.1600-0854.2011.01323.x. Epub 2012 Jan 24.

Supplemental Content

Support Center