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Items: 1 to 20 of 350

1.

Inhibition of Rho Activity Increases Expression of SaeRS-Dependent Virulence Factor Genes in Staphylococcus aureus, Showing a Link between Transcription Termination, Antibiotic Action, and Virulence.

Nagel A, Michalik S, Debarbouille M, Hertlein T, Gesell Salazar M, Rath H, Msadek T, Ohlsen K, van Dijl JM, Völker U, Mäder U.

MBio. 2018 Sep 18;9(5). pii: e01332-18. doi: 10.1128/mBio.01332-18.

2.

VfrB Is a Key Activator of the Staphylococcus aureus SaeRS Two-Component System.

Krute CN, Rice KC, Bose JL.

J Bacteriol. 2017 Feb 14;199(5). pii: e00828-16. doi: 10.1128/JB.00828-16. Print 2017 Mar 1.

3.

Role of Fatty Acid Kinase in Cellular Lipid Homeostasis and SaeRS-Dependent Virulence Factor Expression in Staphylococcus aureus.

Ericson ME, Subramanian C, Frank MW, Rock CO.

MBio. 2017 Aug 1;8(4). pii: e00988-17. doi: 10.1128/mBio.00988-17.

4.

Nutritional Regulation of the Sae Two-Component System by CodY in Staphylococcus aureus.

Mlynek KD, Sause WE, Moormeier DE, Sadykov MR, Hill KR, Torres VJ, Bayles KW, Brinsmade SR.

J Bacteriol. 2018 Mar 26;200(8). pii: e00012-18. doi: 10.1128/JB.00012-18. Print 2018 Apr 15.

5.

Staphylococcus aureus nuclease is an SaeRS-dependent virulence factor.

Olson ME, Nygaard TK, Ackermann L, Watkins RL, Zurek OW, Pallister KB, Griffith S, Kiedrowski MR, Flack CE, Kavanaugh JS, Kreiswirth BN, Horswill AR, Voyich JM.

Infect Immun. 2013 Apr;81(4):1316-24. doi: 10.1128/IAI.01242-12. Epub 2013 Feb 4.

6.

Subinhibitory concentrations of resveratrol reduce alpha-hemolysin production in Staphylococcus aureus isolates by downregulating saeRS.

Duan J, Li M, Hao Z, Shen X, Liu L, Jin Y, Wang S, Guo Y, Yang L, Wang L, Yu F.

Emerg Microbes Infect. 2018 Jul 31;7(1):136. doi: 10.1038/s41426-018-0142-x.

7.
8.

The SaeRS Two-Component System Controls Survival of Staphylococcus aureus in Human Blood through Regulation of Coagulase.

Guo H, Hall JW, Yang J, Ji Y.

Front Cell Infect Microbiol. 2017 May 29;7:204. doi: 10.3389/fcimb.2017.00204. eCollection 2017.

9.

Differential expression and roles of Staphylococcus aureus virulence determinants during colonization and disease.

Jenkins A, Diep BA, Mai TT, Vo NH, Warrener P, Suzich J, Stover CK, Sellman BR.

MBio. 2015 Feb 17;6(1):e02272-14. doi: 10.1128/mBio.02272-14.

10.

Perturbation of Staphylococcus aureus gene expression by the enoyl-acyl carrier protein reductase inhibitor AFN-1252.

Parsons JB, Kukula M, Jackson P, Pulse M, Simecka JW, Valtierra D, Weiss WJ, Kaplan N, Rock CO.

Antimicrob Agents Chemother. 2013 May;57(5):2182-90. doi: 10.1128/AAC.02307-12. Epub 2013 Mar 4.

11.

Influence of the two-component system SaeRS on global gene expression in two different Staphylococcus aureus strains.

Rogasch K, Rühmling V, Pané-Farré J, Höper D, Weinberg C, Fuchs S, Schmudde M, Bröker BM, Wolz C, Hecker M, Engelmann S.

J Bacteriol. 2006 Nov;188(22):7742-58.

12.

Staphylococcus aureus Coordinates Leukocidin Expression and Pathogenesis by Sensing Metabolic Fluxes via RpiRc.

Balasubramanian D, Ohneck EA, Chapman J, Weiss A, Kim MK, Reyes-Robles T, Zhong J, Shaw LN, Lun DS, Ueberheide B, Shopsin B, Torres VJ.

MBio. 2016 Jun 21;7(3). pii: e00818-16. doi: 10.1128/mBio.00818-16.

13.

CodY deletion enhances in vivo virulence of community-associated methicillin-resistant Staphylococcus aureus clone USA300.

Montgomery CP, Boyle-Vavra S, Roux A, Ebine K, Sonenshein AL, Daum RS.

Infect Immun. 2012 Jul;80(7):2382-9. doi: 10.1128/IAI.06172-11. Epub 2012 Apr 23.

14.

Aureusimines in Staphylococcus aureus are not involved in virulence.

Sun F, Cho H, Jeong DW, Li C, He C, Bae T.

PLoS One. 2010 Dec 29;5(12):e15703. doi: 10.1371/journal.pone.0015703.

15.

Staphylococcus aureus Strain Newman D2C Contains Mutations in Major Regulatory Pathways That Cripple Its Pathogenesis.

Sause WE, Copin R, O'Malley A, Chan R, Morrow BJ, Buckley PT, Fernandez J, Lynch AS, Shopsin B, Torres VJ.

J Bacteriol. 2017 Nov 14;199(24). pii: e00476-17. doi: 10.1128/JB.00476-17. Print 2017 Dec 15.

16.

rho is not essential for viability or virulence in Staphylococcus aureus.

Washburn RS, Marra A, Bryant AP, Rosenberg M, Gentry DR.

Antimicrob Agents Chemother. 2001 Apr;45(4):1099-103.

17.

SaeRS-dependent inhibition of biofilm formation in Staphylococcus aureus Newman.

Cue D, Junecko JM, Lei MG, Blevins JS, Smeltzer MS, Lee CY.

PLoS One. 2015 Apr 8;10(4):e0123027. doi: 10.1371/journal.pone.0123027. eCollection 2015.

18.

The SaeRS Two-Component System Is a Direct and Dominant Transcriptional Activator of Toxic Shock Syndrome Toxin 1 in Staphylococcus aureus.

Baroja ML, Herfst CA, Kasper KJ, Xu SX, Gillett DA, Li J, Reid G, McCormick JK.

J Bacteriol. 2016 Sep 9;198(19):2732-42. doi: 10.1128/JB.00425-16. Print 2016 Oct 1.

19.

Termination factor Rho: From the control of pervasive transcription to cell fate determination in Bacillus subtilis.

Bidnenko V, Nicolas P, Grylak-Mielnicka A, Delumeau O, Auger S, Aucouturier A, Guerin C, Repoila F, Bardowski J, Aymerich S, Bidnenko E.

PLoS Genet. 2017 Jul 19;13(7):e1006909. doi: 10.1371/journal.pgen.1006909. eCollection 2017 Jul.

20.

Impact of the functional status of saeRS on in vivo phenotypes of Staphylococcus aureus sarA mutants.

Beenken KE, Mrak LN, Zielinska AK, Atwood DN, Loughran AJ, Griffin LM, Matthews KA, Anthony AM, Spencer HJ, Skinner RA, Post GR, Lee CY, Smeltzer MS.

Mol Microbiol. 2014 Jun;92(6):1299-312. doi: 10.1111/mmi.12629. Epub 2014 May 12.

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